Asha S. Multani

The University of Texas MD Anderson Cancer Center
United States

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Biology and Biochemistry D-index 55 Citations 12,469 145 World Ranking 10363 National Ranking 4530

Overview

What are the main themes of her work throughout her whole career to date

Her Genetics study frequently draws connections to adjacent fields such as Gene rearrangement. Much of her study explores Gene relationship to Gene rearrangement. In her work, she performs multidisciplinary research in DNA and Chromosomal translocation. In her articles, she combines various disciplines, including Chromosomal translocation and Genetics. Her Cancer research study typically links adjacent topics like Myeloid. Asha S. Multani combines topics linked to Cancer research with her work on Myeloid. She combines Molecular biology and Mutation in her studies. Her multidisciplinary approach integrates Mutation and Carcinogenesis in her work. In her research, Asha S. Multani performs multidisciplinary study on Carcinogenesis and Cell cycle.

Asha S. Multani most often published in these fields:

Genetics (100.00%)
Gene (80.00%)
DNA (60.00%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice

Sunil R. Hingorani;Lifu Wang;Asha S. Multani;Chelsea Combs.
Cancer Cell (2005)

2272 Citations

Clonal evolution in breast cancer revealed by single nucleus genome sequencing

Yong Wang;Jill Waters;Marco L. Leung;Anna Unruh.
Nature (2014)

1010 Citations

CCAT2, a novel noncoding RNA mapping to 8q24, underlies metastatic progression and chromosomal instability in colon cancer

Hui Ling;Riccardo Spizzo;Yaser Atlasi;Milena Nicoloso.
Genome Research (2013)

605 Citations

Essential role of limiting telomeres in the pathogenesis of Werner syndrome.

Sandy Chang;Asha S Multani;Noelia G Cabrera;Maria L Naylor.
Nature Genetics (2004)

489 Citations

Pot1 deficiency initiates DNA damage checkpoint activation and aberrant homologous recombination at telomeres.

Ling Wu;Asha S. Multani;Hua He;Wilfredo Cosme-Blanco.
Cell (2006)

482 Citations

Chromosome stability, in the absence of apoptosis, is critical for suppression of tumorigenesis in Trp53 mutant mice.

Geng Liu;John M Parant;Gene Lang;Patty Chau.
Nature Genetics (2004)

423 Citations

Telomere dysfunction suppresses spontaneous tumorigenesis in vivo by initiating p53‐dependent cellular senescence

Wilfredo Cosme-Blanco;Mei Feng Shen;Alexander J.F. Lazar;Sen Pathak.
EMBO Reports (2007)

280 Citations

Control of chromosome stability by the β-TrCP-REST-Mad2 axis

Daniele Guardavaccaro;David Frescas;N. Valerio Dorrello;Angelo Peschiaroli.
Nature (2008)

219 Citations

Elevated telomere-telomere recombination in WRN-deficient, telomere dysfunctional cells promotes escape from senescence and engagement of the ALT pathway

Purnima R. Laud;Asha S. Multani;Susan M. Bailey;Ling Wu.
Genes & Development (2005)

215 Citations

Human Dkk-1, a gene encoding a Wnt antagonist, responds to DNA damage and its overexpression sensitizes brain tumor cells to apoptosis following alkylation damage of DNA.

Jiang Shou;Francis Ali-Osman;Asha S Multani;Sen Pathak.
Oncogene (2002)

206 Citations

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