D-Index & Metrics Best Publications

Angela Clerk

University of Reading
United Kingdom

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Biology and Biochemistry D-index 55 Citations 10,318 134 World Ranking 10494 National Ranking 812

Overview

What is she best known for?

The fields of study Angela Clerk is best known for:

Gene
Enzyme
Phosphorylation

Angela Clerk combines Cell biology and Molecular biology in her research. Angela Clerk integrates many fields in her works, including Molecular biology and Cell biology. By researching both Kinase and MAPK/ERK pathway, Angela Clerk produces research that crosses academic boundaries. Her study brings together the fields of MAPK cascade and MAPK/ERK pathway. Her research links Mitogen-activated protein kinase with MAPK cascade. Angela Clerk carries out multidisciplinary research, doing studies in Mitogen-activated protein kinase and Phosphorylation. In her research, Angela Clerk undertakes multidisciplinary study on Phosphorylation and Transcription factor. In her research, she undertakes multidisciplinary study on Transcription factor and Kinase. Protein kinase A is closely attributed to p38 mitogen-activated protein kinases in her research.

Her most cited work include:

“Stress-Responsive” Mitogen-Activated Protein Kinases (c-Jun N-Terminal Kinases and p38 Mitogen-Activated Protein Kinases) in the Myocardium (427 citations)
Cellular mechanisms of cardiac hypertrophy (387 citations)
Endothelin-1 and fibroblast growth factors stimulate the mitogen-activated protein kinase signaling cascade in cardiac myocytes. The potential role of the cascade in the integration of two signaling pathways leading to myocyte hypertrophy. (345 citations)

What are the main themes of her work throughout her whole career to date

As part of her Gene expression and Transcription factor and Gene studies, Angela Clerk is studying Gene. She integrates Cell biology with Genetics in her research. In her research, she performs multidisciplinary study on Genetics and Cell biology. Angela Clerk integrates several fields in her works, including Kinase and Mitogen-activated protein kinase kinase. She combines Mitogen-activated protein kinase kinase and Kinase in her studies. Biochemistry and Enzyme are two areas of study in which she engages in interdisciplinary work. She performs multidisciplinary study in the fields of Enzyme and Biochemistry via her papers. Internal medicine is frequently linked to Receptor in her study. Her Receptor study frequently involves adjacent topics like Endothelin 1.

Angela Clerk most often published in these fields:

Cell biology (81.40%)
Kinase (58.14%)
Biochemistry (51.16%)

What were the highlights of her more recent work (between 2013-2021)?

Cell biology (66.67%)
Internal medicine (55.56%)
Protein kinase A (44.44%)

In recent works Angela Clerk was focusing on the following fields of study:

Her Heart failure research also covers Phospholamban and Cardiac function curve studies. Angela Clerk has begun a study into Mitogen-activated protein kinase kinase, looking into Cyclin-dependent kinase 9 and ASK1. Her research on Apoptosis is centered around Programmed cell death, PI3K/AKT/mTOR pathway and Cytoprotection. She connects Programmed cell death with Apoptosis in her research. Angela Clerk carries out multidisciplinary research, doing studies in PI3K/AKT/mTOR pathway and Phosphorylation. Angela Clerk regularly links together related areas like Cyclin-dependent kinase 9 in her Phosphorylation studies. Her Cytoprotection study frequently draws connections between related disciplines such as Biochemistry. In her study, she carries out multidisciplinary Biochemistry and Molecular biology research. She conducts interdisciplinary study in the fields of Molecular biology and Cell biology through her works.

Between 2013 and 2021, her most popular works were:

Targeting myocardial remodelling to develop novel therapies for heart failure (92 citations)
Cardiac protein kinases: the cardiomyocyte kinome and differential kinase expression in human failing hearts (41 citations)
Redox Regulation of Cardiac ASK1 (Apoptosis Signal-Regulating Kinase 1) Controls p38-MAPK (Mitogen-Activated Protein Kinase) and Orchestrates Cardiac Remodeling to Hypertension (40 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

“Stress-Responsive” Mitogen-Activated Protein Kinases (c-Jun N-Terminal Kinases and p38 Mitogen-Activated Protein Kinases) in the Myocardium

Peter H. Sugden;Angela Clerk.
Circulation Research (1998)

644 Citations

Endothelin-1 and fibroblast growth factors stimulate the mitogen-activated protein kinase signaling cascade in cardiac myocytes. The potential role of the cascade in the integration of two signaling pathways leading to myocyte hypertrophy.

M A Bogoyevitch;P E Glennon;M B Andersson;Angela Clerk.
Journal of Biological Chemistry (1994)

547 Citations

Cellular mechanisms of cardiac hypertrophy

P. H. Sugden;Angela Clerk.
Journal of Molecular Medicine (1998)

492 Citations

Stimulation of “Stress-regulated” Mitogen-activated Protein Kinases (Stress-activated Protein Kinases/c-Jun N-terminal Kinases and p38-Mitogen-activated Protein Kinases) in Perfused Rat Hearts by Oxidative and Other Stresses

Angela Clerk;Stephen J. Fuller;Ashour Michael;Peter H. Sugden.
Journal of Biological Chemistry (1998)

452 Citations

Stimulation of the p38 Mitogen-activated Protein Kinase Pathway in Neonatal Rat Ventricular Myocytes by the G Protein–coupled Receptor Agonists, Endothelin-1 and Phenylephrine: A Role in Cardiac Myocyte Hypertrophy?

Angela Clerk;Ashour Michael;Peter H. Sugden.
Journal of Cell Biology (1998)

442 Citations

Regulation of the ERK subgroup of MAP kinase cascades through G protein-coupled receptors.

Peter H Sugden;Angela Clerk.
Cellular Signalling (1997)

439 Citations

Differential activation of protein kinase C isoforms by endothelin-1 and phenylephrine and subsequent stimulation of p42 and p44 mitogen-activated protein kinases in ventricular myocytes cultured from neonatal rat hearts.

Angela Clerk;M A Bogoyevitch;M B Anderson;P H Sugden.
Journal of Biological Chemistry (1994)

391 Citations

Regulation of Bcl-2 Family Proteins During Development and in Response to Oxidative Stress in Cardiac Myocytes Association With Changes in Mitochondrial Membrane Potential

Stuart A. Cook;Peter H. Sugden;Angela Clerk.
Circulation Research (1999)

336 Citations

The p38-MAPK inhibitor, SB203580, inhibits cardiac stress-activated protein kinases/c-Jun N-terminal kinases (SAPKs/JNKs)

Angela Clerk;Peter H Sugden.
FEBS Letters (1998)

261 Citations

Stimulation of multiple mitogen-activated protein kinase sub-families by oxidative stress and phosphorylation of the small heat shock protein, HSP25/27, in neonatal ventricular myocytes

Angela Clerk;Ashour Michael;Peter H. Sugden.
Biochemical Journal (1998)

259 Citations

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