Endocrinology, Internal medicine, Apoptosis, Cancer research and Inhibitor of apoptosis are his primary areas of study. Endocrinology is represented through his Follicular phase, Granulosa cell, Ovarian follicle, Follicular atresia and Gonadotropin research. As a member of one scientific family, Benjamin K. Tsang mostly works in the field of Internal medicine, focusing on Follicular fluid and, on occasion, Hormone and Theca interna.
His research investigates the link between Apoptosis and topics such as Downregulation and upregulation that cross with problems in Cyclic guanosine monophosphate, Activator, Pifithrin and Puma. His research integrates issues of Protein kinase B, Cisplatin, Ovarian cancer, Tumor suppressor gene and Programmed cell death in his study of Cancer research. He usually deals with Inhibitor of apoptosis and limits it to topics linked to XIAP and Apoptosis Inhibitor and NODAL.
Benjamin K. Tsang mostly deals with Internal medicine, Endocrinology, Cancer research, Apoptosis and Granulosa cell. His research on Internal medicine often connects related areas such as Folliculogenesis. The Cancer research study combines topics in areas such as Protein kinase B, Cisplatin, Cancer cell, Inhibitor of apoptosis and Ovarian cancer.
The concepts of his Protein kinase B study are interwoven with issues in Caspase 3 and PI3K/AKT/mTOR pathway. His Apoptosis study combines topics in areas such as Downregulation and upregulation and Cell biology. In his research, TGF alpha is intimately related to Transforming growth factor, which falls under the overarching field of Granulosa cell.
Benjamin K. Tsang spends much of his time researching Internal medicine, Cancer research, Ovarian cancer, Endocrinology and Apoptosis. His Gonadotropin and Paracrine signalling study, which is part of a larger body of work in Internal medicine, is frequently linked to Chemerin and Hyperandrogenism, bridging the gap between disciplines. In Cancer research, Benjamin K. Tsang works on issues like Cancer, which are connected to Reactive oxygen species.
His Ovarian cancer study combines topics from a wide range of disciplines, such as Tumor microenvironment, Fallopian tube, Signal transduction, Metastasis and Disease. His research investigates the connection between Endocrinology and topics such as Folliculogenesis that intersect with problems in Follicle-stimulating hormone, Luteinizing hormone and Receptor. Benjamin K. Tsang combines subjects such as Curcumin, Cisplatin and Cell biology with his study of Apoptosis.
Benjamin K. Tsang mostly deals with Apoptosis, Cancer research, Cancer cell, Internal medicine and Endocrinology. His Apoptosis research includes themes of Curcumin, Cisplatin and Cell biology. The various areas that Benjamin K. Tsang examines in his Cancer research study include Cell and Ovarian cancer.
His research integrates issues of Protein kinase B and Signal transduction in his study of Ovarian cancer. His study in the fields of Cholesterol side-chain cleavage enzyme under the domain of Internal medicine overlaps with other disciplines such as Chemerin, Anovulation and Hyperinsulinemia. His work in the fields of Dihydrotestosterone overlaps with other areas such as Hyperandrogenism and Polycystic ovary.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Akt phosphorylation and stabilization of X-linked inhibitor of apoptosis protein (XIAP).
Han C. Dan;Mei Sun;Satoshi Kaneko;Richard I. Feldman.
Journal of Biological Chemistry (2004)
A new, nongenomic estrogen action: the rapid release of intracellular calcium.
Paul Morley;James F. Whitfield;Barbara C. Vanderhyden;Benjamin K. Tsang.
Down-Regulation of X-linked Inhibitor of Apoptosis Protein Induces Apoptosis in Chemoresistant Human Ovarian Cancer Cells
Hiromasa Sasaki;YingLun Sheng;Fumikazu Kotsuji;Benjamin K. Tsang.
Cancer Research (2000)
XIAP Regulates Akt Activity and Caspase-3-dependent Cleavage during Cisplatin-induced Apoptosis in Human Ovarian Epithelial Cancer Cells
Eric Asselin;Gordon B. Mills;Benjamin K. Tsang.
Cancer Research (2001)
The regulation of cell proliferation by calcium and cyclic AMP.
J. F. Whitfield;A. L. Boynton;J. P. Macmanus;M. Sikorska.
Molecular and Cellular Biochemistry (1979)
Akt-Mediated Cisplatin Resistance in Ovarian Cancer: Modulation of p53 Action on Caspase-Dependent Mitochondrial Death Pathway
Xiaokui Yang;Michael Fraser;Ute M. Moll;Ajoy Basak.
Cancer Research (2006)
The roles of calcium and cyclic AMP in cell proliferation.
J. F. Whitfield;A. L. Boynton;J. P. MacManus;R. H. Rixon.
Annals of the New York Academy of Sciences (1980)
Human Ovarian Cancer and Cisplatin Resistance: Possible Role of Inhibitor of Apoptosis Proteins1
Julang Li;Qiang Feng;Jong-Min Kim;Danielle Schneiderman.
Caspase-3 in the rat ovary: localization and possible role in follicular atresia and luteal regression.
David L. Boone;Benjamin K. Tsang.
Biology of Reproduction (1998)
p53 Is a Determinant of X-Linked Inhibitor of Apoptosis Protein/Akt-Mediated Chemoresistance in Human Ovarian Cancer Cells
Michael Fraser;Brendan M. Leung;Xiaojuan Yan;Han C. Dan.
Cancer Research (2003)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: