What is she best known for?
The fields of study she is best known for:
- Enzyme
- Internal medicine
- Biochemistry
Her primary areas of investigation include Cell biology, Biochemistry, Phosphatidylinositol, Molecular biology and Phosphatase.
Her Cell biology research integrates issues from Glycoprotein Ib and Cell membrane.
Her studies in Biochemistry integrate themes in fields like Platelet activation and INPP5E.
Her research in Phosphatidylinositol intersects with topics in Transport protein, Postsynaptic density and Excitotoxicity.
Her work deals with themes such as Myosin, Protein primary structure and Myogenesis, Skeletal muscle, which intersect with Molecular biology.
Her Phosphatase research is multidisciplinary, relying on both Amino acid, NMDA receptor and OCRL, Inositol.
Her most cited work include:
- Reactive oxygen species enhance insulin sensitivity. (434 citations)
- Sec15 is an effector for the Rab11 GTPase in mammalian cells (228 citations)
- Decreased SLIM1 expression and increased gelsolin expression in failing human hearts measured by high-density oligonucleotide arrays. (218 citations)
What are the main themes of her work throughout her whole career to date?
Her scientific interests lie mostly in Cell biology, Biochemistry, Inositol, Phosphatidylinositol and Molecular biology.
As part of her studies on Cell biology, Christina Anne Mitchell often connects relevant areas like Cell growth.
Platelet activation is closely connected to Thrombin in her research, which is encompassed under the umbrella topic of Biochemistry.
Her Inositol research incorporates themes from Phospholipase C, Second messenger system, Enzyme and Pleckstrin homology domain.
She interconnects Myogenesis and Skeletal muscle in the investigation of issues within Molecular biology.
As part of the same scientific family, Christina Anne Mitchell usually focuses on PI3K/AKT/mTOR pathway, concentrating on Cancer research and intersecting with Cancer and Breast cancer.
She most often published in these fields:
- Cell biology (51.08%)
- Biochemistry (26.88%)
- Inositol (20.43%)
What were the highlights of her more recent work (between 2013-2021)?
- Cell biology (51.08%)
- PI3K/AKT/mTOR pathway (12.90%)
- Protein kinase B (9.68%)
In recent papers she was focusing on the following fields of study:
Cell biology, PI3K/AKT/mTOR pathway, Protein kinase B, PTEN and Cancer research are her primary areas of study.
Her study looks at the relationship between Cell biology and fields such as Muscular dystrophy, as well as how they intersect with chemical problems.
Her Protein kinase B research incorporates elements of Cell cycle, Kidney, Polycystic kidney disease and Cell growth.
She has included themes like Cancer, Breast cancer, Metastasis and Receptor in her Cancer research study.
Her Kinase study is concerned with Biochemistry in general.
Particularly relevant to Signal transduction is her body of work in Biochemistry.
Between 2013 and 2021, her most popular works were:
- The Inositol Polyphosphate 5-Phosphatase PIPP Regulates AKT1-Dependent Breast Cancer Growth and Metastasis (63 citations)
- INPP5E regulates phosphoinositide-dependent cilia transition zone function. (58 citations)
- INPP5E interacts with AURKA, linking phosphoinositide signaling to primary cilium stability. (54 citations)
In her most recent research, the most cited papers focused on:
- Enzyme
- Internal medicine
- Biochemistry
Christina Anne Mitchell focuses on Cell biology, PI3K/AKT/mTOR pathway, Protein kinase B, Cilium and Signal transduction.
Her Cell biology study integrates concerns from other disciplines, such as Molecular biology and Biochemistry.
Her work on PTEN as part of general PI3K/AKT/mTOR pathway study is frequently connected to Cre recombinase, therefore bridging the gap between diverse disciplines of science and establishing a new relationship between them.
Her Cilium study also includes fields such as
- Ciliopathy which connect with Smoothened Receptor, Smoothened, Scaffold protein, Hedgehog signaling pathway and Hedgehog,
- INPP5E that connect with fields like Aurora Kinase A, Mitosis and Phosphorylation.
Her Signal transduction study incorporates themes from Tumor progression and Cell growth.
Her work in Phosphoinositide 3-kinase tackles topics such as Pleckstrin homology domain which are related to areas like OCRL and Inositol.
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