What is he best known for?
The fields of study he is best known for:
Bé Wieringa mainly focuses on Internal medicine, Endocrinology, Creatine kinase, Myotonic dystrophy and Genetics.
His work in the fields of Internal medicine, such as Cholesterol, Apolipoprotein E, Lipoprotein and Tetany, intersects with other areas such as Muscle fatigue.
His studies deal with areas such as Bioenergetics, Gene isoform, Transgene and Cell biology as well as Endocrinology.
His Creatine kinase research entails a greater understanding of Biochemistry.
Bé Wieringa has researched Myotonic dystrophy in several fields, including RNA, Myotonin-protein kinase, Trinucleotide repeat expansion and Protein kinase A.
His research integrates issues of MBNL1 and Molecular biology in his study of Myotonin-protein kinase.
His most cited work include:
- Abnormal myotonic dystrophy protein kinase levels produce only mild myopathy in mice (301 citations)
- Skeletal muscles of mice deficient in muscle creatine kinase lack burst activity. (291 citations)
- The SH-SY5Y cell line in Parkinson's disease research: a systematic review (259 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Molecular biology, Biochemistry, Cell biology, Creatine kinase and Gene.
His Molecular biology research includes elements of Gene expression, Protein tyrosine phosphatase, Complementary DNA, Exon and Myotonic dystrophy.
His study in Myotonic dystrophy is interdisciplinary in nature, drawing from both RNA, Myotonin-protein kinase, Trinucleotide repeat expansion and Protein kinase A.
Many of his studies involve connections with topics such as Cytoskeleton and Cell biology.
He interconnects Cytosol, Creatine and Skeletal muscle in the investigation of issues within Creatine kinase.
His Gene study results in a more complete grasp of Genetics.
He most often published in these fields:
- Molecular biology (33.33%)
- Biochemistry (25.00%)
- Cell biology (23.53%)
What were the highlights of his more recent work (between 2011-2020)?
- Cell biology (23.53%)
- Myotonic dystrophy (20.59%)
- Biochemistry (25.00%)
In recent papers he was focusing on the following fields of study:
His primary areas of investigation include Cell biology, Myotonic dystrophy, Biochemistry, Molecular biology and Creatine kinase.
His Cell biology research is multidisciplinary, incorporating perspectives in Transcriptome and Cytosol.
His Myotonic dystrophy study is associated with Genetics.
His work on Cell growth, Glycolysis, Creatine metabolism and Transgene is typically connected to In vivo magnetic resonance spectroscopy as part of general Biochemistry study, connecting several disciplines of science.
His research in Molecular biology intersects with topics in Myotonin-protein kinase, Gene conversion, Protein subunit and Cell culture.
While the research belongs to areas of Creatine kinase, he spends his time largely on the problem of Skeletal muscle, intersecting his research to questions surrounding Cell.
Between 2011 and 2020, his most popular works were:
- The SH-SY5Y cell line in Parkinson's disease research: a systematic review (259 citations)
- TRPM7 Is Required for Breast Tumor Cell Metastasis (140 citations)
- CRISPR/Cas9-Induced (CTG⋅CAG) n Repeat Instability in the Myotonic Dystrophy Type 1 Locus: Implications for Therapeutic Genome Editing (74 citations)
In his most recent research, the most cited papers focused on:
Bé Wieringa mostly deals with Disease, Parkinson's disease, Cell culture, Enzyme and Cell biology.
The Disease study combines topics in areas such as Neuroscience, Cellular differentiation and Cellular model.
His work investigates the relationship between Cell culture and topics such as Citrate synthase that intersect with problems in Adenosine triphosphate and Molecular biology.
Adenosine triphosphate is a subfield of Biochemistry that Bé Wieringa explores.
His work in the fields of Apyrase overlaps with other areas such as Adenosine diphosphate.
His study looks at the relationship between Cell biology and fields such as Zymosan, as well as how they intersect with chemical problems.
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