2007 - Fellow of the American Association for the Advancement of Science (AAAS)
Ambrose L. Cheung focuses on Staphylococcus aureus, Microbiology, Immunology, Staphylococcal infections and Immunity. Ambrose L. Cheung is studying Vancomycin, which is a component of Staphylococcus aureus. His Microbiology research integrates issues from Bacteria, Biofilm, Mutant, Intracellular and Virulence.
His Immunology study incorporates themes from Human pathogen, Cell sorting, Adenosine triphosphate and Escherichia coli. His Staphylococcal infections study combines topics from a wide range of disciplines, such as Glycopeptide antibiotic, Phenotype, Antibacterial agent and Polymyxin B. The concepts of his Immunity study are interwoven with issues in Receptor, TLR2 and Innate immune system.
Ambrose L. Cheung mainly focuses on Microbiology, Staphylococcus aureus, Virulence, Mutant and Gene. His Microbiology study integrates concerns from other disciplines, such as Bacteria, Biofilm, RNAIII and Sigma factor. He has researched Staphylococcus aureus in several fields, including Bacterial adhesin, Immunology and In vivo.
His work deals with themes such as Salicylic acid, Pathogen, Regulator and Internalization, which intersect with Virulence. His studies deal with areas such as Molecular biology, Regulation of gene expression and Transcription, Repressor as well as Mutant. Ambrose L. Cheung has included themes like Promoter, Transcription factor and Locus in his Transcription study.
His primary scientific interests are in Staphylococcus aureus, Microbiology, Virulence, Antibiotics and Mutant. His research in Staphylococcus aureus intersects with topics in Immune system, Immunology, Gene and Regulon. His Microbiology study focuses on Antimicrobial in particular.
His work carried out in the field of Antibiotics brings together such families of science as Escherichia coli, Multidrug tolerance, Adenosine triphosphate and Bacteria. His Mutant research includes themes of Extracellular, Effector, Cell biology and Regulation of gene expression. His Regulation of gene expression study combines topics in areas such as Promoter, Gene expression, Transcriptional regulation, Staphyloxanthin and Open reading frame.
Staphylococcus aureus, Microbiology, Antibiotics, Virulence and Regulon are his primary areas of study. Staphylococcus aureus is a primary field of his research addressed under Bacteria. His Bacteria research is multidisciplinary, relying on both Cell sorting, Intracellular, Adenosine triphosphate and Escherichia coli.
The various areas that Ambrose L. Cheung examines in his Microbiology study include Mutation, Extracellular, Biochemistry, Mutant and Immune system. His research on Antibiotics frequently connects to adjacent areas such as Immunology. The study incorporates disciplines such as Regulator, Phenotype and Daptomycin in addition to Transcription.
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Regulation of virulence determinants in vitro and in vivo in Staphylococcus aureus
Ambrose L Cheung;Arnold S Bayer;Arnold S Bayer;Gongyi Zhang;Hattie Gresham.
Fems Immunology and Medical Microbiology (2004)
IL-17 is essential for host defense against cutaneous Staphylococcus aureus infection in mice
John S. Cho;Eric M. Pietras;Nairy C. Garcia;Romela Irene Ramos.
Journal of Clinical Investigation (2010)
Survival of Staphylococcus aureus inside neutrophils contributes to infection.
Hattie D. Gresham;Jon H. Lowrance;Tony E. Caver;Bridget S. Wilson.
Journal of Immunology (2000)
Staphylococcus aureus protein A induces airway epithelial inflammatory responses by activating TNFR1
Marisa I Gómez;Aram Lee;Bharat Reddy;Amanda Muir.
Nature Medicine (2004)
MyD88 Mediates Neutrophil Recruitment Initiated by IL-1R but Not TLR2 Activation in Immunity against Staphylococcus aureus
Lloyd S. Miller;Ryan M. O'Connell;Miguel A. Gutierrez;Eric M. Pietras.
Immunity (2006)
Inflammasome-mediated production of IL-1β is required for neutrophil recruitment against Staphylococcus aureus in vivo
Lloyd S. Miller;Eric M. Pietras;Lawrence H. Uricchio;Kathleen Hirano.
Journal of Immunology (2007)
Persister formation in Staphylococcus aureus is associated with ATP depletion.
Brian P. Conlon;Sarah E. Rowe;Autumn Brown Gandt;Austin S. Nuxoll.
Nature microbiology (2016)
Interconnections between Sigma B, agr, and proteolytic activity in Staphylococcus aureus biofilm maturation
Katherine J. Lauderdale;Blaise R. Boles;Ambrose L. Cheung;Alexander R. Horswill.
Infection and Immunity (2009)
Heparin Stimulates Staphylococcus aureus Biofilm Formation
Robert M. Q. Shanks;Niles P. Donegan;Martha L. Graber;Sarah E. Buckingham.
Infection and Immunity (2005)
Molecular basis of resistance to muramidase and cationic antimicrobial peptide activity of lysozyme in staphylococci.
Silvia Herbert;Agnieszka Bera;Christiane Nerz;Dirk Kraus.
PLOS Pathogens (2007)
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