What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Cancer research, Carcinogenesis, Transgene, Mammary gland and Genetically modified mouse.
His Cancer research research is multidisciplinary, relying on both Mammary tumor, Tumor progression, Oncogene, Immunology and Mouse mammary tumor virus.
His Carcinogenesis study combines topics from a wide range of disciplines, such as Prostate, Intraepithelial neoplasia, Protein kinase A, Pathology and Adenocarcinoma.
His Transgene study also includes
- Mammary Epithelium that intertwine with fields like Epithelium and Progenitor cell,
- Receptor which intersects with area such as Progesterone receptor and Alternative splicing.
The various areas that Robert D. Cardiff examines in his Mammary gland study include Cyclin D1, Endocrinology, Wnt signaling pathway and Mammary tumor virus.
His Genetically modified mouse research integrates issues from Molecular biology and Receptor tyrosine kinase.
His most cited work include:
- Induction of mammary tumors by expression of polyomavirus middle T oncogene: a transgenic mouse model for metastatic disease. (1246 citations)
- Expression of the neu protooncogene in the mammary epithelium of transgenic mice induces metastatic disease (1067 citations)
- Mammary Hyperplasia and Carcinoma in MMTV-cyclin D1 Transgenic Mice (898 citations)
What are the main themes of his work throughout his whole career to date?
Robert D. Cardiff mainly investigates Cancer research, Pathology, Carcinogenesis, Molecular biology and Cancer.
Robert D. Cardiff has included themes like Breast cancer, Mammary tumor, Mammary gland and Genetically modified mouse, Transgene in his Cancer research study.
His Mammary gland research includes themes of Endocrinology, Hyperplasia and Mammary tumor virus.
His research integrates issues of Tumor progression, Oncogene and Immunology in his study of Genetically modified mouse.
His studies examine the connections between Pathology and genetics, as well as such issues in Prostate, with regards to Adenocarcinoma.
His Molecular biology study combines topics in areas such as Cell culture, Virology, Antigen, Provirus and Mouse mammary tumor virus.
He most often published in these fields:
- Cancer research (40.29%)
- Pathology (30.34%)
- Carcinogenesis (22.57%)
What were the highlights of his more recent work (between 2011-2020)?
- Cancer research (40.29%)
- Pathology (30.34%)
- Breast cancer (17.96%)
In recent papers he was focusing on the following fields of study:
Robert D. Cardiff spends much of his time researching Cancer research, Pathology, Breast cancer, Carcinogenesis and Cancer.
His studies deal with areas such as Epithelial–mesenchymal transition, Metastasis, Mammary gland, Genetically modified mouse and Prostate cancer as well as Cancer research.
His work deals with themes such as Prostate, Signal transduction and PTEN, which intersect with Prostate cancer.
His research in Pathology intersects with topics in Tumor progression, Stem cell, Mammary tumor and In vivo.
Robert D. Cardiff has researched Breast cancer in several fields, including Phenotype and Epigenetics.
His study in Carcinogenesis is interdisciplinary in nature, drawing from both Autophagy, Transgene, Mutation, Mouse mammary tumor virus and PI3K/AKT/mTOR pathway.
Between 2011 and 2020, his most popular works were:
- Lineage analysis of basal epithelial cells reveals their unexpected plasticity and supports a cell-of-origin model for prostate cancer heterogeneity (212 citations)
- Manual Hematoxylin and Eosin Staining of Mouse Tissue Sections (210 citations)
- PIK3CA(H1047R) induces multipotency and multi-lineage mammary tumours. (184 citations)
In his most recent research, the most cited papers focused on:
Robert D. Cardiff mainly focuses on Cancer research, Signal transduction, Prostate cancer, Pathology and Carcinogenesis.
His biological study spans a wide range of topics, including Protein kinase B, Breast cancer, Mammary gland, Metastasis and PI3K/AKT/mTOR pathway.
To a larger extent, Robert D. Cardiff studies Cancer with the aim of understanding Mammary gland.
His Signal transduction research is multidisciplinary, relying on both Molecular biology and Programmed cell death.
His research combines Antigen and Pathology.
Robert D. Cardiff interconnects Autophagy, Apoptosis, Unfolded protein response, Glutamine and Ubiquitin ligase in the investigation of issues within Carcinogenesis.
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