His main research concerns Immunology, CD8, Acquired immunodeficiency syndrome, Virology and Viral load. He incorporates Immunology and CD38 in his studies. His CD8 research is multidisciplinary, relying on both Young adult, Regimen, Incidence and Oncology.
His study in Acquired immunodeficiency syndrome is interdisciplinary in nature, drawing from both Viral disease, Blood drawing, Case-control study and Immunodeficiency. In his study, which falls under the umbrella issue of Virology, Blood plasma, Hepatitis C virus and Lipopolysaccharide is strongly linked to T lymphocyte. His work investigates the relationship between Viral load and topics such as Disease reservoir that intersect with problems in Virus Integration and Polymerase chain reaction.
His primary areas of investigation include Immunology, Internal medicine, Virology, Acquired immunodeficiency syndrome and Viral load. His research on Immunology frequently connects to adjacent areas such as Cytotoxic T cell. His work carried out in the field of Virology brings together such families of science as Lymphatic system and Human leukocyte antigen.
Peter W. Hunt works mostly in the field of Acquired immunodeficiency syndrome, limiting it down to concerns involving Viral disease and, occasionally, Immunopathology. His Viral load research focuses on subjects like Antiretroviral therapy, which are linked to Pediatrics. His research in the fields of CD28 overlaps with other disciplines such as CD38.
Peter W. Hunt mostly deals with Internal medicine, Immunology, Inflammation, Viral load and Cohort. His Internal medicine course of study focuses on Acquired immunodeficiency syndrome and Virology. His research in Immune system and CD8 are components of Immunology.
His CD8 study incorporates themes from T cell and Effector. His Inflammation research is multidisciplinary, incorporating elements of Biomarker, Prospective cohort study and Disease. The various areas that Peter W. Hunt examines in his Viral load study include Methamphetamine, Interquartile range, Cohort study and Stimulant.
His main research concerns Immunology, Internal medicine, Viral load, Inflammation and Immune system. His Immunology study typically links adjacent topics like Cytotoxic T cell. The Internal medicine study which covers Nevirapine that intersects with Confidence interval, Depression and Efavirenz.
The study incorporates disciplines such as Stimulant, Methamphetamine, Myocardial infarction, Antiretroviral therapy and Systemic inflammation in addition to Viral load. The concepts of his Inflammation study are interwoven with issues in Biomarker and Disease. His Immune system research incorporates elements of Host, Haemonchus contortus, Gene and Flock.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The major genetic determinants of HIV-1 control affect HLA class I peptide presentation
Pereyra F;Jia X;McLaren Pj.
Science (2010)
Cell death by pyroptosis drives CD4 T-cell depletion in HIV-1 infection
Gilad Doitsh;Nicole L. K. Galloway;Xin Geng;Zhiyuan Yang.
Nature (2014)
Immune activation set point during early HIV infection predicts subsequent CD4+ T-cell changes independent of viral load.
Steven G. Deeks;Christina M. R. Kitchen;Lea Liu;Hua Guo.
Blood (2004)
T Cell Activation Is Associated with Lower CD4+ T Cell Gains in Human Immunodeficiency Virus-Infected Patients with Sustained Viral Suppression during Antiretroviral Therapy
Peter W Hunt;Jeffrey N Martin;Elizabeth Sinclair;Barry Bredt.
The Journal of Infectious Diseases (2003)
Relationship between T Cell Activation and CD4+ T Cell Count in HIV-Seropositive Individuals with Undetectable Plasma HIV RNA Levels in the Absence of Therapy
Peter W. Hunt;Jason Brenchley;Elizabeth Sinclair;Joseph M. McCune.
The Journal of Infectious Diseases (2008)
Plasma Levels of Bacterial DNA Correlate with Immune Activation and the Magnitude of Immune Restoration in Persons with Antiretroviral-Treated HIV Infection
Wei Jiang;Michael M. Lederman;Peter Hunt;Scott F. Sieg.
The Journal of Infectious Diseases (2009)
L-685,458, an Aspartyl Protease Transition State Mimic, Is a Potent Inhibitor of Amyloid β-Protein Precursor γ-Secretase Activity
Mark S. Shearman;Dirk Beher;Earl E. Clarke;Huw D. Lewis.
Biochemistry (2000)
Comparative Analysis of Measures of Viral Reservoirs in HIV-1 Eradication Studies
Susanne Eriksson;Erin H. Graf;Viktor Dahl;Matthew C. Strain.
PLOS Pathogens (2013)
Dysbiosis of the Gut Microbiota Is Associated with HIV Disease Progression and Tryptophan Catabolism
Ivan Vujkovic-Cvijin;Richard M. Dunham;Shoko Iwai;Michael C. Maher.
Science Translational Medicine (2013)
Tryptophan Catabolism by Indoleamine 2,3-Dioxygenase 1 Alters the Balance of TH17 to Regulatory T Cells in HIV Disease
David Favre;Jeff Mold;Peter W. Hunt;Bittoo Kanwar.
Science Translational Medicine (2010)
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