Kok P. M. van Kessel spends much of his time researching Microbiology, Staphylococcus aureus, Chemotaxis, Antimicrobial peptides and Complement inhibitor. His Microbiology study integrates concerns from other disciplines, such as Alternative complement pathway, Complement system, Classical complement pathway, Complement receptor and Innate immune system. His Staphylococcus aureus research is multidisciplinary, relying on both Virology, Transfection, Anti-inflammatory, Receptor and Virulence.
The concepts of his Chemotaxis study are interwoven with issues in HEK 293 cells, Stimulation, Fusion protein and Cell biology. He combines subjects such as Pseudomonas aeruginosa, Defensin, Immunology and Immunity with his study of Antimicrobial peptides. His work carried out in the field of Complement inhibitor brings together such families of science as Complement control protein and Complement component 2.
His primary areas of investigation include Microbiology, Staphylococcus aureus, Receptor, Biochemistry and Immune system. His Microbiology research includes elements of Innate immune system, Chemotaxis and Antibody, Complement system. The various areas that he examines in his Staphylococcus aureus study include Virulence factor, Virulence, Virology and Immunology.
His biological study spans a wide range of topics, including C5a receptor, Lysis and Cell biology. His Biochemistry research includes themes of Molecular biology and Serum amyloid P component. His study in Immune system is interdisciplinary in nature, drawing from both Phagosome, Antibody opsonization and Antigen.
His main research concerns Immune system, Staphylococcus aureus, Microbiology, Receptor and Antibody. His Immune system research includes elements of Phagocytosis, Cell biology, Chemotaxis and Interleukin 8. His work on Staphylococcal infections as part of general Staphylococcus aureus research is often related to Coagulation Factor Xa, thus linking different fields of science.
His research in Microbiology tackles topics such as Plasma protein binding which are related to areas like Cell and Phage display. The study incorporates disciplines such as C5a receptor, Toxin and Cell culture in addition to Receptor. Kok P. M. van Kessel has researched Antibody in several fields, including Virulence factor, Teichoic acid and Polysaccharide.
Kok P. M. van Kessel mostly deals with Microbiology, Staphylococcus aureus, Receptor, Leukocidin and Immune system. In his study, Cell, Phagocytosis and Phagosome is strongly linked to Plasma protein binding, which falls under the umbrella field of Microbiology. The Staphylococcus aureus study combines topics in areas such as Innate immune system and Virulence.
His Receptor research is multidisciplinary, incorporating perspectives in Phenotype, Toxin and Lysis. The various areas that Kok P. M. van Kessel examines in his Toxin study include C5a receptor, Cell culture and Cell surface receptor. His Immune system study combines topics in areas such as Virulence factor, Phage display, Antibody opsonization and Myeloperoxidase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Staphylococcus aureus resistance to human defensins and evasion of neutrophil killing via the novel virulence factor MprF is based on modification of membrane lipids with L-lysine
Andreas Peschel;Andreas Peschel;Ralph W. Jack;Michael Otto;L. Vincent Collins.
Journal of Experimental Medicine (2001)
Chemotaxis Inhibitory Protein of Staphylococcus aureus, a Bacterial Antiinflammatory Agent
Carla J.C. de Haas;Karin Ellen Veldkamp;Andreas Peschel;Andreas Peschel;Floor Weerkamp.
Journal of Experimental Medicine (2004)
The Innate Immune Modulators Staphylococcal Complement Inhibitor and Chemotaxis Inhibitory Protein of Staphylococcus aureus Are Located on β-Hemolysin-Converting Bacteriophages
Willem J. B. van Wamel;Suzan H. M. Rooijakkers;Maartje Ruyken;Kok P. M. van Kessel.
Journal of Bacteriology (2006)
Immune evasion by a staphylococcal complement inhibitor that acts on C3 convertases.
Suzan H M Rooijakkers;Maartje Ruyken;Anja Roos;Mohamed R Daha.
Nature Immunology (2005)
Lipoprotein metabolism in patients with severe sepsis
Henk J. van Leeuwen;Eric C. J. M. Heezius;Geesje M. Dallinga;Jos A. G. van Strijp.
Critical Care Medicine (2003)
Staphylococcal innate immune evasion
Suzan H.M. Rooijakkers;Kok P.M. van Kessel;Jos A.G. van Strijp.
Trends in Microbiology (2005)
Staphylococcus aureus Strains Lacking d-Alanine Modifications of Teichoic Acids Are Highly Susceptible to Human Neutrophil Killing and Are Virulence Attenuated in Mice
L. Vincent Collins;Sascha A. Kristian;Christopher Weidenmaier;Marion Faigle.
The Journal of Infectious Diseases (2002)
The Staphylococcal Toxin Panton-Valentine Leukocidin Targets Human C5a Receptors
András N. Spaan;Thomas Henry;Willemien J.M. van Rooijen;Magali Perret.
Cell Host & Microbe (2013)
Chemotaxis inhibitory protein of Staphylococcus aureus binds specifically to the C5a and formylated peptide receptor.
Bent Postma;Miriam J. Poppelier;Joost C. van Galen;Eric R. Prossnitz.
Journal of Immunology (2004)
Staphylococcal complement evasion by various convertase-blocking molecules
Ilse Jongerius;Jörg Köhl;Manoj K. Pandey;Maartje Ruyken.
Journal of Experimental Medicine (2007)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: