His primary areas of study are Microbiology, Staphylococcus aureus, Virulence, Innate immune system and Complement system. His research in Microbiology intersects with topics in Complement inhibitor, Classical complement pathway, Alternative complement pathway and Complement receptor. His Staphylococcus aureus study combines topics in areas such as Pathogen, Virology and Receptor, Chemotaxis, Antagonist.
His Virulence research is multidisciplinary, incorporating elements of Defensin and Methicillin-resistant Staphylococcus aureus, Leukocidin. His Innate immune system study integrates concerns from other disciplines, such as Staphylococcal infections and Myeloperoxidase. The subject of his Complement system research is within the realm of Immune system.
Microbiology, Staphylococcus aureus, Immune system, Receptor and Immunology are his primary areas of study. His Microbiology research is multidisciplinary, relying on both Innate immune system, Chemotaxis, Complement system and Virulence. His research integrates issues of Bacteria, Defensin and Virology in his study of Virulence.
The study incorporates disciplines such as Pathogen and In vivo in addition to Staphylococcus aureus. His Immune system research focuses on subjects like Cell biology, which are linked to Fusion protein and Superantigen. As part of the same scientific family, Jos A. G. van Strijp usually focuses on Receptor, concentrating on Leukocidin and intersecting with Toxin.
His main research concerns Microbiology, Staphylococcus aureus, Immune system, Cell biology and Receptor. His Microbiology research incorporates themes from Recombinant DNA, Pathogenesis, Streptococcus, Bacteria and Virulence. His biological study spans a wide range of topics, including Pathogen, Complement system, Antibody opsonization and Vaccine antigen.
His Immune system study incorporates themes from Phagocytosis, Antibody and Chemotaxis. His research investigates the link between Cell biology and topics such as Inflammation that cross with problems in Factor H. His studies deal with areas such as Mutagenesis, Toxin, Cell culture and Leukocidin as well as Receptor.
His primary scientific interests are in Staphylococcus aureus, Microbiology, Immune system, Pathogen and Cell biology. His Staphylococcus aureus research includes themes of Complement system, Vaccine antigen and Virulence. In his study, C5a receptor and Lysis is strongly linked to Receptor, which falls under the umbrella field of Microbiology.
His work on Innate immune system and Langerhans cell is typically connected to Langerin as part of general Immune system study, connecting several disciplines of science. In his study, which falls under the umbrella issue of Pathogen, Staphylococcus epidermidis, Antibody, Antibody opsonization, Immunology and Antibiotic resistance is strongly linked to Staphylococcal infections. His work investigates the relationship between Cell biology and topics such as Inflammation that intersect with problems in In vivo, Alternative complement pathway, Factor H and iC3b.
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Staphylococcus aureus resistance to human defensins and evasion of neutrophil killing via the novel virulence factor MprF is based on modification of membrane lipids with L-lysine
Andreas Peschel;Andreas Peschel;Ralph W. Jack;Michael Otto;L. Vincent Collins.
Journal of Experimental Medicine (2001)
Chemotaxis Inhibitory Protein of Staphylococcus aureus, a Bacterial Antiinflammatory Agent
Carla J.C. de Haas;Karin Ellen Veldkamp;Andreas Peschel;Andreas Peschel;Floor Weerkamp.
Journal of Experimental Medicine (2004)
The Innate Immune Modulators Staphylococcal Complement Inhibitor and Chemotaxis Inhibitory Protein of Staphylococcus aureus Are Located on β-Hemolysin-Converting Bacteriophages
Willem J. B. van Wamel;Suzan H. M. Rooijakkers;Maartje Ruyken;Kok P. M. van Kessel.
Journal of Bacteriology (2006)
Immune evasion by a staphylococcal complement inhibitor that acts on C3 convertases.
Suzan H M Rooijakkers;Maartje Ruyken;Anja Roos;Mohamed R Daha.
Nature Immunology (2005)
Lipoprotein metabolism in patients with severe sepsis
Henk J. van Leeuwen;Eric C. J. M. Heezius;Geesje M. Dallinga;Jos A. G. van Strijp.
Critical Care Medicine (2003)
Staphylococcal innate immune evasion
Suzan H.M. Rooijakkers;Kok P.M. van Kessel;Jos A.G. van Strijp.
Trends in Microbiology (2005)
Staphylococcus aureus Strains Lacking d-Alanine Modifications of Teichoic Acids Are Highly Susceptible to Human Neutrophil Killing and Are Virulence Attenuated in Mice
L. Vincent Collins;Sascha A. Kristian;Christopher Weidenmaier;Marion Faigle.
The Journal of Infectious Diseases (2002)
Human Formyl Peptide Receptor 2 Senses Highly Pathogenic Staphylococcus aureus
Dorothee Kretschmer;Anne-Kathrin Gleske;Maren Rautenberg;Rong Wang.
Cell Host & Microbe (2010)
The Staphylococcal Toxin Panton-Valentine Leukocidin Targets Human C5a Receptors
András N. Spaan;Thomas Henry;Willemien J.M. van Rooijen;Magali Perret.
Cell Host & Microbe (2013)
Staphylococcal complement evasion by various convertase-blocking molecules
Ilse Jongerius;Jörg Köhl;Manoj K. Pandey;Maartje Ruyken.
Journal of Experimental Medicine (2007)
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