What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Cell biology, Molecular biology, Nucleosome, Histone and DNA.
His research in Cell biology tackles topics such as Chromatin which are related to areas like Epigenetics and Biophysics.
Hitoshi Kurumizaka interconnects Ubiquitin, Mutation, FANCD2 and RAD51, DNA repair in the investigation of issues within Molecular biology.
His Nucleosome study is focused on Genetics in general.
The concepts of his Histone study are interwoven with issues in Spermiogenesis and Transformation.
His DNA research includes themes of C-terminus, Mutant and DNA-binding protein.
His most cited work include:
- Crystal structure of the human centromeric nucleosome containing CENP-A (283 citations)
- Independent and sequential recruitment of NHEJ and HR factors to DNA damage sites in mammalian cells. (213 citations)
- Histone acetylation: influence on transcription, nucleosome mobility and positioning, and linker histone-dependent transcriptional repression. (212 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Cell biology, Nucleosome, Chromatin, Histone and DNA.
His research in Cell biology intersects with topics in Histone octamer, DNA damage, Histone H2A, Molecular biology and DNA repair.
His biological study spans a wide range of topics, including Biophysics and Histone H3.
His Chromatin study which covers Transcription that intersects with RNA polymerase II.
His Histone study integrates concerns from other disciplines, such as Acetylation, Genome, Protein structure, Regulation of gene expression and Epigenetics.
The study incorporates disciplines such as Homologous chromosome and Mutant in addition to DNA.
He most often published in these fields:
- Cell biology (54.37%)
- Nucleosome (56.28%)
- Chromatin (47.27%)
What were the highlights of his more recent work (between 2018-2021)?
- Chromatin (47.27%)
- Nucleosome (56.28%)
- Cell biology (54.37%)
In recent papers he was focusing on the following fields of study:
His main research concerns Chromatin, Nucleosome, Cell biology, Histone and DNA.
His Chromatin study combines topics in areas such as Chromatin immunoprecipitation, Transcription, Epigenetics and Mutant.
His work deals with themes such as Computational biology and DNA damage, which intersect with Nucleosome.
His work carried out in the field of Cell biology brings together such families of science as Histone H3, RNA, Transcription factor, Gene and Centromere.
He combines subjects such as Acetylation, Biophysics, Genome, Regulation of gene expression and DNA replication with his study of Histone.
His studies examine the connections between DNA and genetics, as well as such issues in Protein structure, with regards to DNA binding site, Processivity, Guanosine and ATP synthase.
Between 2018 and 2021, his most popular works were:
- A chromatin integration labelling method enables epigenomic profiling with lower input. (53 citations)
- A chromatin integration labelling method enables epigenomic profiling with lower input. (53 citations)
- Structural insight into nucleosome transcription by RNA polymerase II with elongation factors. (50 citations)
In his most recent research, the most cited papers focused on:
Hitoshi Kurumizaka mainly investigates Cell biology, Chromatin, Nucleosome, DNA and Histone.
In Cell biology, Hitoshi Kurumizaka works on issues like Histone H3, which are connected to Centromere and Chimera.
The various areas that Hitoshi Kurumizaka examines in his Chromatin study include Scattering, Chromatin immunoprecipitation, Transcription factor and Transcription.
His studies deal with areas such as Chemical physics, Small-angle X-ray scattering, Crystal structure and Neutron scattering as well as Nucleosome.
DNA binding site, Xenopus, Guanosine and ATP synthase is closely connected to Protein structure in his research, which is encompassed under the umbrella topic of DNA.
His Histone research includes elements of Pyrimidine dimer, DNA damage, DNA repair, Epigenomics and DNA replication.
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