Endocrinology, Internal medicine, Leptin, Adipose tissue and Receptor are his primary areas of study. As part of his studies on Endocrinology, Bradford B. Lowell often connects relevant areas like Transgene. His study in Insulin, Adipocyte, Insulin resistance, Melanocortin and Arcuate nucleus falls under the purview of Internal medicine.
Bradford B. Lowell focuses mostly in the field of Leptin, narrowing it down to topics relating to Melanin-concentrating hormone receptor and, in certain cases, Glucose tolerance test, Lateral hypothalamus and Orexigenic. His work in Adipose tissue addresses issues such as Genetically modified mouse, which are connected to fields such as Beta-1 adrenergic receptor, Cyclase activity, Beta-3 adrenergic receptor, Beta and Regeneration. His Receptor research includes elements of Dentate gyrus and Hormone.
The scientist’s investigation covers issues in Endocrinology, Internal medicine, Neuroscience, Hypothalamus and Receptor. His work in Leptin, Adipose tissue, Homeostasis, Insulin and Glucose homeostasis is related to Endocrinology. His Internal medicine research is multidisciplinary, relying on both Transgene and Neuron.
His Neuroscience study frequently links to related topics such as Glutamatergic. The various areas that Bradford B. Lowell examines in his Hypothalamus study include Neuropeptide, Melanocortin and Nucleus. His Biochemistry study combines topics in areas such as Cell biology and Skeletal muscle.
His primary areas of study are Neuroscience, Internal medicine, Endocrinology, Hypothalamus and Neuron. His study focuses on the intersection of Neuroscience and fields such as Glutamatergic with connections in the field of Vasodilation, Vasoconstriction and Reflex. Internal medicine connects with themes related to Nucleus in his study.
His Hypothalamus research incorporates elements of Receptor, Postsynaptic potential, Biological neural network and Optogenetics. His study looks at the relationship between Receptor and fields such as Obesity, as well as how they intersect with chemical problems. His biological study spans a wide range of topics, including Arcuate nucleus, Gene expression profiling, Orexigenic, Molecular neuroscience and Cell type.
His main research concerns Neuroscience, Hypothalamus, Arcuate nucleus, Perception and Neuron. His study on Lateral parabrachial nucleus, Median eminence and Insular cortex is often connected to Emerging technologies and Spinal circuits as part of broader study in Neuroscience. The concepts of his Hypothalamus study are interwoven with issues in Obesity, Receptor, Melanocortin, Hindbrain and Biological neural network.
The study incorporates disciplines such as Appetite regulation, Appetite, Appetite control and Cognition in addition to Arcuate nucleus. His Perception study incorporates themes from Cerebral cortex, Basolateral amygdala, Sensory cue, Neuroimaging and Satiety Response. His Neuron research is multidisciplinary, incorporating perspectives in Tanycyte, Gene expression profiling, Orexigenic, Molecular neuroscience and Cell type.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mechanisms Controlling Mitochondrial Biogenesis and Respiration through the Thermogenic Coactivator PGC-1
Zhidan Wu;Pere Puigserver;Ulf Andersson;Chenyu Zhang.
Cell (1999)
Role of leptin in the neuroendocrine response to fasting
Rexford S. Ahima;Daniel Prabakaran;Christos Mantzoros;Daqing Qu.
Nature (1996)
Transcriptional co-activator PGC-1α drives the formation of slow-twitch muscle fibres
Jiandie Lin;Hai Wu;Paul T. Tarr;Chen Yu Zhang.
Nature (2002)
Mitochondrial dysfunction and type 2 diabetes.
Bradford B. Lowell;Gerald I. Shulman.
Science (2005)
Leptin levels reflect body lipid content in mice: Evidence for diet-induced resistance to leptin action
Robert C. Frederich;Andreas Hamann;Stephen Anderson;Bettina Löllmann.
Nature Medicine (1995)
Towards a molecular understanding of adaptive thermogenesis.
Bradford B. Lowell;Bruce M. Spiegelman.
Nature (2000)
Development of obesity in transgenic mice after genetic ablation of brown adipose tissue
Bradford B. Lowell;Vedrana S-Susulic;Andreas Hamann;Joel A. Lawitts.
Nature (1993)
Mice lacking melanin-concentrating hormone are hypophagic and lean
Masako Shimada;Nicholas A. Tritos;Bradford B. Lowell;Jeffrey S. Flier.
Nature (1998)
Defects in Adaptive Energy Metabolism with CNS-Linked Hyperactivity in PGC-1α Null Mice
Jiandie Lin;Pei Hsuan Wu;Paul T. Tarr;Katrin S. Lindenberg.
Cell (2004)
Leptin action on GABAergic neurons prevents obesity and reduces inhibitory tone to POMC neurons.
Linh Vong;Chianping Ye;Zongfang Yang;Brian Choi.
Neuron (2011)
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