James F. Whitfield mainly investigates Internal medicine, Endocrinology, Cell biology, Calcium and Parathyroid hormone. His work in Internal medicine tackles topics such as Mechanism of action which are related to areas like Prolactin, Endocrine gland and Adenosine triphosphate. James F. Whitfield has included themes like Receptor and Bone healing in his Endocrinology study.
James F. Whitfield combines subjects such as Cell growth, Cell division, Anatomy, Calmodulin and Cell cycle with his study of Cell biology. The Calcium study combines topics in areas such as Extracellular and DNA, DNA synthesis. His studies in Parathyroid hormone integrate themes in fields like Adenylyl cyclase, Hormone, Ovariectomized rat, Protein kinase C and Osteoporosis.
Internal medicine, Endocrinology, Cell biology, Parathyroid hormone and Biochemistry are his primary areas of study. His study in Calcium, Osteoporosis, Stimulation, Receptor and Hormone are all subfields of Internal medicine. His research in Calcium intersects with topics in Extracellular, Cell and DNA synthesis.
He interconnects Cell cycle, Cell division, In vitro and Cell growth in the investigation of issues within Cell biology. James F. Whitfield has researched Parathyroid hormone in several fields, including Calcitonin and Stereochemistry. He works mostly in the field of Biochemistry, limiting it down to concerns involving Molecular biology and, occasionally, Kinase and Mitogen-activated protein kinase kinase.
James F. Whitfield mainly investigates Cell biology, Endocrinology, Internal medicine, Receptor and Neuroscience. His Cell biology research incorporates elements of Genetically modified mouse, Amyloid beta and Biochemistry. His study explores the link between Biochemistry and topics such as Cell culture that cross with problems in Parathyroid hormone-related protein and Hormone.
His Endocrinology research is multidisciplinary, incorporating elements of Vascular endothelial growth factor A, Vascular endothelial growth factor and Signal transduction. His research investigates the connection between Receptor and topics such as NPS-2143 that intersect with problems in Astrocyte and Calcilytic. His study in Lactam is interdisciplinary in nature, drawing from both Protein kinase C and Parathyroid hormone.
The scientist’s investigation covers issues in Receptor, Internal medicine, Endocrinology, NPS-2143 and Alzheimer's disease. His studies in Receptor integrate themes in fields like Amyloid beta and Neuroscience. Internal medicine and Signal transduction are commonly linked in his work.
As a member of one scientific family, James F. Whitfield mostly works in the field of Signal transduction, focusing on Cortical bone and, on occasion, Protein kinase C, Adenylyl cyclase and Parathyroid hormone. His biological study spans a wide range of topics, including Dentate gyrus, Hippocampal formation, Cognitive decline, Neurogenesis and Subgranular zone. His work carried out in the field of Cell biology brings together such families of science as Amino acid, Biochemistry, Enzyme and Cell culture.
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Topoisomerase II-reactive chemotherapeutic drugs induce apoptosis in thymocytes.
P R Walker;C Smith;T Youdale;J Leblanc.
Cancer Research (1991)
A new, nongenomic estrogen action: the rapid release of intracellular calcium.
Paul Morley;James F. Whitfield;Barbara C. Vanderhyden;Benjamin K. Tsang.
The regulation of cell proliferation by calcium and cyclic AMP.
J. F. Whitfield;A. L. Boynton;J. P. Macmanus;M. Sikorska.
Molecular and Cellular Biochemistry (1979)
THE POSITIVE CONTROL OF CELL PROLIFERATION BY THE INTERPLAY OF CALCIUM IONS AND CYCLIC NUCLEOTIDES. A REVIEW
J. F. Whitfield;J. P. MacManus;R. H. Rixon;A. L. Boynton.
In Vitro Cellular & Developmental Biology – Plant (1976)
The roles of calcium and cyclic AMP in cell proliferation.
J. F. Whitfield;A. L. Boynton;J. P. MacManus;R. H. Rixon.
Annals of the New York Academy of Sciences (1980)
The protein kinase-C activation domain of the parathyroid hormone.
H Jouishomme;J F Whitfield;B Chakravarthy;J P Durkin.
Calmodulin stimulates DNA synthesis by rat liver cells.
A.L. Boynton;J.F. Whitfield;J.P. MacManus.
Biochemical and Biophysical Research Communications (1980)
Parathyroid hormone fragments may stimulate bone growth in ovariectomized rats by activating adenylyl cyclase
Ray H. Rixon;James F. Whitfield;Lyne Gagnon;Richard J. Isaacs.
Journal of Bone and Mineral Research (2009)
Adrenergic antagonists, and a possible link between the increase in cyclic adenosine 3',5'-monophosphate and DNA synthesis during liver regeneration.
John P. Macmanus;Brian M. Braceland;Tony Youdale;James F. Whitfield.
Journal of Cellular Physiology (1973)
Calcium, cyclic adenosine 3',5'-monophosphate, and the control of cell proliferation: a review.
J. F. Whitfield;R. H. Rixon;J. P. MacManus;S. D. Balk.
In Vitro Cellular & Developmental Biology – Plant (1973)
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