Yibin Wang mostly deals with Internal medicine, Cell biology, Endocrinology, Signal transduction and Kinase. His study in Heart failure, Muscle hypertrophy, Myocyte, Phospholamban and Contractility are all subfields of Internal medicine. His Heart failure study combines topics from a wide range of disciplines, such as Catabolism, Metabolism and Cardiac muscle.
His Cell biology research is multidisciplinary, incorporating perspectives in Cytoprotection and Apoptosis. The Endocrinology study combines topics in areas such as Endoplasmic reticulum and Downregulation and upregulation. His Signal transduction study incorporates themes from Cancer research and Protein kinase A, Phosphorylation.
Yibin Wang mainly investigates Internal medicine, Cell biology, Endocrinology, Heart failure and Signal transduction. His Internal medicine study frequently draws connections to other fields, such as Cardiology. His Cell biology research includes themes of Chromatin, Molecular biology and Gene expression.
He interconnects Endoplasmic reticulum, Phospholamban, Ventricular remodeling and Protein kinase A in the investigation of issues within Endocrinology. His Heart failure research integrates issues from Bioinformatics, Creatine kinase, Myocardial infarction, Contractility and In vivo. His p38 mitogen-activated protein kinases study integrates concerns from other disciplines, such as Mitogen-activated protein kinase and Apoptosis.
Yibin Wang mainly focuses on Internal medicine, Heart failure, Cell biology, Endocrinology and 2019-20 coronavirus outbreak. His biological study spans a wide range of topics, including Type 2 diabetes and Cardiology. His Heart failure research is multidisciplinary, incorporating elements of Transcriptome, Myocardial infarction and Diastole.
The concepts of his Cell biology study are interwoven with issues in RNA splicing, RBFOX1 and Gene. His Endocrinology research incorporates elements of Extracellular matrix and Matrix metalloproteinase. Yibin Wang focuses mostly in the field of Stem cell, narrowing it down to matters related to Knockout mouse and, in some cases, p38 mitogen-activated protein kinases, Cell growth, mTORC1 and Signal transduction.
His primary areas of investigation include Internal medicine, 2019-20 coronavirus outbreak, Severe acute respiratory syndrome coronavirus 2, Angiotensin II Receptor Blockers and Heart failure. Yibin Wang regularly links together related areas like Endocrinology in his Internal medicine studies. His Endocrinology research incorporates elements of Area under the curve and Valine.
In his work, Discontinuation, Blood pressure control, ACE - Angiotensin-converting enzyme, Pneumonia and Renin–angiotensin system is strongly intertwined with Pharmacology, which is a subfield of Angiotensin II Receptor Blockers. His Heart failure research includes themes of Cohort and Patient population. The various areas that Yibin Wang examines in his Cardiology study include Phenotype, Western diet and Disease, Cardio metabolic.
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Association of Inpatient Use of Angiotensin-Converting Enzyme Inhibitors and Angiotensin II Receptor Blockers With Mortality Among Patients With Hypertension Hospitalized With COVID-19.
Peng Zhang;Lihua Zhu;Jingjing Cai;Fang Lei.
Circulation Research (2020)
Cardiac Muscle Cell Hypertrophy and Apoptosis Induced by Distinct Members of the p38 Mitogen-activated Protein Kinase Family
Yibin Wang;Shuang Huang;Valerie P. Sah;John Ross.
Journal of Biological Chemistry (1998)
Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy
Eiki Takimoto;Hunter C Champion;Manxiang Li;Diego Belardi.
Nature Medicine (2005)
Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.
Beth A. Rose;Thomas Force;Yibin Wang.
Physiological Reviews (2010)
Enhanced Gαq signaling: A common pathway mediates cardiac hypertrophy and apoptotic heart failure
John W. Adams;Yoshihito Sakata;Michael G. Davis;Valerie P. Sah.
Proceedings of the National Academy of Sciences of the United States of America (1998)
p38 MAP kinase inhibition enables proliferation of adult mammalian cardiomyocytes
Felix B. Engel;Michael Schebesta;Mychelle T. Duong;Gang Lu.
Genes & Development (2005)
Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy.
Susumu Minamisawa;Masahiko Hoshijima;Guoxiang Chu;Christopher A Ward.
Cell (1999)
Oxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load.
Eiki Takimoto;Hunter C. Champion;Manxiang Li;Shuxun Ren.
Journal of Clinical Investigation (2005)
Sustained activation of JNK/p38 MAPK pathways in response to cisplatin leads to Fas ligand induction and cell death in ovarian carcinoma cells.
Abdellah Mansouri;Lon D. Ridgway;Anita L. Korapati;Qingxiu Zhang.
Journal of Biological Chemistry (2003)
Growth retardation and cysteine deficiency in gamma-glutamyl transpeptidase-deficient mice
M W Lieberman;A L Wiseman;Z Z Shi;B Z Carter.
Proceedings of the National Academy of Sciences of the United States of America (1996)
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