Member of the Association of American Physicians
Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Cell biology, Molecular biology and Endocrinology are his primary areas of study. His research in Cystic fibrosis transmembrane conductance regulator intersects with topics in Integral membrane protein, Chloride channel, Potentiator and Phosphorylation. His biological study spans a wide range of topics, including Mutant, Immunology, Bioinformatics and Pathology.
He interconnects Exocytosis and Endocytosis in the investigation of issues within Cell biology. His Molecular biology research integrates issues from Purine nucleoside phosphorylase, Cell culture, Cell and Gene delivery. His Endocrinology research includes elements of Epithelium, Internal medicine and Knockout rat.
His primary scientific interests are in Cystic fibrosis transmembrane conductance regulator, Cystic fibrosis, Molecular biology, Cell biology and Biochemistry. Cystic fibrosis transmembrane conductance regulator is a subfield of Internal medicine that Eric J. Sorscher investigates. Eric J. Sorscher has researched Cystic fibrosis in several fields, including Bioinformatics, Mutation, Immunology, Pathology and Lung.
His Molecular biology research includes themes of Cell culture, Transfection, Cell, Mutant and Genetic enhancement. Eric J. Sorscher has included themes like Protein structure, Integral membrane protein and Endocytosis in his Cell biology study. In his research on the topic of Mucociliary clearance, In vivo and Transepithelial chloride transport is strongly related with Pharmacology.
His scientific interests lie mostly in Cystic fibrosis, Cystic fibrosis transmembrane conductance regulator, Cell biology, Cancer research and Ivacaftor. His Cystic fibrosis research is multidisciplinary, incorporating elements of Precision medicine, Function, Intensive care medicine, Muscle hypertrophy and Lung disease. His Cystic fibrosis transmembrane conductance regulator study combines topics from a wide range of disciplines, such as Chloride channel, Bioinformatics and Missense mutation.
His studies in Cell biology integrate themes in fields like Protein structure, Oxidative stress, Patch clamp and Transmembrane protein. In Cancer research, he works on issues like Purine nucleoside phosphorylase, which are connected to Adenosine, Molecular biology and Escherichia coli. The various areas that Eric J. Sorscher examines in his Ivacaftor study include Mutant, Potentiator, Disease, Pathology and Rat model.
Eric J. Sorscher mainly focuses on Cystic fibrosis, Lumacaftor, Ivacaftor, Precision medicine and Bioinformatics. Cystic fibrosis is the subject of his research, which falls under Internal medicine. His Lumacaftor research is multidisciplinary, relying on both Ribosome and Transmembrane protein.
Eric J. Sorscher combines subjects such as Disease, Mutant and Potentiator with his study of Ivacaftor. The study incorporates disciplines such as Gating, Long-term potentiation, Wild type and Phosphorylation, Cell biology in addition to Disease. The concepts of his Bioinformatics study are interwoven with issues in Cftr modulator, Respiratory tree and Cystic fibrosis transmembrane conductance regulator.
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Characterization and Dynamics of Aggresome Formation by a Cytosolic Gfp-Chimera✪
Rafael García-Mata;Zsuzsa Bebök;Eric J. Sorscher;Elizabeth S. Sztul.
Journal of Cell Biology (1999)
Regulation of plasma membrane recycling by CFTR
Neil A. Bradbury;Tamas Jilling;Gabor Berta;Eric J. Sorscher.
From CFTR biology toward combinatorial pharmacotherapy: expanded classification of cystic fibrosis mutations.
Gudio Veit;Radu G. Avramescu;Annette N. Chiang;Scott A. Houck.
Molecular Biology of the Cell (2016)
Evidence that Systemic Gentamicin Suppresses Premature Stop Mutations in Patients with Cystic Fibrosis
J. P. Clancy;Zsuzsa Bebök;Fadel Ruiz;Chris King.
American Journal of Respiratory and Critical Care Medicine (2001)
Reduced histone deacetylase 7 activity restores function to misfolded CFTR in cystic fibrosis
Darren M Hutt;David Herman;Ana P C Rodrigues;Sabrina Noel.
Nature Chemical Biology (2010)
A macromolecular complex of β2 adrenergic receptor, CFTR, and ezrin/radixin/moesin-binding phosphoprotein 50 is regulated by PKA
Anjaparavanda P. Naren;Bryan Cobb;Chunying Li;Koushik Roy.
Proceedings of the National Academy of Sciences of the United States of America (2003)
Expression and polarized targeting of a rab3 isoform in epithelial cells.
E Weber;G Berta;A Tousson;P St John.
Journal of Cell Biology (1994)
Tumor cell bystander killing in colonic carcinoma utilizing the Escherichia coli DeoD gene to generate toxic purines.
Sorscher Ej;Peng S;Bebok Z;Allan Pw.
Gene Therapy (1994)
Aminoglycoside suppression of a premature stop mutation in a Cftr-/- mouse carrying a human CFTR-G542X transgene.
Ming Du;Julie R. Jones;Jessica Lanier;Kim M. Keeling.
Journal of Molecular Medicine (2002)
Failure of cAMP agonists to activate rescued ΔF508 CFTR in CFBE41o– airway epithelial monolayers
Zsuzsa Bebok;James F. Collawn;John Wakefield;William Parker.
The Journal of Physiology (2005)
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