Internal medicine is closely attributed to Transforming growth factor in his research. His Transforming growth factor study frequently intersects with other fields, such as Endocrinology. Many of his studies on Endocrinology involve topics that are commonly interrelated, such as Transforming growth factor beta. He regularly links together related areas like Internal medicine in his Transforming growth factor beta studies. Much of his study explores Embryonic stem cell relationship to FGF and mesoderm formation. His FGF and mesoderm formation study frequently draws connections between adjacent fields such as Endoderm. He performs multidisciplinary studies into Endoderm and Mesoderm in his work. He combines Mesoderm and Ectoderm in his research. His work often combines Ectoderm and Embryogenesis studies.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The Two-Handed E Box Binding Zinc Finger Protein SIP1 Downregulates E-Cadherin and Induces Invasion
Joke Comijn;Geert Berx;Petra Vermassen;Kristin Verschueren.
Molecular Cell (2001)
Identification of a potent Xenopus mesoderm-inducing factor as a homologue of activin A.
J C Smith;B M Price;K Van Nimmen;Danny Huylebroeck.
Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency
An Herreman;Dieter Hartmann;Wim Annaert;Paul Saftig.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Osteogenic protein-1 binds to activin type II receptors and induces certain activin-like effects.
H Yamashita;P ten Dijke;D Huylebroeck;T K Sampath.
Journal of Cell Biology (1995)
SIP1, a novel zinc finger/homeodomain repressor, interacts with Smad proteins and binds to 5'-CACCT sequences in candidate target genes.
Kristin Verschueren;Jacques E. Remacle;Clara Collart;Clara Collart;Harry Kraft;Harry Kraft.
Journal of Biological Chemistry (1999)
In vitro mutagenesis of a full-length cDNA clone of Semliki Forest virus: the small 6,000-molecular-weight membrane protein modulates virus release
P Liljeström;S Lusa;Danny Huylebroeck;H Garoff.
Journal of Virology (1991)
Smad5 knockout mice die at mid-gestation due to multiple embryonic and extraembryonic defects
H Chang;Danny Huylebroeck;K Verschueren;Qiuxia Guo.
Loss-of-function mutations in LEMD3 result in osteopoikilosis, Buschke-Ollendorff syndrome and melorheostosis.
Jan Hellemans;Olena Preobrazhenska;Andy Willaert;Philippe Debeer.
Nature Genetics (2004)
Distinct spatial and temporal expression patterns of two type I receptors for bone morphogenetic proteins during mouse embryogenesis
N Dewulf;K Verschueren;O Lonnoy;A Morén.
Mice lacking ZFHX1B, the gene that codes for Smad-interacting protein-1, reveal a role for multiple neural crest cell defects in the etiology of Hirschsprung disease-mental retardation syndrome.
Tom Van de Putte;Mitsuji Maruhashi;Annick Francis;Lucien Nelles.
American Journal of Human Genetics (2003)
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