D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 69 Citations 21,036 145 World Ranking 4608 National Ranking 2291

Research.com Recognitions

Awards & Achievements

1995 - Fellow of the American Academy of Microbiology Molecular, Cellular and General Biology of Eukaryotes

1994 - Fellow of the American Association for the Advancement of Science (AAAS)

1993 - Member of the National Academy of Medicine (NAM)

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • DNA

The scientist’s investigation covers issues in Growth factor, Platelet-derived growth factor, Platelet-derived growth factor receptor, Molecular biology and Immunology. His work carried out in the field of Growth factor brings together such families of science as Wound healing, Fibroblast and Transforming growth factor, Endocrinology. Thomas F. Deuel focuses mostly in the field of Platelet-derived growth factor, narrowing it down to topics relating to Epidermal growth factor and, in certain cases, Protamine sulfate and Basic fibroblast growth factor.

His Platelet-derived growth factor receptor study combines topics in areas such as Autocrine signalling and Cell biology. Thomas F. Deuel interconnects Amino acid, Pleiotrophin, Platelet, Peptide sequence and Phosphorylation in the investigation of issues within Molecular biology. As a part of the same scientific family, he mostly works in the field of Internal medicine, focusing on Cellular differentiation and, on occasion, In situ hybridization, Nervous system and Neuron.

His most cited work include:

  • Platelet-derived growth factor is structurally related to the putative transforming protein p28sis of simian sarcoma virus. (1370 citations)
  • Accelerated healing of incisional wounds in rats induced by transforming growth factor-beta. (856 citations)
  • Platelet-derived growth factor and transforming growth factor-beta enhance tissue repair activities by unique mechanisms. (497 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Molecular biology, Platelet-derived growth factor receptor, Platelet-derived growth factor, Growth factor and Pleiotrophin. His Molecular biology research includes elements of Promoter, 3T3 cells, Gene, Phosphorylation and Gene product. His research integrates issues of Epidermal growth factor, Autocrine signalling, Monocyte, Cell surface receptor and Cell biology in his study of Platelet-derived growth factor receptor.

In his research on the topic of Platelet-derived growth factor, Transforming growth factor is strongly related with Wound healing. His biological study spans a wide range of topics, including Fibroblast, Endocrinology and Chemotaxis. His Pleiotrophin research is multidisciplinary, incorporating elements of Cancer research, Angiogenesis, Gene expression, Protein tyrosine phosphatase and Midkine.

He most often published in these fields:

  • Molecular biology (48.70%)
  • Platelet-derived growth factor receptor (41.56%)
  • Platelet-derived growth factor (35.06%)

What were the highlights of his more recent work (between 2003-2017)?

  • Pleiotrophin (29.22%)
  • Cancer research (16.88%)
  • Internal medicine (20.13%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Pleiotrophin, Cancer research, Internal medicine, Endocrinology and Angiogenesis. The various areas that Thomas F. Deuel examines in his Pleiotrophin study include Gene expression, Protein tyrosine phosphatase, Midkine, Molecular biology and Angiotensin II. His Molecular biology study combines topics in areas such as Bone morphogenetic protein 4 and Bone morphogenetic protein 2.

His work carried out in the field of Endocrinology brings together such families of science as Alpha and Growth factor. Thomas F. Deuel has included themes like Endothelial stem cell, Transdifferentiation and Immunology in his Angiogenesis study. His study in Transdifferentiation is interdisciplinary in nature, drawing from both MMP2, Platelet-derived growth factor and Neovascularization.

Between 2003 and 2017, his most popular works were:

  • Identification, cloning, and expression of human estrogen receptor-α36, a novel variant of human estrogen receptor-α66 (298 citations)
  • A variant of estrogen receptor-α, hER-α36: Transduction of estrogen- and antiestrogen-dependent membrane-initiated mitogenic signaling (296 citations)
  • Anaplastic lymphoma kinase is activated through the pleiotrophin/receptor protein-tyrosine phosphatase β/ζ signaling pathway : An alternative mechanism of receptor tyrosine kinase activation (115 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

His scientific interests lie mostly in Pleiotrophin, Protein tyrosine phosphatase, Cancer research, Signal transduction and Tyrosine phosphorylation. The study incorporates disciplines such as Angiogenesis and Cell biology in addition to Pleiotrophin. His work deals with themes such as Vasculogenesis, Cytokine and Pathology, which intersect with Cancer research.

The concepts of his Signal transduction study are interwoven with issues in Estrogen and Transactivation. His research investigates the connection between Tyrosine phosphorylation and topics such as Receptor tyrosine kinase that intersect with problems in Molecular biology and Tyrosine kinase. Thomas F. Deuel has researched Immunology in several fields, including Neovascularization, MMP2, Platelet-derived growth factor, Transdifferentiation and In vivo.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Platelet-derived growth factor is structurally related to the putative transforming protein p28sis of simian sarcoma virus.

Michael D. Waterfield;Geoffrey T. Scrace;Nigel Whittle;Paul Stroobant.
Nature (1983)

1959 Citations

Accelerated healing of incisional wounds in rats induced by transforming growth factor-beta.

Thomas A. Mustoe;Glenn F. Pierce;Arlen Thomason;Peggy Gramates.
Science (1987)

1407 Citations

Platelet-derived growth factor and transforming growth factor-beta enhance tissue repair activities by unique mechanisms.

G F Pierce;T A Mustoe;J Lingelbach;V R Masakowski.
Journal of Cell Biology (1989)

801 Citations

Chemotaxis of monocytes and neutrophils to platelet-derived growth factor.

T F Deuel;R M Senior;J S Huang;G L Griffin.
Journal of Clinical Investigation (1982)

757 Citations

Role of platelet-derived growth factor in wound healing.

Glenn F. Pierce;Thomas A. Mustoe;Bruce W. Altrock;Thomas F. Deuel.
Journal of Cellular Biochemistry (1991)

608 Citations

Platelet Factor 4 Is Chemotactic for Neutrophils and Monocytes

Thomas F. Deuel;Robert M. Senior;Donald Chang;Gail L. Griffin.
Proceedings of the National Academy of Sciences of the United States of America (1981)

605 Citations

Cloning and expression of a developmentally regulated protein that induces mitogenic and neurite outgrowth activity

Yue-Sheng Li;Peter G. Milner;Anil K. Chauhan;Mark A. Watson.
Science (1990)

602 Citations

Polypeptide Growth Factors: Roles in Normal and Abnormal Cell Growth

Thomas F. Deuel.
Annual Review of Cell Biology (1987)

517 Citations

Growth factor-induced acceleration of tissue repair through direct and inductive activities in a rabbit dermal ulcer model.

T A Mustoe;G F Pierce;C Morishima;T F Deuel.
Journal of Clinical Investigation (1991)

501 Citations

Pleiotrophin signals increased tyrosine phosphorylation of β-catenin through inactivation of the intrinsic catalytic activity of the receptor-type protein tyrosine phosphatase β/ζ

Kung Meng;A. Rodríguez-Peña;Todor Dimitrov;Wen Chen.
Proceedings of the National Academy of Sciences of the United States of America (2000)

492 Citations

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