What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Immune system
Cell biology, Integrin, CD18, Cell adhesion and Selectin are his primary areas of study.
Scott I. Simon has included themes like Intercellular Adhesion Molecule-1, Immunology and Shear stress in his Integrin study.
His research in CD18 intersects with topics in Adhesion, Whole blood, Chemotaxis, Cell adhesion molecule and Monocyte.
The concepts of his Adhesion study are interwoven with issues in Biophysics, Shear flow and Nanotechnology.
In most of his Cell adhesion studies, his work intersects topics such as Endothelium.
Scott I. Simon has researched Selectin in several fields, including Protein kinase A, Molecular biology, E-selectin and L-selectin.
His most cited work include:
- Relative Contribution of LFA-1 and Mac-1 to Neutrophil Adhesion and Migration (351 citations)
- Neutrophil transendothelial migration is independent of tight junctions and occurs preferentially at tricellular corners. (255 citations)
- Molecular mechanics and dynamics of leukocyte recruitment during inflammation. (240 citations)
What are the main themes of his work throughout his whole career to date?
Scott I. Simon mostly deals with Cell biology, Immunology, Integrin, Cell adhesion and Biophysics.
His work carried out in the field of Cell biology brings together such families of science as Endothelium and CD18.
His research investigates the connection between CD18 and topics such as Chemotaxis that intersect with problems in Flow cytometry.
In his work, Haematopoiesis is strongly intertwined with Progenitor cell, which is a subfield of Immunology.
His Integrin research is multidisciplinary, incorporating perspectives in Molecular biology, Signal transduction and ICAM-1.
His Cell adhesion study is concerned with Adhesion in general.
He most often published in these fields:
- Cell biology (37.39%)
- Immunology (30.43%)
- Integrin (24.35%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (37.39%)
- Immunology (30.43%)
- Inflammation (16.52%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Cell biology, Immunology, Inflammation, Integrin and Endothelium.
His Cell biology research focuses on Cell adhesion and how it connects with Downregulation and upregulation.
His Immunology study incorporates themes from Lymph and CD11c.
Scott I. Simon interconnects Wound healing, Endothelial dysfunction, Endoplasmic reticulum, Intracellular and Thrombin in the investigation of issues within Inflammation.
The Endothelium study combines topics in areas such as Atrial natriuretic peptide, Glycocalyx and Vascular permeability.
Scott I. Simon focuses mostly in the field of Adhesion, narrowing it down to topics relating to CD18 and, in certain cases, Cytoskeleton.
Between 2013 and 2021, his most popular works were:
- Mechanosensing at the Vascular Interface (102 citations)
- Catecholamine stress alters neutrophil trafficking and impairs wound healing by β2-adrenergic receptor-mediated upregulation of IL-6. (59 citations)
- Foamy Monocytes Form Early and Contribute to Nascent Atherosclerosis in Mice With Hypercholesterolemia (45 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Inflammation
Scott I. Simon focuses on Immunology, Cell biology, Inflammation, Tumor necrosis factor alpha and Endothelium.
The study incorporates disciplines such as Mesenchymal stem cell and Lymph in addition to Immunology.
His Cell biology research incorporates themes from Receptor, Integrin and Voltage-dependent calcium channel.
The Integrin study which covers Cell adhesion that intersects with Selectin.
His Inflammation study deals with Monocyte intersecting with CD11c and Endocrinology.
His Tumor necrosis factor alpha study integrates concerns from other disciplines, such as T cell and Immunity.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
