Barbara Cannon mainly focuses on Internal medicine, Endocrinology, Adipose tissue, Brown adipose tissue and Thermogenin. Her Cold acclimation research extends to Internal medicine, which is thematically connected. Her study in the field of Lipolysis, Obesity and Hormone is also linked to topics like Sympathetic nervous system and Thyroid hormone receptor beta.
Her Adipose tissue research includes themes of Transcriptome and Downregulation and upregulation. In general Brown adipose tissue study, her work on Nonshivering thermogenesis often relates to the realm of Thyroid hormone receptor alpha, thereby connecting several areas of interest. Her Thermogenin research is multidisciplinary, relying on both UCP3, Uncoupling protein, Gene expression, Regulation of gene expression and Membrane protein.
Endocrinology, Internal medicine, Brown adipose tissue, Adipose tissue and Thermogenin are her primary areas of study. Her work in Thermogenesis and White adipose tissue are all subfields of Endocrinology research. Her research is interdisciplinary, bridging the disciplines of Regulation of gene expression and Internal medicine.
Her work carried out in the field of Brown adipose tissue brings together such families of science as Messenger RNA, Cold acclimation, Mitochondrion, Cell biology and Hamster. Her Adipose tissue study combines topics in areas such as Transcriptome and Cellular differentiation. Her work deals with themes such as UCP3, Uncoupling protein and Nonshivering thermogenesis, which intersect with Thermogenin.
The scientist’s investigation covers issues in Endocrinology, Internal medicine, Brown adipose tissue, Adipose tissue and Thermogenesis. Barbara Cannon works mostly in the field of Endocrinology, limiting it down to topics relating to Browning and, in certain cases, Cold acclimation, Calorie restriction, Gut flora, Prebiotic and White adipose tissue. In the subject of general Internal medicine, her work in Metabolism, Metabolic effects, Mitochondrial ROS and Oxidative stress is often linked to Philosophy, thereby combining diverse domains of study.
In general Brown adipose tissue, her work in Thermogenin and Diet induced thermogenesis is often linked to Biological sciences linking many areas of study. Her Adipose tissue study incorporates themes from Metabolic profile, Computational biology, Gene expression and Bioinformatics. Her Thermogenesis research integrates issues from Metabolic control analysis, Cell, Fetal bovine serum and Rosiglitazone.
Her primary areas of study are Internal medicine, Endocrinology, Brown adipose tissue, Adipose tissue and Thermogenesis. Her Internal medicine study often links to related topics such as Mitochondrion. Her research in the fields of Mitochondrial ROS, Adaptive Thermogenesis and Cold stimulation overlaps with other disciplines such as Sympathetic nervous system.
Her Brown adipose tissue study integrates concerns from other disciplines, such as Mitochondrial DNA and Lipolysis. The various areas that Barbara Cannon examines in her Adipose tissue study include Catecholamine, Browning, Obesity and Homeostasis. Her studies deal with areas such as Transgene and Leptin as well as Thermogenesis.
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Brown Adipose Tissue: Function and Physiological Significance
Barbara Cannon;Jan Nedergaard.
Physiological Reviews (2004)
Unexpected evidence for active brown adipose tissue in adult humans
Jan Nedergaard;Tore Bengtsson;Barbara Cannon.
American Journal of Physiology-endocrinology and Metabolism (2007)
Chronic Peroxisome Proliferator-activated Receptor γ (PPARγ) Activation of Epididymally Derived White Adipocyte Cultures Reveals a Population of Thermogenically Competent, UCP1-containing Adipocytes Molecularly Distinct from Classic Brown Adipocytes
Natasa Petrovic;Tomas B. Walden;Irina G. Shabalina;James A. Timmons.
Journal of Biological Chemistry (2010)
UCP1 Ablation Induces Obesity and Abolishes Diet-Induced Thermogenesis in Mice Exempt from Thermal Stress by Living at Thermoneutrality
Helena M. Feldmann;Valeria Golozoubova;Barbara Cannon;Jan Nedergaard.
Cell Metabolism (2009)
The presence of UCP1 demonstrates that metabolically active adipose tissue in the neck of adult humans truly represents brown adipose tissue
Maria Cristina Zingaretti;Francesca Crosta;Alessandra Vitali;Mario Guerrieri.
The FASEB Journal (2009)
Myogenic gene expression signature establishes that brown and white adipocytes originate from distinct cell lineages
James A. Timmons;James A. Timmons;James A. Timmons;Kristian Wennmalm;Ola Larsson;Tomas B. Walden;Tomas B. Walden.
Proceedings of the National Academy of Sciences of the United States of America (2007)
UCP1: the only protein able to mediate adaptive non-shivering thermogenesis and metabolic inefficiency.
Jan Nedergaard;Valeria Golozoubova;Anita Matthias;Abolfazl Asadi.
Biochimica et Biophysica Acta (2001)
Nonshivering thermogenesis and its adequate measurement in metabolic studies
Barbara Cannon;Jan Nedergaard.
The Journal of Experimental Biology (2011)
Recruited vs. nonrecruited molecular signatures of brown, “brite,” and white adipose tissues
Tomas B. Waldén;Ida R. Hansen;James A. Timmons;James A. Timmons;Barbara Cannon;Barbara Cannon.
American Journal of Physiology-endocrinology and Metabolism (2012)
UCP1 in Brite/Beige Adipose Tissue Mitochondria Is Functionally Thermogenic
Irina G. Shabalina;Natasa Petrovic;Jasper M.A. de Jong;Anastasia V. Kalinovich.
Cell Reports (2013)
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