Elizabeth L. Hartland mostly deals with Microbiology, Virulence, Secretion, Effector and Escherichia coli. Elizabeth L. Hartland incorporates Microbiology and Intestinal mucosa in her studies. Her Virulence study is concerned with the field of Genetics as a whole.
Her research integrates issues of Fas receptor, TRADD, Death domain and Enteropathogenic Escherichia coli in her study of Secretion. Effector is a subfield of Cell biology that Elizabeth L. Hartland investigates. Elizabeth L. Hartland interconnects Plasmid and Secretory protein in the investigation of issues within Escherichia coli.
Elizabeth L. Hartland mainly focuses on Microbiology, Virulence, Effector, Cell biology and Legionella pneumophila. Her Microbiology study incorporates themes from Enteropathogenic Escherichia coli, Enterobacteriaceae, Escherichia coli and Mutant. In her research, Protein tyrosine phosphatase is intimately related to Yersinia enterocolitica, which falls under the overarching field of Virulence.
Her Effector research integrates issues from Secretion, Type three secretion system, Molecular biology, Innate immune system and Death domain. Her Cell biology research is multidisciplinary, relying on both Burkholderia cenocepacia, Burkholderia and Glycoprotein. As a member of one scientific family, Elizabeth L. Hartland mostly works in the field of Legionella pneumophila, focusing on Legionella and, on occasion, Human pathogen.
Elizabeth L. Hartland mainly investigates Cell biology, Effector, Legionella pneumophila, Legionella and Microbiology. She works mostly in the field of Cell biology, limiting it down to topics relating to Proteome and, in certain cases, Regulon, Quorum sensing, Regulation of gene expression and Proteomics, as a part of the same area of interest. Her study in Effector is interdisciplinary in nature, drawing from both Secretion, Protein domain, Immunity, Intracellular and Rab.
Her research integrates issues of Signal transduction and Vacuole in her study of Secretion. Her Legionella pneumophila research incorporates elements of Eukaryotic transcription, Transcription, Cell nucleus, Gene and RNA polymerase II. Her research in Microbiology intersects with topics in Innate immune system, Legionnaires' disease, Escherichia coli and Virulence.
Elizabeth L. Hartland spends much of her time researching Effector, Cell biology, Protein domain, Legionella pneumophila and Death domain. The Effector study combines topics in areas such as Secretion, Cytokine secretion, Type three secretion system and Rab. Her studies deal with areas such as Proteomics, Glycoprotein, Burkholderia cenocepacia, Burkholderia and Regulation of gene expression as well as Cell biology.
Her study with Protein domain involves better knowledge in Genetics. Her Legionella pneumophila research incorporates themes from Legionella and Phenotype, Genome, Gene, Horizontal gene transfer. Her work on TRADD as part of general Death domain study is frequently linked to FADD, bridging the gap between disciplines.
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Molecular Pathogenesis of Infections Caused by Legionella pneumophila
Hayley J. Newton;Desmond K. Y. Ang;Ian R. van Driel;Elizabeth L. Hartland.
Clinical Microbiology Reviews (2010)
Enteropathogenic and enterohaemorrhagic Escherichia coli: even more subversive elements.
Alexander R. C. Wong;Jaclyn S. Pearson;Michael D. Bright;Diana Munera.
Molecular Microbiology (2011)
A Common Fold Mediates Vertebrate Defense and Bacterial Attack
Carlos J. Rosado;Ashley M. Buckle;Ruby H. P. Law;Rebecca E. Butcher;Rebecca E. Butcher.
Science (2007)
Binding of intimin from enteropathogenic Escherichia coli to Tir and to host cells.
Elizabeth L. Hartland;Miranda Batchelor;Robin M. Delahay;Christine Hale.
Molecular Microbiology (1999)
A type III effector antagonizes death receptor signalling during bacterial gut infection
Jaclyn S Pearson;Cristina Giogha;Sze Ying Ong;Catherine Lydia Kennedy.
Nature (2013)
The type III effectors NleE and NleB from enteropathogenic E. coli and OspZ from Shigella block nuclear translocation of NF-kappaB p65
Hayley J Newton;Jaclyn S Pearson;Luminita Badea;Michelle Kelly.
PLOS Pathogens (2010)
Two novel point mutations in clinical Staphylococcus aureus reduce linezolid susceptibility and switch on the stringent response to promote persistent infection.
Wei Gao;Kyra Chua;Kyra Chua;John Keith Davies;Hayley J Newton.
PLOS Pathogens (2010)
Discovery of an archetypal protein transport system in bacterial outer membranes
Joel Selkrig;Khedidja Mosbahi;Chaille T Webb;Matthew J Belousoff.
Nature Structural & Molecular Biology (2012)
Identification of a novel fimbrial gene cluster related to long polar fimbriae in locus of enterocyte effacement-negative strains of enterohemorrhagic Escherichia coli.
Stephen Doughty;Joan Sloan;Vicki Bennett-Wood;Vicki Bennett-Wood;Marcus Robertson.
Infection and Immunity (2002)
Analysis of the Legionella longbeachae Genome and Transcriptome Uncovers Unique Strategies to Cause Legionnaires' Disease
Christel Cazalet;Laura Gomez-Valero;Christophe Rusniok;Mariella Lomma.
PLOS Genetics (2010)
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