World's Best Scientists 2026 revealed!

D-Index & Metrics

Genetics

D-Index
60
Citations
20178
World Ranking
3119
National Ranking
1362

Sandy Chang publication distribution in Genetics in 2026

The chart shows the distribution of publications by all Research.com ranked scientists in the field of Genetics in 2026. The highlighted bar marks where Sandy Chang sits on this spectrum.

45–54 publications: 6 scientists 55–64 publications: 10 scientists 65–74 publications: 35 scientists 75–84 publications: 84 scientists 85–94 publications: 102 scientists 95–104 publications: 151 scientists 105–114 publications: 175 scientists 115–124 publications: 203 scientists 125–134 publications: 217 scientists 135–144 publications: 205 scientists 145–154 publications: 193 scientists 155–164 publications: 188 scientists 165–174 publications: 170 scientists 175–184 publications: 178 scientists 185–194 publications: 164 scientists 195–204 publications: 173 scientists 205–214 publications: 159 scientists 215–224 publications: 134 scientists 225–234 publications: 143 scientists 235–244 publications: 105 scientists 245–254 publications: 114 scientists 255–264 publications: 92 scientists 265–274 publications: 88 scientists 275–284 publications: 87 scientists 285–294 publications: 80 scientists 295–304 publications: 62 scientists 305–314 publications: 75 scientists 315–324 publications: 67 scientists 325–334 publications: 60 scientists 335–344 publications: 52 scientists 345–354 publications: 40 scientists 355–364 publications: 48 scientists 365–374 publications: 47 scientists 375–384 publications: 46 scientists 385–394 publications: 31 scientists 395–404 publications: 27 scientists 405–414 publications: 40 scientists 415–424 publications: 30 scientists 425–434 publications: 43 scientists 435–444 publications: 29 scientists 445–454 publications: 14 scientists 455–464 publications: 28 scientists 465–474 publications: 21 scientists 475–484 publications: 21 scientists 485–494 publications: 22 scientists 495–504 publications: 17 scientists 505–514 publications: 12 scientists 515–524 publications: 11 scientists 525–534 publications: 8 scientists 535–544 publications: 8 scientists 545–554 publications: 14 scientists 555–564 publications: 4 scientists 565–574 publications: 11 scientists 575–584 publications: 5 scientists 585–594 publications: 11 scientists 595–604 publications: 12 scientists 605–614 publications: 7 scientists 615–624 publications: 6 scientists 625–634 publications: 10 scientists 635–644 publications: 9 scientists 645–654 publications: 10 scientists 655–664 publications: 6 scientists 665–674 publications: 6 scientists 675–684 publications: 6 scientists 685–694 publications: 4 scientists 695–702 publications: 6 scientists 703+ publications: 100 scientists
45 publications 703+

This scientist: 108 publications — 11th percentile

11% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 703 publications or more.

Sandy Chang D-index placement in Genetics in 2026

The chart shows the D-index (discipline H-index) distribution of Genetics scientists ranked by Research.com in 2026. The highlighted bar marks where Sandy Chang sits on this spectrum.

40–41 D-Index: 24 scientists 42–43 D-Index: 52 scientists 44–45 D-Index: 84 scientists 46–47 D-Index: 112 scientists 48–49 D-Index: 118 scientists 50–51 D-Index: 141 scientists 52–53 D-Index: 143 scientists 54–55 D-Index: 145 scientists 56–57 D-Index: 179 scientists 58–59 D-Index: 162 scientists 60–61 D-Index: 175 scientists 62–63 D-Index: 191 scientists 64–65 D-Index: 172 scientists 66–67 D-Index: 184 scientists 68–69 D-Index: 164 scientists 70–71 D-Index: 158 scientists 72–73 D-Index: 150 scientists 74–75 D-Index: 136 scientists 76–77 D-Index: 127 scientists 78–79 D-Index: 127 scientists 80–81 D-Index: 111 scientists 82–83 D-Index: 110 scientists 84–85 D-Index: 110 scientists 86–87 D-Index: 84 scientists 88–89 D-Index: 102 scientists 90–91 D-Index: 66 scientists 92–93 D-Index: 72 scientists 94–95 D-Index: 70 scientists 96–97 D-Index: 54 scientists 98–99 D-Index: 60 scientists 100–101 D-Index: 49 scientists 102–103 D-Index: 55 scientists 104–105 D-Index: 45 scientists 106–107 D-Index: 42 scientists 108–109 D-Index: 28 scientists 110–111 D-Index: 39 scientists 112–113 D-Index: 25 scientists 114–115 D-Index: 31 scientists 116–117 D-Index: 29 scientists 118–119 D-Index: 34 scientists 120–121 D-Index: 29 scientists 122–123 D-Index: 29 scientists 124–125 D-Index: 18 scientists 126–127 D-Index: 27 scientists 128–129 D-Index: 22 scientists 130–131 D-Index: 16 scientists 132–133 D-Index: 11 scientists 134–135 D-Index: 17 scientists 136–137 D-Index: 12 scientists 138–139 D-Index: 21 scientists 140–141 D-Index: 4 scientists 142–143 D-Index: 9 scientists 144–145 D-Index: 14 scientists 146–147 D-Index: 6 scientists 148–149 D-Index: 10 scientists 150–151 D-Index: 7 scientists 152–153 D-Index: 9 scientists 154–155 D-Index: 8 scientists 156–157 D-Index: 8 scientists 158–159 D-Index: 9 scientists 160+ D-Index: 96 scientists
40 D-Index 160+

This scientist: 60 D-Index — 29th percentile

29% of scientists in this discipline score the same or lower.

The last bar groups every scientist with 160 D-Index or more.

Overview

Sandy Chang is affiliated with Yale University in the United States. Their research primarily focuses on biochemistry, genetics, and molecular biology, with a significant number of publications in these areas. The scientist's work spans major subfields such as molecular biology, physiology, surgery, and oncology.

The core topics covered in their research include telomeres, telomerase, and senescence; DNA repair mechanisms; CRISPR and genetic engineering; nuclear structure and function; genomics and chromatin dynamics; advanced biosensing and bioanalysis techniques; and RNA interference and gene delivery.

Frequent publication venues for Sandy Chang include:

  • Nature Communications
  • Nucleic Acids Research
  • Current Opinion in Genetics & Development
  • BioEssays
  • Journal of Clinical Oncology

Some of the recent papers by Sandy Chang are:

  • "Distinct functions of POT1 proteins contribute to the regulation of telomerase recruitment to telomeres" (2021, Nature Communications)
  • "Shelterin and the replisome: at the intersection of telomere repair and replication" (2020, Current Opinion in Genetics & Development)
  • "Microcephalin 1/BRIT1-TRF2 interaction promotes telomere replication and repair, linking telomere dysfunction to primary microcephaly" (2020, Nature Communications)
  • "Homology directed telomere clustering, ultrabright telomere formation and nuclear envelope rupture in cells lacking TRF2B and RAP1" (2023, Nature Communications)
  • "Pot1b −/− tumors activate G-quadruplex-induced DNA damage to promote telomere hyper-elongation" (2023, Nucleic Acids Research)

The scientist has collaborated frequently with several coauthors, most notably:

  • Rekha Rai
  • Yong Chen
  • Tori Sodeinde
  • Wenqi Sun
  • Amer Al-Hiyasat

Their research exhibits a consistent focus on telomere biology, specifically exploring the mechanisms of telomerase regulation, telomere replication, repair, and associated cellular processes. Investigations also address the connection between telomere dysfunction and diseases such as primary microcephaly and tumor progression. Through their body of work, they contribute to a deeper understanding of DNA repair, genome stability, and cellular aging.

Best Publications

  • Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice

    Sunil R. Hingorani;Lifu Wang;Asha S. Multani;Chelsea Combs

  • Longevity, stress response, and cancer in aging telomerase-deficient mice.

    Karl Lenhard Rudolph;Sandy Chang;Han Woong Lee;Maria Blasco

  • Telomere dysfunction promotes non-reciprocal translocations and epithelial cancers in mice

    Steven E. Artandi;Sandy Chang;Sandy Chang;Shwu-Luan Lee;Scott Alson

  • Endogenous oncogenic K-rasG12D stimulates proliferation and widespread neoplastic and developmental defects

    David A Tuveson;Alice T Shaw;Alice T Shaw;Alice T Shaw;Nicholas A Willis;Daniel P Silver

  • Inhibition of experimental liver cirrhosis in mice by telomerase gene delivery.

    Karl Lenhard Rudolph;Sandy Chang;Melissa Millard;Nicole Schreiber-Agus

  • Mutations in the mineralocorticoid receptor gene cause autosomal dominant pseudohypoaldosteronism type I.

    D S Geller;J Rodriguez-Soriano;A Vallo Boado;S Schifter

  • Essential role of limiting telomeres in the pathogenesis of Werner syndrome.

    Sandy Chang;Asha S Multani;Noelia G Cabrera;Maria L Naylor

  • The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations.

    David O. Ferguson;JoAnn M. Sekiguchi;Sandy Chang;Karen M. Frank

  • Pot1 deficiency initiates DNA damage checkpoint activation and aberrant homologous recombination at telomeres.

    Ling Wu;Asha S. Multani;Hua He;Wilfredo Cosme-Blanco

  • Telomere dysfunction and tumour suppression: the senescence connection

    Yibin Deng;Suzanne S. Chan;Sandy Chang

  • Chromosome stability, in the absence of apoptosis, is critical for suppression of tumorigenesis in Trp53 mutant mice.

    Geng Liu;John M Parant;Gene Lang;Patty Chau

  • TERRA and hnRNPA1 orchestrate an RPA-to-POT1 switch on telomeric single-stranded DNA.

    Rachel Litman Flynn;Richard C. Centore;Roderick J. O’Sullivan;Rekha Rai

  • Telomere dysfunction impairs DNA repair and enhances sensitivity to ionizing radiation.

    Kwok Kin Wong;Sandy Chang;Sarah R. Weiler;Shridar Ganesan

  • Mre11 Nuclease Activity Has Essential Roles in DNA Repair and Genomic Stability Distinct from ATM Activation

    Jeffrey Buis;Yipin Wu;Yibin Deng;Jennifer Leddon

  • WRN helicase is a synthetic lethal target in microsatellite unstable cancers.

    Edmond M Chan;Edmond M Chan;Tsukasa Shibue;James M McFarland;Benjamin Gaeta

  • Telomere dysfunction provokes regional amplification and deletion in cancer genomes

    Rónán C O'Hagan;Sandy Chang;Richard S Maser;Ramya Mohan

  • Prelamin A and lamin A appear to be dispensable in the nuclear lamina

    Loren G. Fong;Jennifer K. Ng;Jan Lammerding;Timothy A. Vickers

  • Telomere dysfunction suppresses spontaneous tumorigenesis in vivo by initiating p53‐dependent cellular senescence

    Wilfredo Cosme-Blanco;Mei Feng Shen;Alexander J.F. Lazar;Sen Pathak

  • Deficiencies in lamin B1 and lamin B2 cause neurodevelopmental defects and distinct nuclear shape abnormalities in neurons.

    Catherine Coffinier;Hea-Jin Jung;Chika Nobumori;Sandy Chang

  • Dysfunctional telomeres activate an ATM-ATR-dependent DNA damage response to suppress tumorigenesis.

    Xiaolan Guo;Yibin Deng;Yahong Lin;Wilfredo Cosme-Blanco

Frequent Co-Authors

Asha S. Multani
Asha S. Multani The University of Texas MD Anderson Cancer Center
Ronald A. DePinho
Ronald A. DePinho The University of Texas MD Anderson Cancer Center
Sen Pathak
Sen Pathak The University of Texas MD Anderson Cancer Center
Linda P. Fried
Linda P. Fried Columbia University
Mary E. Tinetti
Mary E. Tinetti Yale University
Todd R. Golub
Todd R. Golub Harvard University
Steven E. Artandi
Steven E. Artandi Stanford University
Alice T. Shaw
Alice T. Shaw Harvard University
Lynda Chin
Lynda Chin The University of Texas Health Science Center at Houston
Ralph H. Hruban
Ralph H. Hruban Johns Hopkins University School of Medicine

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