What is he best known for?
The fields of study he is best known for:
His primary areas of study are Shigella flexneri, Secretion, Microbiology, Cell biology and Virulence.
His Shigella flexneri research is multidisciplinary, incorporating elements of Extracellular, Cytoplasm and Transfection.
His Secretion research incorporates themes from Shigella, Mutant and Effector.
His research investigates the link between Microbiology and topics such as Type three secretion system that cross with problems in Organelle, Membrane and Red blood cell.
His Cell biology study incorporates themes from In vitro and Translocon.
He combines subjects such as Plasmid, Operon and Enterobacteriaceae with his study of Virulence.
His most cited work include:
- Nonpolar mutagenesis of the ipa genes defines IpaB, IpaC, and IpaD as effectors of Shigella flexneri entry into epithelial cells. (672 citations)
- The tripartite type III secreton of Shigella flexneri inserts IpaB and IpaC into host membranes. (436 citations)
- The virulence plasmid pWR100 and the repertoire of proteins secreted by the type III secretion apparatus of Shigella flexneri. (331 citations)
What are the main themes of his work throughout his whole career to date?
Claude Parsot mainly investigates Shigella flexneri, Secretion, Microbiology, Effector and Biochemistry.
The various areas that he examines in his Shigella flexneri study include Molecular biology, Cytoplasm, Secretory protein and Chaperone.
His study in Secretion is interdisciplinary in nature, drawing from both Intracellular parasite, Bacteria, Mutant, Enterobacteriaceae and Cell biology.
His work deals with themes such as Plasmid, Shigella, Type three secretion system, Virulence and Peptide sequence, which intersect with Microbiology.
His Effector study combines topics from a wide range of disciplines, such as Plasma protein binding, Ubiquitin, Ubiquitin ligase, Coactivator and Innate immune system.
His work in the fields of Biochemistry, such as Enzyme, Proteinase K, Gram-negative bacteria and Lysis, intersects with other areas such as Congo red.
He most often published in these fields:
- Shigella flexneri (67.89%)
- Secretion (63.30%)
- Microbiology (56.88%)
What were the highlights of his more recent work (between 2010-2019)?
- Shigella flexneri (67.89%)
- Secretion (63.30%)
- Effector (44.95%)
In recent papers he was focusing on the following fields of study:
Shigella flexneri, Secretion, Effector, Microbiology and Immune system are his primary areas of study.
Shigella flexneri and Molecular biology are commonly linked in his work.
Claude Parsot has included themes like Bacterial outer membrane, Cytoplasm and Intracellular parasite in his Secretion study.
His work carried out in the field of Effector brings together such families of science as Plasma protein binding, Ubiquitin, Ubiquitin ligase and Kinase activity.
The concepts of his Microbiology study are interwoven with issues in Lymphocyte, Programmed cell death and Vacuole.
In the subject of general Immune system, his work in Acquired immune system and Priming is often linked to B-1 cell, thereby combining diverse domains of study.
Between 2010 and 2019, his most popular works were:
- Escape of Actively Secreting Shigella flexneri from ATG8/LC3-Positive Vacuoles Formed during Cell-To-Cell Spread Is Facilitated by IcsB and VirA (61 citations)
- Escape of Actively Secreting Shigella flexneri from ATG8/LC3-Positive Vacuoles Formed during Cell-To-Cell Spread Is Facilitated by IcsB and VirA (61 citations)
- A Fluorescent Reporter Reveals On/Off Regulation of the Shigella Type III Secretion Apparatus during Entry and Cell-to-Cell Spread (52 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Secretion, Shigella flexneri, ATG8, Actin and Intracellular.
His ATG8 research overlaps with other disciplines such as Vacuole, Bacterial outer membrane, Microbiology, RAB1 and Cytoplasm.
In his research, Claude Parsot performs multidisciplinary study on Vacuole and Context.
The study incorporates disciplines such as Host cell cytoplasm, Green fluorescent protein, Intracellular parasite and Effector in addition to Actin.
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