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Immunology

D-Index
54
Citations
12083
World Ranking
3874
National Ranking
1775

Overview

Lloyd S. Miller is affiliated with Johnson & Johnson in the United States and has a substantial body of research primarily situated within the fields of Medicine and Immunology and Microbiology. Their work spans a variety of subfields including Immunology, Dermatology, Surgery, Infectious Diseases, and Molecular Biology. The main topics of their research focus on Dermatology and Skin Diseases, Immune Response and Inflammation, Antimicrobial Resistance in Staphylococcus, Psoriasis: Treatment and Pathogenesis, Wound Healing and Treatments, Antimicrobial Peptides and Activities, and Orthopedic Infections and Treatments.

Miller's publications are frequently found in respected research venues. The most common outlets include:

  • Journal of Investigative Dermatology
  • The Journal of Immunology
  • Journal of Clinical Investigation
  • SSRN Electronic Journal
  • Nature Communications

Their recent research contributions encompass several notable papers, such as:

  • Neutrophil extracellular trap-associated RNA and LL37 enable self-amplifying inflammation in psoriasis (2020, Nature Communications)
  • Bacteria induce skin regeneration via IL-1β signaling (2021, Cell Host & Microbe)
  • Epicutaneous Staphylococcus aureus induces IL-36 to enhance IgE production and ensuing allergic disease (2021, Journal of Clinical Investigation)
  • Tick extracellular vesicles enable arthropod feeding and promote distinct outcomes of bacterial infection (2021, Nature Communications)
  • Which Way Do We Go? Complex Interactions in Atopic Dermatitis Pathogenesis (2020, Journal of Investigative Dermatology)

Lloyd S. Miller frequently collaborates with other researchers. Their most common coauthors include:

  • Nathan K. Archer
  • Martin P. Alphonse
  • Dustin Dikeman
  • Christine Youn
  • Luis A. Garza

The breadth of Miller's research integrates clinical and molecular investigations, with particular attention to inflammatory skin disorders, microbial interactions impacting skin biology, and immune mechanisms related to disease pathogenesis. Their work in antimicrobial resistance and immune response contributes to understanding complex skin diseases and treatment strategies.

Best Publications

  • The role of the transcription factor CREB in immune function.

    Andy Y. Wen;Kathleen M. Sakamoto;Lloyd S. Miller

  • IL-17 is essential for host defense against cutaneous Staphylococcus aureus infection in mice

    John S. Cho;Eric M. Pietras;Nairy C. Garcia;Romela Irene Ramos

  • Immunity against Staphylococcus aureus cutaneous infections

    Lloyd S. Miller;John S. Cho

  • MyD88 Mediates Neutrophil Recruitment Initiated by IL-1R but Not TLR2 Activation in Immunity against Staphylococcus aureus

    Lloyd S. Miller;Ryan M. O'Connell;Miguel A. Gutierrez;Eric M. Pietras

  • Inflammasome-mediated production of IL-1β is required for neutrophil recruitment against Staphylococcus aureus in vivo

    Lloyd S. Miller;Eric M. Pietras;Lawrence H. Uricchio;Kathleen Hirano

  • A critical role for hemolysins and bacterial lipoproteins in Staphylococcus aureus-induced activation of the Nlrp3 inflammasome.

    Raúl Muñoz-Planillo;Luigi Franchi;Lloyd S. Miller;Gabriel Núñez

  • Toll-like receptors in skin.

    Lloyd S. Miller

  • Dynamics of Neutrophil Infiltration during Cutaneous Wound Healing and Infection Using Fluorescence Imaging

    Min Ho Kim;Wei Liu;Dori L Borjesson;Fitz Roy E Curry

  • IL-23 and IL-17A, but Not IL-12 and IL-22, Are Required for Optimal Skin Host Defense against Candida albicans

    Shinji Kagami;Heather L. Rizzo;Stephen E. Kurtz;Lloyd S. Miller

  • Neutrophil-derived IL-1β Is Sufficient for Abscess Formation in Immunity against Staphylococcus aureus in Mice

    John S. Cho;Yi Guo;Romela Irene Ramos;Frank Hebroni

  • Neutrophil extracellular trap-associated RNA and LL37 enable self-amplifying inflammation in psoriasis.

    Franziska Herster;Zsofia Bittner;Nathan K. Archer;Sabine Dickhöfer

  • A Mouse Model of Post-Arthroplasty Staphylococcus aureus Joint Infection to Evaluate In Vivo the Efficacy of Antimicrobial Implant Coatings

    Nicholas M. Bernthal;Alexandra I. Stavrakis;Fabrizio Billi;John S. Cho

  • Staphylococcus aureus Epicutaneous Exposure Drives Skin Inflammation via IL-36-Mediated T Cell Responses.

    Haiyun Liu;Nathan K. Archer;Carly A. Dillen;Yu Wang

  • Development of a vaccine against Staphylococcus aureus invasive infections: Evidence based on human immunity, genetics and bacterial evasion mechanisms.

    Lloyd S Miller;Vance G Fowler;Sanjay K Shukla;Sanjay K Shukla;Warren E Rose

  • TGF-α Regulates TLR Expression and Function on Epidermal Keratinocytes

    Lloyd S. Miller;Ole E. Sørensen;Philip T. Liu;H. Ray Jalian

  • Leukotriene B4 Driven Neutrophil Recruitment to the Skin Is Essential for Allergic Skin Inflammation

    Michiko K. Oyoshi;Rui He;Yitang Li;Subhanjan Mondal

  • Innate and adaptive immune responses against Staphylococcus aureus skin infections

    Sheila Krishna;Lloyd S. Miller

  • Toll-like receptors in the skin.

    Lloyd S. Miller;Robert L. Modlin

  • Host-pathogen interactions between the skin and Staphylococcus aureus.

    Sheila Krishna;Lloyd S Miller

  • Bacteria induce skin regeneration via IL-1β signaling

    Gaofeng Wang;Gaofeng Wang;Evan Sweren;Haiyun Liu;Eric Wier

  • Inflammasome-mediated production of IL-1β is required for neutrophil recruitment against Staphylococcus aureus in vivo (Journal of Immunolgy (2007) 179 (6933-6942))

    L. S. Miller;E. M. Pietras;L. H. Uricchio;K. Hirano

  • Human keratinocytes express functional toll-like receptor 3, 4, 5, and 9. Commentary

    Lloyd S. Miller;Robert L. Modlin;Maria C. Lebre;Angelic M. G. Van Der Aar

  • IL-17 is essential for host defense against cutaneous Staphylococcus aureus infection

    John Cho;Eric Pietras;Nairy Garcia;Romela Irene Ramos

Frequent Co-Authors

Scott I. Simon
Scott I. Simon University of California, Davis
Ambrose L. Cheung
Ambrose L. Cheung Dartmouth College
Robert L. Modlin
Robert L. Modlin University of California, Los Angeles
Raif S. Geha
Raif S. Geha Boston Children's Hospital
Genhong Cheng
Genhong Cheng University of California, Los Angeles
Yibin Wang
Yibin Wang University of California, Los Angeles
Yoichiro Iwakura
Yoichiro Iwakura Tokyo University of Science
Scott G. Filler
Scott G. Filler University of California, Los Angeles
Michael R. Yeaman
Michael R. Yeaman University of California, Los Angeles
Hai-Quan Mao
Hai-Quan Mao Johns Hopkins University

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