1995 - Fellow of the American Academy of Microbiology Molecular, Cellular and General Biology of Eukaryotes
1994 - Fellow of the American Association for the Advancement of Science (AAAS)
1993 - Member of the National Academy of Medicine (NAM)
Member of the Association of American Physicians
The scientist’s investigation covers issues in Growth factor, Platelet-derived growth factor, Platelet-derived growth factor receptor, Molecular biology and Immunology. His work carried out in the field of Growth factor brings together such families of science as Wound healing, Fibroblast and Transforming growth factor, Endocrinology. Thomas F. Deuel focuses mostly in the field of Platelet-derived growth factor, narrowing it down to topics relating to Epidermal growth factor and, in certain cases, Protamine sulfate and Basic fibroblast growth factor.
His Platelet-derived growth factor receptor study combines topics in areas such as Autocrine signalling and Cell biology. Thomas F. Deuel interconnects Amino acid, Pleiotrophin, Platelet, Peptide sequence and Phosphorylation in the investigation of issues within Molecular biology. As a part of the same scientific family, he mostly works in the field of Internal medicine, focusing on Cellular differentiation and, on occasion, In situ hybridization, Nervous system and Neuron.
His primary scientific interests are in Molecular biology, Platelet-derived growth factor receptor, Platelet-derived growth factor, Growth factor and Pleiotrophin. His Molecular biology research includes elements of Promoter, 3T3 cells, Gene, Phosphorylation and Gene product. His research integrates issues of Epidermal growth factor, Autocrine signalling, Monocyte, Cell surface receptor and Cell biology in his study of Platelet-derived growth factor receptor.
In his research on the topic of Platelet-derived growth factor, Transforming growth factor is strongly related with Wound healing. His biological study spans a wide range of topics, including Fibroblast, Endocrinology and Chemotaxis. His Pleiotrophin research is multidisciplinary, incorporating elements of Cancer research, Angiogenesis, Gene expression, Protein tyrosine phosphatase and Midkine.
His primary areas of study are Pleiotrophin, Cancer research, Internal medicine, Endocrinology and Angiogenesis. The various areas that Thomas F. Deuel examines in his Pleiotrophin study include Gene expression, Protein tyrosine phosphatase, Midkine, Molecular biology and Angiotensin II. His Molecular biology study combines topics in areas such as Bone morphogenetic protein 4 and Bone morphogenetic protein 2.
His work carried out in the field of Endocrinology brings together such families of science as Alpha and Growth factor. Thomas F. Deuel has included themes like Endothelial stem cell, Transdifferentiation and Immunology in his Angiogenesis study. His study in Transdifferentiation is interdisciplinary in nature, drawing from both MMP2, Platelet-derived growth factor and Neovascularization.
His scientific interests lie mostly in Pleiotrophin, Protein tyrosine phosphatase, Cancer research, Signal transduction and Tyrosine phosphorylation. The study incorporates disciplines such as Angiogenesis and Cell biology in addition to Pleiotrophin. His work deals with themes such as Vasculogenesis, Cytokine and Pathology, which intersect with Cancer research.
The concepts of his Signal transduction study are interwoven with issues in Estrogen and Transactivation. His research investigates the connection between Tyrosine phosphorylation and topics such as Receptor tyrosine kinase that intersect with problems in Molecular biology and Tyrosine kinase. Thomas F. Deuel has researched Immunology in several fields, including Neovascularization, MMP2, Platelet-derived growth factor, Transdifferentiation and In vivo.
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Platelet-derived growth factor is structurally related to the putative transforming protein p28sis of simian sarcoma virus.
Michael D. Waterfield;Geoffrey T. Scrace;Nigel Whittle;Paul Stroobant.
Accelerated healing of incisional wounds in rats induced by transforming growth factor-beta.
Thomas A. Mustoe;Glenn F. Pierce;Arlen Thomason;Peggy Gramates.
Platelet-derived growth factor and transforming growth factor-beta enhance tissue repair activities by unique mechanisms.
G F Pierce;T A Mustoe;J Lingelbach;V R Masakowski.
Journal of Cell Biology (1989)
Chemotaxis of monocytes and neutrophils to platelet-derived growth factor.
T F Deuel;R M Senior;J S Huang;G L Griffin.
Journal of Clinical Investigation (1982)
Role of platelet-derived growth factor in wound healing.
Glenn F. Pierce;Thomas A. Mustoe;Bruce W. Altrock;Thomas F. Deuel.
Journal of Cellular Biochemistry (1991)
Platelet Factor 4 Is Chemotactic for Neutrophils and Monocytes
Thomas F. Deuel;Robert M. Senior;Donald Chang;Gail L. Griffin.
Proceedings of the National Academy of Sciences of the United States of America (1981)
Cloning and expression of a developmentally regulated protein that induces mitogenic and neurite outgrowth activity
Yue-Sheng Li;Peter G. Milner;Anil K. Chauhan;Mark A. Watson.
Polypeptide Growth Factors: Roles in Normal and Abnormal Cell Growth
Thomas F. Deuel.
Annual Review of Cell Biology (1987)
Growth factor-induced acceleration of tissue repair through direct and inductive activities in a rabbit dermal ulcer model.
T A Mustoe;G F Pierce;C Morishima;T F Deuel.
Journal of Clinical Investigation (1991)
Pleiotrophin signals increased tyrosine phosphorylation of β-catenin through inactivation of the intrinsic catalytic activity of the receptor-type protein tyrosine phosphatase β/ζ
Kung Meng;A. Rodríguez-Peña;Todor Dimitrov;Wen Chen.
Proceedings of the National Academy of Sciences of the United States of America (2000)
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