What is he best known for?
The fields of study he is best known for:
- Gene
- Signal transduction
- Internal medicine
His primary scientific interests are in Internal medicine, Kinase, Cell biology, Endocrinology and Protein kinase A.
His Internal medicine study combines topics in areas such as Protein turnover and Cardiology.
The concepts of his Kinase study are interwoven with issues in Molecular biology and Phosphorylation.
His research integrates issues of Apoptosis and Cardiac muscle in his study of Cell biology.
In general Endocrinology study, his work on Myocyte often relates to the realm of Organism, thereby connecting several areas of interest.
His study in Protein kinase A is interdisciplinary in nature, drawing from both Mitogen-activated protein kinase, Protein kinase C and MAPK/ERK pathway.
His most cited work include:
- Stimulation of the Stress-Activated Mitogen-Activated Protein Kinase Subfamilies in Perfused Heart: p38/RK Mitogen-Activated Protein Kinases and c-Jun N-Terminal Kinases Are Activated by Ischemia/Reperfusion (494 citations)
- “Stress-Responsive” Mitogen-Activated Protein Kinases (c-Jun N-Terminal Kinases and p38 Mitogen-Activated Protein Kinases) in the Myocardium (465 citations)
- Cellular mechanisms of cardiac hypertrophy (374 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Internal medicine, Endocrinology, Cell biology, Kinase and Protein kinase A.
Internal medicine is frequently linked to Cardiology in his study.
His research in Endocrinology intersects with topics in Endothelin 1 and Protein biosynthesis.
He has researched Kinase in several fields, including Molecular biology and Phosphorylation.
His biological study spans a wide range of topics, including Small G Protein, G protein-coupled receptor and Angiotensin II.
His Mitogen-activated protein kinase kinase research is multidisciplinary, incorporating perspectives in MAP2K7 and MAP kinase kinase kinase.
He most often published in these fields:
- Internal medicine (47.13%)
- Endocrinology (43.10%)
- Cell biology (37.36%)
What were the highlights of his more recent work (between 2008-2021)?
- Kinase (35.63%)
- Internal medicine (47.13%)
- Cell biology (37.36%)
In recent papers he was focusing on the following fields of study:
Peter H. Sugden mainly focuses on Kinase, Internal medicine, Cell biology, Endocrinology and Receptor.
Peter H. Sugden works in the field of Kinase, namely Mitogen-activated protein kinase.
His research combines Cardiology and Internal medicine.
His studies deal with areas such as Immediate early gene and Endothelin 1 as well as Cell biology.
His work deals with themes such as Transgene and MAPK/ERK pathway, which intersect with Endocrinology.
His study in the field of c-Raf and MAP2K7 also crosses realms of MST1.
Between 2008 and 2021, his most popular works were:
- Cardiovascular side effects of cancer therapies: a position statement from the Heart Failure Association of the European Society of Cardiology. (296 citations)
- Conditional Transgenic Expression of Fibroblast Growth Factor 9 in the Adult Mouse Heart Reduces Heart Failure Mortality After Myocardial Infarction (47 citations)
- Towards a re-definition of 'cardiac hypertrophy' through a rational characterization of left ventricular phenotypes : a position paper of the Working Group 'Myocardial Function' of the ESC (43 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Signal transduction
- Internal medicine
His main research concerns Internal medicine, Cell biology, Heart failure, Protein kinase A and Kinase.
Peter H. Sugden has included themes like Pharmaceutical sciences and Endocrinology in his Internal medicine study.
His study in the field of Tumor necrosis factor alpha is also linked to topics like Proinflammatory cytokine.
Peter H. Sugden regularly links together related areas like Molecular biology in his Cell biology studies.
His c-Raf and MAP2K7 study, which is part of a larger body of work in Protein kinase A, is frequently linked to MST1, bridging the gap between disciplines.
His work on MAPK/ERK pathway, p38 mitogen-activated protein kinases and GSK-3 as part of general Kinase study is frequently connected to Transgenesis, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
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