2005 - Fellow of the American Association for the Advancement of Science (AAAS)
His primary areas of investigation include Cancer research, Carcinogenesis, Transgene, Mammary gland and Genetically modified mouse. His Cancer research research is multidisciplinary, relying on both Mammary tumor, Tumor progression, Oncogene, Immunology and Mouse mammary tumor virus. His Carcinogenesis study combines topics from a wide range of disciplines, such as Prostate, Intraepithelial neoplasia, Protein kinase A, Pathology and Adenocarcinoma.
His Transgene study also includes
Robert D. Cardiff mainly investigates Cancer research, Pathology, Carcinogenesis, Molecular biology and Cancer. Robert D. Cardiff has included themes like Breast cancer, Mammary tumor, Mammary gland and Genetically modified mouse, Transgene in his Cancer research study. His Mammary gland research includes themes of Endocrinology, Hyperplasia and Mammary tumor virus.
His research integrates issues of Tumor progression, Oncogene and Immunology in his study of Genetically modified mouse. His studies examine the connections between Pathology and genetics, as well as such issues in Prostate, with regards to Adenocarcinoma. His Molecular biology study combines topics in areas such as Cell culture, Virology, Antigen, Provirus and Mouse mammary tumor virus.
Robert D. Cardiff spends much of his time researching Cancer research, Pathology, Breast cancer, Carcinogenesis and Cancer. His studies deal with areas such as Epithelial–mesenchymal transition, Metastasis, Mammary gland, Genetically modified mouse and Prostate cancer as well as Cancer research. His work deals with themes such as Prostate, Signal transduction and PTEN, which intersect with Prostate cancer.
His research in Pathology intersects with topics in Tumor progression, Stem cell, Mammary tumor and In vivo. Robert D. Cardiff has researched Breast cancer in several fields, including Phenotype and Epigenetics. His study in Carcinogenesis is interdisciplinary in nature, drawing from both Autophagy, Transgene, Mutation, Mouse mammary tumor virus and PI3K/AKT/mTOR pathway.
Robert D. Cardiff mainly focuses on Cancer research, Signal transduction, Prostate cancer, Pathology and Carcinogenesis. His biological study spans a wide range of topics, including Protein kinase B, Breast cancer, Mammary gland, Metastasis and PI3K/AKT/mTOR pathway. To a larger extent, Robert D. Cardiff studies Cancer with the aim of understanding Mammary gland.
His Signal transduction research is multidisciplinary, relying on both Molecular biology and Programmed cell death. His research combines Antigen and Pathology. Robert D. Cardiff interconnects Autophagy, Apoptosis, Unfolded protein response, Glutamine and Ubiquitin ligase in the investigation of issues within Carcinogenesis.
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Induction of mammary tumors by expression of polyomavirus middle T oncogene: a transgenic mouse model for metastatic disease.
Chantale T. Guy;Robert D. Cardiff;William J. Muller.
Molecular and Cellular Biology (1992)
Expression of the neu protooncogene in the mammary epithelium of transgenic mice induces metastatic disease
Chantale T. Guy;Marc A. Webster;Michael Schaller;Thomas J. Parsons.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Mammary Hyperplasia and Carcinoma in MMTV-cyclin D1 Transgenic Mice
Timothy C. Wang;Robert D. Cardiff;Lawrence Zukerberg;Emma Lees.
MYC inactivation uncovers pluripotent differentiation and tumour dormancy in hepatocellular cancer
Catherine M. Shachaf;Andrew M. Kopelman;Constadina Arvanitis;Åsa Karlsson.
Roles for Nkx3.1 in prostate development and cancer
Rajula Bhatia-Gaur;Annemarie A. Donjacour;Peter J. Sciavolino;Peter J. Sciavolino;Minjung Kim;Minjung Kim.
Genes & Development (1999)
Prostate Pathology of Genetically Engineered Mice: Definitions and Classification. The Consensus Report from the Bar Harbor Meeting of the Mouse Models of Human Cancer Consortium Prostate Pathology Committee
Scott B. Shappell;George V. Thomas;Richard L. Roberts;Ron Herbert.
Cancer Research (2004)
The transcriptional repressor Snail promotes mammary tumor recurrence
Susan E. Moody;Denise Perez;Tien-Chi Pan;Christopher J. Sarkisian.
Cancer Cell (2005)
Somatic inactivation of E-cadherin and p53 in mice leads to metastatic lobular mammary carcinoma through induction of anoikis resistance and angiogenesis
Patrick W.B. Derksen;Xiaoling Liu;Francis Saridin;Hanneke van der Gulden.
Cancer Cell (2006)
PTEN tumor suppressor regulates p53 protein levels and activity through phosphatase-dependent and -independent mechanisms
Daniel J. Freeman;Andrew G. Li;Gang Wei;Heng-Hong Li.
Cancer Cell (2003)
Elevated expression of activated forms of Neu/ErbB-2 and ErbB-3 are involved in the induction of mammary tumors in transgenic mice: implications for human breast cancer.
Peter M. Siegel;Eamonn D. Ryan;Robert D. Cardiff;William J. Muller.
The EMBO Journal (1999)
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