His main research concerns Internal medicine, Endocrinology, Diabetes mellitus, Insulin resistance and Gut flora. His study in Insulin, Inflammation, Adipose tissue, Glucose tolerance test and Insulin receptor are all subfields of Internal medicine. His Adipose tissue study incorporates themes from Lipopolysaccharide and CD14.
Endocrinology is closely attributed to Receptor in his work. His Diabetes mellitus research is multidisciplinary, incorporating elements of Carbohydrate metabolism and Obesity. He has included themes like Microbial metabolism, Host, Type 2 diabetes and Pharmacology in his Gut flora study.
Rémy Burcelin focuses on Internal medicine, Endocrinology, Diabetes mellitus, Insulin and Gut flora. His Glucagon-like peptide-1 research extends to the thematically linked field of Internal medicine. His research combines Inflammation and Endocrinology.
His research in Inflammation intersects with topics in Lipid metabolism and CD14. His Diabetes mellitus research integrates issues from Enteric nervous system and Vagus nerve. His biological study spans a wide range of topics, including Microbiome, Type 2 diabetes and Metabolomics.
His main research concerns Internal medicine, Endocrinology, Gut flora, Diabetes mellitus and Immunology. Rémy Burcelin has researched Internal medicine in several fields, including Liraglutide and Gastroenterology. The concepts of his Endocrinology study are interwoven with issues in Receptor and Bacterial translocation.
The Gut flora study combines topics in areas such as Glucose tolerance test, Immune system, Metabolomics, Glycemic and Metabolic pathway. Rémy Burcelin works on Diabetes mellitus which deals in particular with Type 2 diabetes. Rémy Burcelin interconnects Offspring, Lipid metabolism and Insulin resistance in the investigation of issues within Immunology.
The scientist’s investigation covers issues in Gut flora, Immunology, Internal medicine, Endocrinology and Diabetes mellitus. His study in Gut flora is interdisciplinary in nature, drawing from both Metabolic pathway, Secretory IgA and Metabolomics. As part of the same scientific family, Rémy Burcelin usually focuses on Immunology, concentrating on Lipid metabolism and intersecting with Intestinal permeability, Immune system and Inflammation.
His Internal medicine research is multidisciplinary, relying on both Phenotype, Feces and Lysozyme. His Endocrinology study combines topics from a wide range of disciplines, such as Taste and Lingual papilla. His study looks at the relationship between Dysbiosis and fields such as Risk factor, as well as how they intersect with chemical problems.
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Metabolic endotoxemia initiates obesity and insulin resistance
Patrice D. Cani;Jacques Amar;Miguel Angel Iglesias;Marjorie Poggi.
Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice
Patrice D. Cani;Rodrigo Bibiloni;Claude Knauf;Aurélie Waget.
Host-Gut Microbiota Metabolic Interactions
Jeremy K. Nicholson;Elaine Holmes;James Kinross;Remy Burcelin.
Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia.
Patrice D. Cani;Audrey M. Neyrinck;F Fava;Claude Knauf.
Metformin alters the gut microbiome of individuals with treatment-naive type 2 diabetes, contributing to the therapeutic effects of the drug
Hao Wu;Eduardo Esteve;Eduardo Esteve;Valentina Tremaroli;Muhammad Tanweer Khan.
Nature Medicine (2017)
Intestinal mucosal adherence and translocation of commensal bacteria at the early onset of type 2 diabetes: molecular mechanisms and probiotic treatment
Jacques Amar;Chantal Chabo;Aurelie Waget;Pascale Klopp.
Embo Molecular Medicine (2011)
Energy intake is associated with endotoxemia in apparently healthy men
Jacques Amar;Rémy Burcelin;Jean Bernard Ruidavets;Patrice D Cani.
The American Journal of Clinical Nutrition (2008)
Metabolic adaptation to a high-fat diet is associated with a change in the gut microbiota
Matteo Serino;Elodie Luche;Sandra Grès;Audrey Baylac.
The AMP-activated protein kinase α2 catalytic subunit controls whole-body insulin sensitivity
Benoit Viollet;Fabrizio Andreelli;Sebastian B. Jørgensen;Christophe Perrin.
Journal of Clinical Investigation (2003)
Apelin Stimulates Glucose Utilization in Normal and Obese Insulin-Resistant Mice
Cédric Dray;Cédric Dray;Claude Knauf;Claude Knauf;Danièle Daviaud;Danièle Daviaud;Aurélie Waget;Aurélie Waget.
Cell Metabolism (2008)
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