His primary areas of study are Cell biology, Immunology, Cellular differentiation, T cell and Tumor necrosis factor alpha. His Cell biology study incorporates themes from Receptor and Transcription factor. As part of his studies on Immunology, he often connects relevant subjects like NFKB1.
His work in Cellular differentiation addresses issues such as Germinal center, which are connected to fields such as T-helper cell differentiation, miR-155, microRNA, Small RNA and Follicular B cell. His work investigates the relationship between T cell and topics such as Cytotoxic T cell that intersect with problems in Jurkat cells. His work deals with themes such as Gene targeting, RELB and Epidermis, which intersect with IκB kinase.
His scientific interests lie mostly in Cell biology, Immunology, Cancer research, Signal transduction and B cell. His Cell biology research is mostly focused on the topic IκB kinase. As part of one scientific family, Marc Schmidt-Supprian deals mainly with the area of Immunology, narrowing it down to issues related to the NFKB1, and often NF-κB.
His Cancer research research includes themes of Diffuse large B-cell lymphoma, Lymphoma, Cancer cell and Bone marrow. His work carried out in the field of Signal transduction brings together such families of science as Molecular biology, Marginal zone, Plasma cell and Ectopic expression. His B cell course of study focuses on TRAF3 and BCL6.
Marc Schmidt-Supprian spends much of his time researching Cancer research, Lymphoma, Cell biology, B cell and Follicular lymphoma. The concepts of his Cancer research study are interwoven with issues in Cancer cell, Spleen, NF-κB and Carcinogenesis. His studies deal with areas such as Cellular differentiation, Diffuse large B-cell lymphoma, Primary mediastinal B-cell lymphoma, REL and B-cell lymphoma as well as Carcinogenesis.
In his study, which falls under the umbrella issue of Lymphoma, Transgene, Precursor cell, Marginal zone and Transcriptome is strongly linked to Receptor. Borrowing concepts from Genome-wide association study, Marc Schmidt-Supprian weaves in ideas under Cell biology. In his research, Kinase, Cytotoxic T cell, Cytotoxicity, Signal transduction and Protein kinase C is intimately related to Stromal cell, which falls under the overarching field of B cell.
His primary scientific interests are in Cell biology, Cancer research, Homeostasis, Follicular lymphoma and Lymphoma. His study in Cell biology is interdisciplinary in nature, drawing from both Cell, Immune system, CD1D, T helper cell and Lymphocyte. His research integrates issues of Protein kinase C, Signal transduction, B cell and Cytotoxicity in his study of Cancer research.
His study in Homeostasis is interdisciplinary in nature, drawing from both T cell, Interleukin 17, Antigen and Single-cell analysis. His research brings together the fields of Effector and T cell. Marc Schmidt-Supprian has included themes like CD74, MHC class II, Antigen processing, Antigen presentation and Proinflammatory cytokine in his Follicular lymphoma study.
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Regulation of the Germinal Center Response by MicroRNA-155
To-Ha Thai;Dinis Pedro Calado;Stefano Casola;K. Mark Ansel.
TNF-mediated inflammatory skin disease in mice with epidermis-specific deletion of IKK2
Manolis Pasparakis;Gilles Courtois;Martin Hafner;Martin Hafner;Marc Schmidt-Supprian;Marc Schmidt-Supprian.
Vagaries of conditional gene targeting
Marc Schmidt-Supprian;Klaus Rajewsky.
Nature Immunology (2007)
Phosphatidylcholine Synthesis for Lipid Droplet Expansion Is Mediated by Localized Activation of CTP:Phosphocholine Cytidylyltransferase
Natalie Krahmer;Yi Guo;Florian Wilfling;Maximiliane Hilger.
Cell Metabolism (2011)
Canonical NF-κB Activity, Dispensable for B Cell Development, Replaces BAFF-Receptor Signals and Promotes B Cell Proliferation upon Activation
Yoshiteru Sasaki;Emmanuel Derudder;Elias Hobeika;Roberta Pelanda.
TNF family member B cell-activating factor (BAFF) receptor-dependent and -independent roles for BAFF in B cell physiology.
Yoshiteru Sasaki;Stefano Casola;Jeffery L. Kutok;Klaus Rajewsky.
Journal of Immunology (2004)
Trans-presentation of IL-6 by dendritic cells is required for the priming of pathogenic T H 17 cells
Sylvia Heink;Nir Yogev;Christoph Garbers;Marina Herwerth.
Nature Immunology (2017)
Cleavage of roquin and regnase-1 by the paracaspase MALT1 releases their cooperatively repressed targets to promote T(H)17 differentiation
Katharina M. Jeltsch;Desheng Hu;Sven Brenner;Jessica Zöller.
Nature Immunology (2014)
K+ Efflux-Independent NLRP3 Inflammasome Activation by Small Molecules Targeting Mitochondria
Christina J. Groß;Ritu Mishra;Katharina S. Schneider;Guillaume Médard.
A bacterial E3 ubiquitin ligase IpaH9.8 targets NEMO/IKK|[gamma]| to dampen the host NF-|[kappa]|B-mediated inflammatory response
Hiroshi Ashida;Minsoo Kim;Marc Schmidt-Supprian;Averil Ma.
Nature Cell Biology (2010)
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