Louis M. Havekes mostly deals with Internal medicine, Endocrinology, Apolipoprotein E, Very low-density lipoprotein and Lipoprotein. As a member of one scientific family, Louis M. Havekes mostly works in the field of Internal medicine, focusing on Hyperlipidemia and, on occasion, Hypertriglyceridemia. Cholesterol, Adipose tissue, Apolipoprotein B, Insulin and Insulin resistance are among the areas of Endocrinology where the researcher is concentrating his efforts.
His Cholesterol study integrates concerns from other disciplines, such as Coronary artery disease and Postprandial. His research in Apolipoprotein E intersects with topics in Arteriosclerosis, Immunology and Lesion. His study in Lipoprotein is interdisciplinary in nature, drawing from both Trichloroacetic acid, Endothelial stem cell, Superoxide dismutase and Low-density lipoprotein.
Louis M. Havekes mainly investigates Internal medicine, Endocrinology, Apolipoprotein E, Cholesterol and Very low-density lipoprotein. His Internal medicine research focuses on Apolipoprotein B, Lipoprotein lipase, Receptor, Low-density lipoprotein and High-density lipoprotein. His research related to Lipoprotein, Insulin resistance, Insulin, Triglyceride and LDL receptor might be considered part of Endocrinology.
His Apolipoprotein E study incorporates themes from Lesion, Genetically modified mouse, Immunology and Hyperlipidemia. His Cholesterol research integrates issues from Arteriosclerosis and Lipid metabolism. The study incorporates disciplines such as Hypertriglyceridemia, Lipolysis and Steatosis in addition to Very low-density lipoprotein.
His primary scientific interests are in Internal medicine, Endocrinology, Apolipoprotein E, Cholesterol and Insulin resistance. His Very low-density lipoprotein, Insulin, Lipoprotein, Triglyceride and Adipose tissue investigations are all subjects of Endocrinology research. His Very low-density lipoprotein research incorporates themes from LDL receptor, Hypertriglyceridemia and Lipid droplet.
His Apolipoprotein E research incorporates elements of Inflammation, Lesion, Lipid metabolism and Pharmacology. Louis M. Havekes combines subjects such as Steatohepatitis, Rosiglitazone and Glucose uptake with his study of Insulin resistance. His Cholesterylester transfer protein course of study focuses on Apolipoprotein B and Lipidomics.
His primary scientific interests are in Internal medicine, Endocrinology, Brown adipose tissue, Adipose tissue and Lipolysis. His Internal medicine study is mostly concerned with Steatosis, Lipoprotein, Obesity, Lipoprotein lipase and Cholesterol. His research brings together the fields of Apolipoprotein E and Endocrinology.
The study incorporates disciplines such as LDL receptor, Fatty acid, White adipose tissue, Receptor and Thermogenesis in addition to Brown adipose tissue. In his research on the topic of Adipose tissue, Thyroid, Energy homeostasis and Resting energy expenditure is strongly related with Hormone. As part of one scientific family, Louis M. Havekes deals mainly with the area of Lipolysis, narrowing it down to issues related to the CD36, and often Basal, Scavenger receptor and Adipocyte.
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Postprandial lipoprotein metabolism in normolipidemic men with and without coronary artery disease.
P H Groot;W A van Stiphout;X H Krauss;H Jansen.
Arteriosclerosis, Thrombosis, and Vascular Biology (1991)
Role of ApoCs in Lipoprotein Metabolism Functional Differences Between ApoC1, ApoC2, and ApoC3
Miek C. Jong;Marten H. Hofker;Louis M. Havekes.
Arteriosclerosis, Thrombosis, and Vascular Biology (1999)
Stimulation of lipogenesis by pharmacological activation of the liver X receptor leads to production of large, triglyceride-rich very low density lipoprotein particles
Aldo Grefhorst;Baukje M Elzinga;Peter J Voshol;Torsten Plösch.
Journal of Biological Chemistry (2002)
Apolipoprotein E4 allele in a population-based study of early-onset Alzheimer's disease
C.M. van Duijn;P. de Knijff;M. Cruts;A. Wehnert;A. Wehnert.
Nature Genetics (1994)
Selective parasympathetic innervation of subcutaneous and intra-abdominal fat — functional implications
Felix Kreier;Eric Fliers;Peter J. Voshol;Corbert G. Van Eden.
Journal of Clinical Investigation (2002)
Metabolomics in the context of systems biology: bridging traditional Chinese medicine and molecular pharmacology.
Mei Wang;Robert-Jan A. N. Lamers;Robert-Jan A. N. Lamers;Henrie A. A. J. Korthout;Joop H. J. van Nesselrooij.
Phytotherapy Research (2005)
Brown fat activation reduces hypercholesterolaemia and protects from atherosclerosis development.
Jimmy F. P. Berbée;Mariëtte R Boon;P. Padmini S. J. Khedoe;Alexander Bartelt.
Nature Communications (2015)
ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis.
Frank G. Schaap;Patrick C.N. Rensen;Peter J. Voshol;Carlos Vrins.
Journal of Biological Chemistry (2004)
Diet-induced hyperlipoproteinemia and atherosclerosis in apolipoprotein E3-Leiden transgenic mice.
B. J. M. Van Vlijmen;A. M. J. M. Van Den Maagdenberg;M. J. J. Gijbels;H. Van Der Boom.
Journal of Clinical Investigation (1994)
Hepatic Steatosis: A Mediator of the Metabolic Syndrome: Lessons From Animal Models
M. den Boer;P.J. Voshol;F. Kuipers;L.M. Havekes.
Arteriosclerosis, Thrombosis, and Vascular Biology (2004)
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