D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 57 Citations 9,396 165 World Ranking 2464 National Ranking 84

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Bacteria
  • Enzyme

Elizabeth L. Hartland mostly deals with Microbiology, Virulence, Secretion, Effector and Escherichia coli. Elizabeth L. Hartland incorporates Microbiology and Intestinal mucosa in her studies. Her Virulence study is concerned with the field of Genetics as a whole.

Her research integrates issues of Fas receptor, TRADD, Death domain and Enteropathogenic Escherichia coli in her study of Secretion. Effector is a subfield of Cell biology that Elizabeth L. Hartland investigates. Elizabeth L. Hartland interconnects Plasmid and Secretory protein in the investigation of issues within Escherichia coli.

Her most cited work include:

  • Molecular Pathogenesis of Infections Caused by Legionella pneumophila (320 citations)
  • Enteropathogenic and enterohaemorrhagic Escherichia coli: even more subversive elements. (286 citations)
  • A Common Fold Mediates Vertebrate Defense and Bacterial Attack (235 citations)

What are the main themes of her work throughout her whole career to date?

Elizabeth L. Hartland mainly focuses on Microbiology, Virulence, Effector, Cell biology and Legionella pneumophila. Her Microbiology study incorporates themes from Enteropathogenic Escherichia coli, Enterobacteriaceae, Escherichia coli and Mutant. In her research, Protein tyrosine phosphatase is intimately related to Yersinia enterocolitica, which falls under the overarching field of Virulence.

Her Effector research integrates issues from Secretion, Type three secretion system, Molecular biology, Innate immune system and Death domain. Her Cell biology research is multidisciplinary, relying on both Burkholderia cenocepacia, Burkholderia and Glycoprotein. As a member of one scientific family, Elizabeth L. Hartland mostly works in the field of Legionella pneumophila, focusing on Legionella and, on occasion, Human pathogen.

She most often published in these fields:

  • Microbiology (87.16%)
  • Virulence (66.51%)
  • Effector (53.67%)

What were the highlights of her more recent work (between 2017-2021)?

  • Cell biology (46.33%)
  • Effector (53.67%)
  • Legionella pneumophila (39.91%)

In recent papers she was focusing on the following fields of study:

Elizabeth L. Hartland mainly investigates Cell biology, Effector, Legionella pneumophila, Legionella and Microbiology. She works mostly in the field of Cell biology, limiting it down to topics relating to Proteome and, in certain cases, Regulon, Quorum sensing, Regulation of gene expression and Proteomics, as a part of the same area of interest. Her study in Effector is interdisciplinary in nature, drawing from both Secretion, Protein domain, Immunity, Intracellular and Rab.

Her research integrates issues of Signal transduction and Vacuole in her study of Secretion. Her Legionella pneumophila research incorporates elements of Eukaryotic transcription, Transcription, Cell nucleus, Gene and RNA polymerase II. Her research in Microbiology intersects with topics in Innate immune system, Legionnaires' disease, Escherichia coli and Virulence.

Between 2017 and 2021, her most popular works were:

  • More than 18,000 effectors in the Legionella genus genome provide multiple, independent combinations for replication in human cells. (63 citations)
  • More than 18,000 effectors in the Legionella genus genome provide multiple, independent combinations for replication in human cells. (63 citations)
  • More than 18,000 effectors in the Legionella genus genome provide multiple, independent combinations for replication in human cells. (63 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Bacteria
  • Enzyme

Elizabeth L. Hartland spends much of her time researching Effector, Cell biology, Protein domain, Legionella pneumophila and Death domain. The Effector study combines topics in areas such as Secretion, Cytokine secretion, Type three secretion system and Rab. Her studies deal with areas such as Proteomics, Glycoprotein, Burkholderia cenocepacia, Burkholderia and Regulation of gene expression as well as Cell biology.

Her study with Protein domain involves better knowledge in Genetics. Her Legionella pneumophila research incorporates themes from Legionella and Phenotype, Genome, Gene, Horizontal gene transfer. Her work on TRADD as part of general Death domain study is frequently linked to FADD, bridging the gap between disciplines.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Molecular Pathogenesis of Infections Caused by Legionella pneumophila

Hayley J. Newton;Desmond K. Y. Ang;Ian R. van Driel;Elizabeth L. Hartland.
Clinical Microbiology Reviews (2010)

520 Citations

Enteropathogenic and enterohaemorrhagic Escherichia coli: even more subversive elements.

Alexander R. C. Wong;Jaclyn S. Pearson;Michael D. Bright;Diana Munera.
Molecular Microbiology (2011)

351 Citations

A Common Fold Mediates Vertebrate Defense and Bacterial Attack

Carlos J. Rosado;Ashley M. Buckle;Ruby H. P. Law;Rebecca E. Butcher;Rebecca E. Butcher.
Science (2007)

314 Citations

Binding of intimin from enteropathogenic Escherichia coli to Tir and to host cells.

Elizabeth L. Hartland;Miranda Batchelor;Robin M. Delahay;Christine Hale.
Molecular Microbiology (1999)

264 Citations

A type III effector antagonizes death receptor signalling during bacterial gut infection

Jaclyn S Pearson;Cristina Giogha;Sze Ying Ong;Catherine Lydia Kennedy.
Nature (2013)

256 Citations

The type III effectors NleE and NleB from enteropathogenic E. coli and OspZ from Shigella block nuclear translocation of NF-kappaB p65

Hayley J Newton;Jaclyn S Pearson;Luminita Badea;Michelle Kelly.
PLOS Pathogens (2010)

245 Citations

Two novel point mutations in clinical Staphylococcus aureus reduce linezolid susceptibility and switch on the stringent response to promote persistent infection.

Wei Gao;Kyra Chua;Kyra Chua;John Keith Davies;Hayley J Newton.
PLOS Pathogens (2010)

226 Citations

Discovery of an archetypal protein transport system in bacterial outer membranes

Joel Selkrig;Khedidja Mosbahi;Chaille T Webb;Matthew J Belousoff.
Nature Structural & Molecular Biology (2012)

214 Citations

Identification of a novel fimbrial gene cluster related to long polar fimbriae in locus of enterocyte effacement-negative strains of enterohemorrhagic Escherichia coli.

Stephen Doughty;Joan Sloan;Vicki Bennett-Wood;Vicki Bennett-Wood;Marcus Robertson.
Infection and Immunity (2002)

204 Citations

Analysis of the Legionella longbeachae Genome and Transcriptome Uncovers Unique Strategies to Cause Legionnaires' Disease

Christel Cazalet;Laura Gomez-Valero;Christophe Rusniok;Mariella Lomma.
PLOS Genetics (2010)

195 Citations

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