Victor J. Torres mainly focuses on Microbiology, Staphylococcus aureus, Virulence, Staphylococcal infections and Pathogen. His Microbiology study combines topics from a wide range of disciplines, such as Immunology, Antigen, Receptor and CXC chemokine receptors, Chemokine receptor. His Staphylococcus aureus research integrates issues from Innate immune system, Immune system, Pathogenesis and Heme.
His Immune system research is multidisciplinary, relying on both Toxin and Transcriptome. Victor J. Torres interconnects Tropism and Bacteria in the investigation of issues within Virulence. His studies in Staphylococcal infections integrate themes in fields like Phagocytosis and Effector.
His primary areas of study are Microbiology, Staphylococcus aureus, Virulence, Leukocidin and Immunology. Within one scientific family, Victor J. Torres focuses on topics pertaining to Immune system under Microbiology, and may sometimes address concerns connected to Cytolysis. As part of one scientific family, Victor J. Torres deals mainly with the area of Staphylococcus aureus, narrowing it down to issues related to the Pathogen, and often Host.
As a part of the same scientific family, Victor J. Torres mostly works in the field of Virulence, focusing on Regulation of gene expression and, on occasion, Transcription factor. His Leukocidin study incorporates themes from Aureus infection, Biofilm, Virology, Cytotoxicity and Monoclonal antibody. His Immunology research is multidisciplinary, incorporating elements of Cytotoxic T cell, Antimicrobial and Antibiotics.
Staphylococcus aureus, Microbiology, Virulence, Leukocidin and Immunology are his primary areas of study. In general Staphylococcus aureus study, his work on Staphylococcus often relates to the realm of Cation diffusion facilitator, thereby connecting several areas of interest. His Microbiology research includes themes of Epitope, Innate immune system, Immune system and In vivo.
As a member of one scientific family, Victor J. Torres mostly works in the field of Immune system, focusing on Cytolysis and, on occasion, Ex vivo. His research in Virulence intersects with topics in Toxin, Pathogen and Methicillin-resistant Staphylococcus aureus. His work deals with themes such as Perforation, Antibody, Monoclonal antibody and Vaccination, which intersect with Leukocidin.
His primary areas of investigation include Microbiology, Virulence, Cell biology, Staphylococcus aureus and Innate immune system. In most of his Microbiology studies, his work intersects topics such as Immune system. His Virulence research is multidisciplinary, incorporating perspectives in Oxidative stress, Human morbidity, Plasmid and Purr.
His Cell biology research includes elements of Inflammation, Adipose tissue macrophages, White adipose tissue, Tissue homeostasis and CD11c. His Staphylococcus aureus research incorporates elements of Pathogen, Paraquat, Efflux and Intracellular. His Innate immune system study also includes fields such as
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Metal Chelation and Inhibition of Bacterial Growth in Tissue Abscesses
Brian D. Corbin;Erin H. Seeley;Andrea Raab;Joerg Feldmann.
Focused specificity of intestinal TH17 cells towards commensal bacterial antigens
Yi Yang;Miriam B. Torchinsky;Michael Gobert;Huizhong Xiong.
Staphylococcus aureus IsdB Is a Hemoglobin Receptor Required for Heme Iron Utilization
Victor J. Torres;Gleb Pishchany;Munir Humayun;Olaf Schneewind.
Journal of Bacteriology (2006)
Inhibition of primary human T cell proliferation by Helicobacter pylori vacuolating toxin (VacA) is independent of VacA effects on IL-2 secretion
Mark S. Sundrud;Victor J. Torres;Derya Unutmaz;Timothy L. Cover.
Proceedings of the National Academy of Sciences of the United States of America (2004)
CCR5 is a receptor for Staphylococcus aureus leukotoxin ED
Francis Alonzo;Lina Kozhaya;Stephen A. Rawlings;Tamara Reyes-Robles.
Staphylococcus aureus Redirects Central Metabolism to Increase Iron Availability
David B Friedman;Devin L Stauff;Gleb Pishchany;Corbin W Whitwell.
PLOS Pathogens (2006)
A Staphylococcus aureus Regulatory System that Responds to Host Heme and Modulates Virulence
Victor J. Torres;Devin L. Stauff;Gleb Pishchany;Jelena S. Bezbradica.
Cell Host & Microbe (2007)
The Bicomponent Pore-Forming Leucocidins of Staphylococcus aureus
Francis Alonzo;Victor J. Torres.
Microbiology and Molecular Biology Reviews (2014)
Characterization of a new cytotoxin that contributes to Staphylococcus aureus pathogenesis
Ashley L. DuMont;Tyler K. Nygaard;Robert L. Watkins;Amanda Smith.
Molecular Microbiology (2011)
Staphylococcus aureus LukAB cytotoxin kills human neutrophils by targeting the CD11b subunit of the integrin Mac-1
Ashley L. DuMont;Pauline Yoong;Christopher Day;Francis Alonzo.
Proceedings of the National Academy of Sciences of the United States of America (2013)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: