Her Genetics study frequently draws connections to adjacent fields such as Gene rearrangement. Much of her study explores Gene relationship to Gene rearrangement. In her work, she performs multidisciplinary research in DNA and Chromosomal translocation. In her articles, she combines various disciplines, including Chromosomal translocation and Genetics. Her Cancer research study typically links adjacent topics like Myeloid. Asha S. Multani combines topics linked to Cancer research with her work on Myeloid. She combines Molecular biology and Mutation in her studies. Her multidisciplinary approach integrates Mutation and Carcinogenesis in her work. In her research, Asha S. Multani performs multidisciplinary study on Carcinogenesis and Cell cycle.
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Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice
Sunil R. Hingorani;Lifu Wang;Asha S. Multani;Chelsea Combs.
Cancer Cell (2005)
Clonal evolution in breast cancer revealed by single nucleus genome sequencing
Yong Wang;Jill Waters;Marco L. Leung;Anna Unruh.
CCAT2, a novel noncoding RNA mapping to 8q24, underlies metastatic progression and chromosomal instability in colon cancer
Hui Ling;Riccardo Spizzo;Yaser Atlasi;Milena Nicoloso.
Genome Research (2013)
Essential role of limiting telomeres in the pathogenesis of Werner syndrome.
Sandy Chang;Asha S Multani;Noelia G Cabrera;Maria L Naylor.
Nature Genetics (2004)
Pot1 deficiency initiates DNA damage checkpoint activation and aberrant homologous recombination at telomeres.
Ling Wu;Asha S. Multani;Hua He;Wilfredo Cosme-Blanco.
Chromosome stability, in the absence of apoptosis, is critical for suppression of tumorigenesis in Trp53 mutant mice.
Geng Liu;John M Parant;Gene Lang;Patty Chau.
Nature Genetics (2004)
Telomere dysfunction suppresses spontaneous tumorigenesis in vivo by initiating p53‐dependent cellular senescence
Wilfredo Cosme-Blanco;Mei Feng Shen;Alexander J.F. Lazar;Sen Pathak.
EMBO Reports (2007)
Control of chromosome stability by the β-TrCP-REST-Mad2 axis
Daniele Guardavaccaro;David Frescas;N. Valerio Dorrello;Angelo Peschiaroli.
Elevated telomere-telomere recombination in WRN-deficient, telomere dysfunctional cells promotes escape from senescence and engagement of the ALT pathway
Purnima R. Laud;Asha S. Multani;Susan M. Bailey;Ling Wu.
Genes & Development (2005)
Human Dkk-1, a gene encoding a Wnt antagonist, responds to DNA damage and its overexpression sensitizes brain tumor cells to apoptosis following alkylation damage of DNA.
Jiang Shou;Francis Ali-Osman;Asha S Multani;Sen Pathak.
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