Roland Rad spends much of his time researching Immunology, Helicobacter pylori, Cancer research, CagA and Carcinogenesis. His study in the field of Myelodysplastic syndromes is also linked to topics like Spliceosome. His Cancer research research integrates issues from CA19-9, Pancreatic cancer, Ductal cells, Pancreas and Notch signaling pathway.
His Carcinogenesis research is multidisciplinary, incorporating elements of DNA Mutational Analysis, KRAS, Breast cancer, PI3K/AKT/mTOR pathway and Transplantation. His KRAS study combines topics in areas such as Cell Plasticity, Oncogene, Signal transduction and Bioinformatics. His Cancer study is concerned with the larger field of Genetics.
The scientist’s investigation covers issues in Cancer research, Genetics, Pancreatic cancer, Cancer and Gene. Roland Rad has included themes like Carcinogenesis, KRAS, Metastasis, Tumor progression and PI3K/AKT/mTOR pathway in his Cancer research study. His KRAS research includes themes of Oncogene, Transplantation, YAP1 and Bioinformatics.
His Pancreatic cancer study incorporates themes from Tumor microenvironment, Cancer cell, Transcriptome, Pancreas and In vivo. While the research belongs to areas of Gene, Roland Rad spends his time largely on the problem of Computational biology, intersecting his research to questions surrounding CRISPR. His Mutation study integrates concerns from other disciplines, such as Leukemia and Myeloid leukemia.
Cancer research, Pancreatic cancer, Cell biology, Computational biology and Transcriptome are his primary areas of study. His work deals with themes such as Carcinogenesis, Cancer, Metastasis, Oncogene and KRAS, which intersect with Cancer research. His study looks at the relationship between Carcinogenesis and fields such as Gene silencing, as well as how they intersect with chemical problems.
His work on Tumor progression as part of general Cancer research is frequently linked to SUMO protein, thereby connecting diverse disciplines of science. His Pancreatic cancer study combines topics from a wide range of disciplines, such as Tumor microenvironment, Stromal cell and Hepatocyte growth factor. His study in Computational biology is interdisciplinary in nature, drawing from both Proteome and Proteomics, Gene, Genome.
His main research concerns Cancer research, Computational biology, Steatohepatitis, Transcriptome and Proteomics. His studies deal with areas such as Platelet aggregation inhibitor, Steatosis, Pancreatic cancer, CD8 and Immunotherapy as well as Cancer research. His Pancreatic cancer research entails a greater understanding of Cancer.
He has researched Computational biology in several fields, including Gene targeting, Genetically Engineered Mouse, Gene, CRISPR and Epigenome. In general Gene study, his work on Mutation and Somatic cell often relates to the realm of In utero, thereby connecting several areas of interest. His research on Transcriptome also deals with topics like
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The landscape of cancer genes and mutational processes in breast cancer
Philip J. Stephens;Patrick S. Tarpey;Helen Davies;Peter Van Loo;Peter Van Loo.
CLINICAL RELEVANCE OF THE HELICOBACTER PYLORI GENE FOR BLOOD-GROUP ANTIGEN-BINDING ADHESIN
Markus Gerhard;Norbert Lehn;Nina Neumayer;Thomas Borén.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Generation of transgene-free induced pluripotent mouse stem cells by the piggyBac transposon
Kosuke Yusa;Roland Rad;Junji Takeda;Allan Bradley.
Nature Methods (2009)
Targeted Restoration of the Intestinal Microbiota with a Simple, Defined Bacteriotherapy Resolves Relapsing Clostridium difficile Disease in Mice
Trevor D. Lawley;Simon Clare;Alan W. Walker;Mark D. Stares.
PLOS Pathogens (2012)
Cytokine gene polymorphisms influence mucosal cytokine expression, gastric inflammation, and host specific colonisation during Helicobacter pylori infection
R Rad;A Dossumbekova;B Neu;R Lang.
Leukemia-Associated Somatic Mutations Drive Distinct Patterns of Age-Related Clonal Hemopoiesis
Thomas McKerrell;Naomi Park;Thaidy Moreno;Carolyn S. Grove.
Cell Reports (2015)
Direct identification of clinically relevant neoepitopes presented on native human melanoma tissue by mass spectrometry
Michal Bassani-Sternberg;Eva Braunlein;Richard Klar;Thomas Engleitner;Thomas Engleitner.
Nature Communications (2016)
CD25+/Foxp3+ T cells regulate gastric inflammation and Helicobacter pylori colonization in vivo.
Roland Rad;Lena Brenner;Stefan Bauer;Susanne Schwendy.
Toll-Like Receptor Expression in Human Keratinocytes: Nuclear Factor κB Controlled Gene Activation by Staphylococcus aureus is Toll-Like Receptor 2 But Not Toll-Like Receptor 4 or Platelet Activating Factor Receptor Dependent
Martin Mempel;Verena Voelcker;Gabriele Köllisch;Christian Plank.
Journal of Investigative Dermatology (2003)
Selective Requirement of PI3K/PDK1 Signaling for Kras Oncogene-Driven Pancreatic Cell Plasticity and Cancer
Stefan Eser;Nina Reiff;Marlena Messer;Barbara Seidler.
Cancer Cell (2013)
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