What is he best known for?
The fields of study he is best known for:
- Internal medicine
- Gene
- Cancer
Cancer research, Pancreatic cancer, Pathology, Immunology and Internal medicine are his primary areas of study.
His Cancer research study combines topics from a wide range of disciplines, such as KRAS, Pancreatic Intraepithelial Neoplasia, Carcinogenesis, Cell cycle and Tumor progression.
His study explores the link between Pancreatic cancer and topics such as Pancreas that cross with problems in Pancreatitis and Notch signaling pathway.
Roland M. Schmid combines subjects such as Confocal, Cancer, Metastasis and Hepatocyte with his study of Pathology.
His study focuses on the intersection of Immunology and fields such as Cell biology with connections in the field of Transcriptome and NFKB1.
His Internal medicine research incorporates elements of Gastroenterology, Endocrinology and Oncology.
His most cited work include:
- gp130-Mediated Stat3 Activation in Enterocytes Regulates Cell Survival and Cell-Cycle Progression during Colitis-Associated Tumorigenesis (772 citations)
- Identification, Culture, and Characterization of Pancreatic Stellate Cells in Rats and Humans (764 citations)
- Intestinal Tumorigenesis Initiated by Dedifferentiation and Acquisition of Stem-Cell-like Properties (684 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Internal medicine, Cancer research, Surgery, Pancreatic cancer and Gastroenterology.
His work on Prospective cohort study as part of his general Internal medicine study is frequently connected to In patient, thereby bridging the divide between different branches of science.
His Cancer research research is multidisciplinary, incorporating elements of Carcinogenesis and KRAS.
Roland M. Schmid studies Surgery, focusing on Endoscopy in particular.
His research in Pancreatic cancer intersects with topics in Pancreatic disease, Pancreas and Pathology.
Gastroenterology connects with themes related to Reflux in his study.
He most often published in these fields:
- Internal medicine (34.54%)
- Cancer research (19.28%)
- Surgery (12.16%)
What were the highlights of his more recent work (between 2017-2021)?
- Internal medicine (34.54%)
- Cancer research (19.28%)
- Gastroenterology (11.13%)
In recent papers he was focusing on the following fields of study:
Roland M. Schmid spends much of his time researching Internal medicine, Cancer research, Gastroenterology, Cohort and Cardiology.
Internal medicine is closely attributed to Intensive care in his research.
His Cancer research research includes themes of Carcinogenesis, Metaplasia, KRAS and Pancreatic cancer.
His Pancreatic cancer research incorporates themes from Gene dosage, CDKN2A and YAP1.
His biological study spans a wide range of topics, including Logistic regression and Pneumonia.
The concepts of his Prospective cohort study study are interwoven with issues in Colonoscopy, Clinical endpoint and Adenoma.
Between 2017 and 2021, his most popular works were:
- Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes (147 citations)
- Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes (147 citations)
- Mutant KRAS -driven cancers depend on PTPN11 /SHP2 phosphatase (124 citations)
In his most recent research, the most cited papers focused on:
- Internal medicine
- Gene
- Cancer
Roland M. Schmid mainly investigates Internal medicine, Gastroenterology, Cancer research, Cohort and Carcinogenesis.
His study in the fields of Hemodynamics under the domain of Internal medicine overlaps with other disciplines such as Enterococcus faecalis.
His Gastroenterology research includes elements of Proteobacteria, Cumulative incidence, Firmicutes and Bacteroidetes.
His Cancer research research is multidisciplinary, relying on both Systemic administration, Pancreatic cancer, Oncolytic virus, KRAS and Barrett's esophagus.
In Pancreatic cancer, Roland M. Schmid works on issues like Random forest, which are connected to Radiology.
His studies deal with areas such as Gut flora, Signal transduction, Protein tyrosine phosphatase, Targeted therapy and Tumor progression as well as Carcinogenesis.
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