World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
53
Citations
16837
World Ranking
15930
National Ranking
6616

Research.com Recognitions

  • 2007 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

Steven E. Artandi is affiliated with Stanford University in the United States. Their research primarily spans the fields of Biochemistry, Genetics and Molecular Biology, and Medicine, with a significant focus on Molecular Biology, Physiology, Genetics, Immunology, and Surgery as subfields.

The scientist's work addresses several main topics, including:

  • Telomeres, Telomerase, and Senescence
  • RNA Research and Splicing
  • CRISPR and Genetic Engineering
  • Single-cell and spatial transcriptomics
  • Pancreatic function and diabetes
  • RNA modifications and cancer
  • Diabetes and associated disorders

Among recent publications attributed to Steven E. Artandi are:

  • Regulation of human telomerase in homeostasis and disease, 2020, Nature Reviews Molecular Cell Biology
  • Targeted replacement of full-length CFTR in human airway stem cells by CRISPR-Cas9 for pan-mutation correction in the endogenous locus, 2021, Molecular Therapy
  • Acinar cell clonal expansion in pancreas homeostasis and carcinogenesis, 2021, Nature
  • Loss of Human TGS1 Hypermethylase Promotes Increased Telomerase RNA and Telomere Elongation, 2020, Cell Reports
  • Adversarial domain translation networks for integrating large-scale atlas-level single-cell datasets, 2022, Nature Computational Science

Frequent co-authors working alongside Steven E. Artandi include Lü Chen, Caitlin M. Roake, Patrick Neuhöfer, Jingsi Ming, and Roozbeh Dehghannasiri.

The scientist has published multiple articles in the following venues with notable frequency:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Nature
  • Cancer Research
  • Nature Communications
  • Diabetes

Steven E. Artandi's research includes studies related to telomerase regulation, CRISPR gene editing methodologies, pancreatic biology, and computational approaches in single-cell data integration.

In 2007, the scientist was recognized as a Fellow of the American Association for the Advancement of Science (AAAS).

Best Publications

  • Telomere dysfunction promotes non-reciprocal translocations and epithelial cancers in mice

    Steven E. Artandi;Sandy Chang;Sandy Chang;Shwu-Luan Lee;Scott Alson

  • Genomic maps of long noncoding RNA occupancy reveal principles of RNA-chromatin interactions.

    Ci Chu;Kun Qu;Franklin L. Zhong;Steven E. Artandi

  • p53 Deficiency Rescues the Adverse Effects of Telomere Loss and Cooperates with Telomere Dysfunction to Accelerate Carcinogenesis

    Lynda Chin;Steven E. Artandi;Qiong Shen;Alice Tam

  • Telomeres and telomerase in cancer.

    Steven E. Artandi;Ronald A. DePinho

  • Telomerase modulates Wnt signalling by association with target gene chromatin

    Jae Il Park;Andrew S. Venteicher;Ji Yeon Hong;Jinkuk Choi

  • A Human Telomerase Holoenzyme Protein Required for Cajal Body Localization and Telomere Synthesis

    Andrew S. Venteicher;Eladio B. Abreu;Zhaojing Meng;Kelly E. McCann

  • Short Telomeres and Stem Cell Exhaustion Model Duchenne Muscular Dystrophy in mdx/mTR Mice

    Alessandra Sacco;Foteini Mourkioti;Rose Tran;Jinkuk Choi

  • Conditional telomerase induction causes proliferation of hair follicle stem cells

    Kavita Y. Sarin;Peggie Cheung;Daniel Gilison;Eunice Lee

  • A critical role for telomeres in suppressing and facilitating carcinogenesis.

    Steven E Artandi;Ronald A DePinho

  • Identification of ATPases Pontin and Reptin as Telomerase Components Essential for Holoenzyme Assembly

    Andrew S. Venteicher;Zhaojing Meng;Philip J. Mason;Timothy D. Veenstra

  • Constitutive telomerase expression promotes mammary carcinomas in aging mice

    Steven E. Artandi;Steven E. Artandi;Scott Alson;Maja K. Tietze;Maja K. Tietze;Norman E. Sharpless

  • In Situ Genetic Correction of the Sickle Cell Anemia Mutation in Human Induced Pluripotent Stem Cells Using Engineered Zinc Finger Nucleases

    Vittorio Sebastiano;Morgan L. Maeder;James F. Angstman;Bahareh Haddad

  • TERT promotes epithelial proliferation through transcriptional control of a Myc- and Wnt-related developmental program.

    Jinkuk Choi;Lucinda K Southworth;Kavita Y Sarin;Andrew S Venteicher

  • Dual SMAD Signaling Inhibition Enables Long-Term Expansion of Diverse Epithelial Basal Cells.

    Hongmei Mou;Vladimir Vinarsky;Purushothama Rao Tata;Karissa Brazauskas

  • Telomere dysfunction impairs DNA repair and enhances sensitivity to ionizing radiation.

    Kwok Kin Wong;Sandy Chang;Sarah R. Weiler;Shridar Ganesan

  • Inhibition of transcriptional regulator Yin-Yang-1 by association with c-Myc

    A. Shrivastava;S. Saleque;Ganjam V. Kalpana;S. Artandi

  • TPP1 OB-Fold Domain Controls Telomere Maintenance by Recruiting Telomerase to Chromosome Ends

    Franklin L. Zhong;Luis F.Z. Batista;Adam Freund;Matthew F. Pech

  • Disruption of telomerase trafficking by TCAB1 mutation causes dyskeratosis congenita

    Franklin Zhong;Sharon A. Savage;Marina Shkreli;Neelam Giri

  • Telomere dysfunction provokes regional amplification and deletion in cancer genomes

    Rónán C O'Hagan;Sandy Chang;Richard S Maser;Ramya Mohan

  • Telomere shortening and loss of self-renewal in dyskeratosis congenita induced pluripotent stem cells

    Luis F. Z. Batista;Matthew F. Pech;Franklin L. Zhong;Ha Nam Nguyen

Frequent Co-Authors

Lu Chen
Lu Chen Stanford University
Ronald A. DePinho
Ronald A. DePinho The University of Texas MD Anderson Cancer Center
Kathryn Calame
Kathryn Calame Columbia University
Howard Y. Chang
Howard Y. Chang Amgen (United States)
Anne Brunet
Anne Brunet Stanford University
Pierre D. McCrea
Pierre D. McCrea The University of Texas MD Anderson Cancer Center
Timothy D. Veenstra
Timothy D. Veenstra Lake Erie College of Osteopathic Medicine
Lynda Chin
Lynda Chin The University of Texas Health Science Center at Houston
Sandy Chang
Sandy Chang Yale University
Sharon A. Savage
Sharon A. Savage National Institutes of Health

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