What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Cancer
Immunology, Virology, Viral load, Internal medicine and Viral disease are his primary areas of study.
His studies link Acquired immunodeficiency syndrome with Immunology.
As part of one scientific family, Frederick Hecht deals mainly with the area of Virology, narrowing it down to issues related to the Chronic infection, and often CTL*.
His studies in Viral load integrate themes in fields like Prospective cohort study, Disease reservoir, Polymerase chain reaction and Intensive care medicine.
His studies deal with areas such as Gastroenterology, Pill and Surgery as well as Internal medicine.
His T cell study incorporates themes from Inflammation, Cytotoxic T cell, Pharmacotherapy and Antigen.
His most cited work include:
- Microbial translocation is a cause of systemic immune activation in chronic HIV infection (2742 citations)
- Adherence to protease inhibitors, HIV-1 viral load, and development of drug resistance in an indigent population. (943 citations)
- The major genetic determinants of HIV-1 control affect HLA class I peptide presentation (910 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Immunology, Genetics, Virology, Internal medicine and Viral load.
His research on Immunology frequently links to adjacent areas such as Cytotoxic T cell.
Frederick Hecht combines subjects such as Epitope and RNA with his study of Virology.
His Internal medicine research is multidisciplinary, relying on both Endocrinology and Oncology.
His research investigates the connection with Chromosome and areas like Chromosomal translocation which intersect with concerns in Karyotype, Cytogenetics, Molecular biology, Leukemia and Cancer research.
He is interested in Lentivirus, which is a field of Viral disease.
He most often published in these fields:
- Immunology (26.83%)
- Genetics (18.25%)
- Virology (15.91%)
What were the highlights of his more recent work (between 2009-2021)?
- Immunology (26.83%)
- Virology (15.91%)
- Viral load (10.14%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Immunology, Virology, Viral load, Randomized controlled trial and Internal medicine.
His Immunology study frequently draws connections between related disciplines such as Cytotoxic T cell.
His research investigates the connection between Virology and topics such as RNA that intersect with issues in Transcription.
His Viral load study integrates concerns from other disciplines, such as Cellular immunity, Human leukocyte antigen, Disease and Antiretroviral therapy.
Frederick Hecht has researched Randomized controlled trial in several fields, including Weight loss, Overweight, Physical therapy, Mindfulness and Type 2 diabetes.
His biological study spans a wide range of topics, including Diabetes mellitus, Endocrinology, Gastroenterology and Oncology.
Between 2009 and 2021, his most popular works were:
- The major genetic determinants of HIV-1 control affect HLA class I peptide presentation (910 citations)
- Comparative Analysis of Measures of Viral Reservoirs in HIV-1 Eradication Studies (422 citations)
- Tryptophan Catabolism by Indoleamine 2,3-Dioxygenase 1 Alters the Balance of TH17 to Regulatory T Cells in HIV Disease (386 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Cancer
His primary areas of investigation include Immunology, Virology, Viral load, T cell and Internal medicine.
The study incorporates disciplines such as Cytotoxic T cell and CD38 in addition to Immunology.
His study in Virology focuses on Viral replication in particular.
His Viral load research incorporates themes from Chronic infection, Viremia, Drug holiday and Disease.
His T cell research is multidisciplinary, incorporating perspectives in Immune checkpoint, Inflammation, Antigen, Interleukin 2 and Antibody.
The Virus study combines topics in areas such as Transmission and Acquired immunodeficiency syndrome.
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