His primary areas of investigation include Cell biology, Immunology, Cytotoxic T cell, CD8 and T cell. Effector is the focus of his Cell biology research. His Effector research incorporates themes from T-helper cell differentiation and Signal transduction.
His Immunology study frequently links to other fields, such as Interleukin 12. His research in CD8 intersects with topics in Chronic infection, Transcription factor and Molecular biology. His Cellular differentiation research is multidisciplinary, relying on both Granzyme and Cell fate determination.
The scientist’s investigation covers issues in Cell biology, Immunology, T cell, Cytotoxic T cell and Cellular differentiation. Steven L. Reiner works on Cell biology which deals in particular with Effector. His biological study deals with issues like T lymphocyte, which deal with fields such as Cellular immunity.
He studied Cytotoxic T cell and CD8 that intersect with Chronic infection. His work deals with themes such as Progenitor cell, Granzyme, Signal transduction and Cell fate determination, which intersect with Cellular differentiation. In his study, CD40 is inextricably linked to Molecular biology, which falls within the broad field of IL-2 receptor.
His primary scientific interests are in Cell biology, T cell, Cellular differentiation, Cell fate determination and Effector. The various areas that Steven L. Reiner examines in his Cell biology study include Cell, Cell division and Transcription factor, CD8, Eomesodermin. His CD8 research is multidisciplinary, incorporating elements of Cytotoxic T cell, Myeloid leukemia and Immunosurveillance.
To a larger extent, Steven L. Reiner studies Immunology with the aim of understanding T cell. His Immunology study incorporates themes from Evolutionary biology and Haematopoiesis. His Effector research includes themes of Acquired immune system, Antigen, Homeostasis, Phenotype and Cell cycle.
Steven L. Reiner mainly focuses on Cell biology, Cellular differentiation, Effector, Cell fate determination and Cell division. His Cell biology research incorporates elements of Transcription factor, Compartment and Lymphocyte. His research integrates issues of Transgene, Lineage, Innate lymphoid cell and Eomesodermin in his study of Cellular differentiation.
His work carried out in the field of Effector brings together such families of science as T cell and Antigen. His Cell fate determination research incorporates themes from Cell, PI3K/AKT/mTOR pathway, Signal transduction and Metabolic pathway. Immune system is a subfield of Immunology that Steven L. Reiner investigates.
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The lineage decisions of helper T cells
Kenneth M. Murphy;Steven L. Reiner.
Nature Reviews Immunology (2002)
Effector and memory CD8 + T cell fate coupled by T-bet and eomesodermin
Andrew M Intlekofer;Naofumi Takemoto;E John Wherry;E John Wherry;Sarah A Longworth.
Nature Immunology (2005)
Role of T-bet in Commitment of TH1 Cells Before IL-12-Dependent Selection
Alan C. Mullen;Frances A. High;Anne S. Hutchins;Hubert W. Lee.
Control of Effector CD8+ T Cell Function by the Transcription Factor Eomesodermin
Erika L. Pearce;Alan C. Mullen;Gislâine A. Martins;Connie M. Krawczyk.
Helper T Cell Differentiation Is Controlled by the Cell Cycle
Jennifer J. Bird;Daniel R. Brown;Alan C. Mullen;Naomi H. Moskowitz.
Asymmetric T lymphocyte division in the initiation of adaptive immune responses.
John T. Chang;Vikram R. Palanivel;Ichiko Kinjyo;Felix Schambach.
Progenitor and terminal subsets of CD8+ T cells cooperate to contain chronic viral infection.
Michael A. Paley;Daniela C. Kroy;Pamela M. Odorizzi;Jonathan B. Johnnidis.
The Transcription Factors T-bet and Eomes Control Key Checkpoints of Natural Killer Cell Maturation
Scott M. Gordon;Julie Chaix;Levi J. Rupp;Junmin Wu.
VACCINATION WITH DNA ENCODING THE IMMUNODOMINANT LACK PARASITE ANTIGEN CONFERS PROTECTIVE IMMUNITY TO MICE INFECTED WITH LEISHMANIA MAJOR
Sanjay Gurunathan;David L. Sacks;Daniel R. Brown;Steven L. Reiner.
Journal of Experimental Medicine (1997)
A Role for the Transcriptional Repressor Blimp-1 in CD8+ T Cell Exhaustion during Chronic Viral Infection
Haina Shin;Shawn D. Blackburn;Andrew M. Intlekofer;Charlly Kao.
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