D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 56 Citations 11,045 123 World Ranking 1528 National Ranking 54

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Bacteria

His primary areas of study are Microbiology, Staphylococcus aureus, Virulence, Innate immune system and Complement system. His research in Microbiology intersects with topics in Complement inhibitor, Classical complement pathway, Alternative complement pathway and Complement receptor. His Staphylococcus aureus study combines topics in areas such as Pathogen, Virology and Receptor, Chemotaxis, Antagonist.

His Virulence research is multidisciplinary, incorporating elements of Defensin and Methicillin-resistant Staphylococcus aureus, Leukocidin. His Innate immune system study integrates concerns from other disciplines, such as Staphylococcal infections and Myeloperoxidase. The subject of his Complement system research is within the realm of Immune system.

His most cited work include:

  • Staphylococcus aureus resistance to human defensins and evasion of neutrophil killing via the novel virulence factor MprF is based on modification of membrane lipids with L-lysine (602 citations)
  • The Innate Immune Modulators Staphylococcal Complement Inhibitor and Chemotaxis Inhibitory Protein of Staphylococcus aureus Are Located on β-Hemolysin-Converting Bacteriophages (388 citations)
  • Chemotaxis Inhibitory Protein of Staphylococcus aureus, a Bacterial Antiinflammatory Agent (362 citations)

What are the main themes of his work throughout his whole career to date?

Microbiology, Staphylococcus aureus, Immune system, Receptor and Immunology are his primary areas of study. His Microbiology research is multidisciplinary, relying on both Innate immune system, Chemotaxis, Complement system and Virulence. His research integrates issues of Bacteria, Defensin and Virology in his study of Virulence.

The study incorporates disciplines such as Pathogen and In vivo in addition to Staphylococcus aureus. His Immune system research focuses on subjects like Cell biology, which are linked to Fusion protein and Superantigen. As part of the same scientific family, Jos A. G. van Strijp usually focuses on Receptor, concentrating on Leukocidin and intersecting with Toxin.

He most often published in these fields:

  • Microbiology (54.49%)
  • Staphylococcus aureus (32.02%)
  • Immune system (24.72%)

What were the highlights of his more recent work (between 2017-2021)?

  • Microbiology (54.49%)
  • Staphylococcus aureus (32.02%)
  • Immune system (24.72%)

In recent papers he was focusing on the following fields of study:

His main research concerns Microbiology, Staphylococcus aureus, Immune system, Cell biology and Receptor. His Microbiology research incorporates themes from Recombinant DNA, Pathogenesis, Streptococcus, Bacteria and Virulence. His biological study spans a wide range of topics, including Pathogen, Complement system, Antibody opsonization and Vaccine antigen.

His Immune system study incorporates themes from Phagocytosis, Antibody and Chemotaxis. His research investigates the link between Cell biology and topics such as Inflammation that cross with problems in Factor H. His studies deal with areas such as Mutagenesis, Toxin, Cell culture and Leukocidin as well as Receptor.

Between 2017 and 2021, his most popular works were:

  • Human CD45 is an F-component-specific receptor for the staphylococcal toxin Panton–Valentine leukocidin (43 citations)
  • Human skin commensals augment Staphylococcus aureus pathogenesis (30 citations)
  • Immune Evasion by Staphylococcus aureus. (25 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Bacteria

His primary scientific interests are in Staphylococcus aureus, Microbiology, Immune system, Pathogen and Cell biology. His Staphylococcus aureus research includes themes of Complement system, Vaccine antigen and Virulence. In his study, C5a receptor and Lysis is strongly linked to Receptor, which falls under the umbrella field of Microbiology.

His work on Innate immune system and Langerhans cell is typically connected to Langerin as part of general Immune system study, connecting several disciplines of science. In his study, which falls under the umbrella issue of Pathogen, Staphylococcus epidermidis, Antibody, Antibody opsonization, Immunology and Antibiotic resistance is strongly linked to Staphylococcal infections. His work investigates the relationship between Cell biology and topics such as Inflammation that intersect with problems in In vivo, Alternative complement pathway, Factor H and iC3b.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Staphylococcus aureus resistance to human defensins and evasion of neutrophil killing via the novel virulence factor MprF is based on modification of membrane lipids with L-lysine

Andreas Peschel;Andreas Peschel;Ralph W. Jack;Michael Otto;L. Vincent Collins.
Journal of Experimental Medicine (2001)

940 Citations

Chemotaxis Inhibitory Protein of Staphylococcus aureus, a Bacterial Antiinflammatory Agent

Carla J.C. de Haas;Karin Ellen Veldkamp;Andreas Peschel;Andreas Peschel;Floor Weerkamp.
Journal of Experimental Medicine (2004)

520 Citations

The Innate Immune Modulators Staphylococcal Complement Inhibitor and Chemotaxis Inhibitory Protein of Staphylococcus aureus Are Located on β-Hemolysin-Converting Bacteriophages

Willem J. B. van Wamel;Suzan H. M. Rooijakkers;Maartje Ruyken;Kok P. M. van Kessel.
Journal of Bacteriology (2006)

510 Citations

Immune evasion by a staphylococcal complement inhibitor that acts on C3 convertases.

Suzan H M Rooijakkers;Maartje Ruyken;Anja Roos;Mohamed R Daha.
Nature Immunology (2005)

488 Citations

Lipoprotein metabolism in patients with severe sepsis

Henk J. van Leeuwen;Eric C. J. M. Heezius;Geesje M. Dallinga;Jos A. G. van Strijp.
Critical Care Medicine (2003)

349 Citations

Staphylococcal innate immune evasion

Suzan H.M. Rooijakkers;Kok P.M. van Kessel;Jos A.G. van Strijp.
Trends in Microbiology (2005)

313 Citations

Staphylococcus aureus Strains Lacking d-Alanine Modifications of Teichoic Acids Are Highly Susceptible to Human Neutrophil Killing and Are Virulence Attenuated in Mice

L. Vincent Collins;Sascha A. Kristian;Christopher Weidenmaier;Marion Faigle.
The Journal of Infectious Diseases (2002)

297 Citations

Human Formyl Peptide Receptor 2 Senses Highly Pathogenic Staphylococcus aureus

Dorothee Kretschmer;Anne-Kathrin Gleske;Maren Rautenberg;Rong Wang.
Cell Host & Microbe (2010)

274 Citations

The Staphylococcal Toxin Panton-Valentine Leukocidin Targets Human C5a Receptors

András N. Spaan;Thomas Henry;Willemien J.M. van Rooijen;Magali Perret.
Cell Host & Microbe (2013)

267 Citations

Staphylococcal complement evasion by various convertase-blocking molecules

Ilse Jongerius;Jörg Köhl;Manoj K. Pandey;Maartje Ruyken.
Journal of Experimental Medicine (2007)

259 Citations

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