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Genetics

D-Index
62
Citations
11533
World Ranking
2998
National Ranking
371

Overview

William G. Wood is affiliated with the University of Oxford in the United Kingdom. Their research spans multiple areas within biochemistry, genetics, molecular biology, and medicine, with a focus on immunology, molecular biology, and cell biology. Their contributions also extend into epidemiology and pulmonary and respiratory medicine.

The scientist's work covers a variety of topics including:

  • Phagocytosis and Immune Regulation
  • Autophagy in Disease and Therapy
  • Cellular Mechanics and Interactions
  • Invertebrate Immune Response Mechanisms
  • Immune cells in cancer
  • Extracellular vesicles in disease
  • Advanced biosensing and bioanalysis techniques

Frequent coauthors of William G. Wood include Andrew J. Davidson, Clelia Amato, R. Heron, Luigi Zechini, and Alessandro Scopelliti. These collaborations have contributed to a cohesive body of work primarily published in several scientific journals.

Common publication venues for their work are:

  • Current Biology
  • Nature Cell Biology
  • Drug and Alcohol Review
  • International Journal of Molecular Sciences
  • Science

Among the recent papers authored or coauthored by Wood are:

  • The Apoptosis Paradox in Cancer, 2022, International Journal of Molecular Sciences
  • Live cell tracking of macrophage efferocytosis during Drosophila embryo development in vivo, 2022, Science
  • Macrophages Use Distinct Actin Regulators to Switch Engulfment Strategies and Ensure Phagocytic Plasticity In Vivo, 2020, Cell Reports
  • Piezo acts as a molecular brake on wound closure to ensure effective inflammation and maintenance of epithelial integrity, 2022, Current Biology
  • A conserved myotubularin-related phosphatase regulates autophagy by maintaining autophagic flux, 2020, The Journal of Cell Biology

Best Publications

  • Molecular basis for mild forms of homozygous beta-thalassaemia

    D J Weatherall;L Pressley;W G Wood;Higgs

  • Transcription of antisense RNA leading to gene silencing and methylation as a novel cause of human genetic disease.

    Cristina Tufarelli;Jackie A Sloane Stanley;David Garrick;Jackie A Sharpe

  • A Regulatory SNP Causes a Human Genetic Disease by Creating a New Transcriptional Promoter

    Marco De Gobbi;Vip Viprakasit;Jim R Hughes;Chris Fisher

  • Genetic dissection of the α-globin super-enhancer in vivo

    Deborah Hay;Jim R. Hughes;Christian Babbs;James O.J. Davies

  • Intragenic Enhancers Act as Alternative Promoters

    Monika S. Kowalczyk;Jim R. Hughes;David Garrick;Magnus D. Lynch

  • Association between active genes occurs at nuclear speckles and is modulated by chromatin environment.

    Jill M. Brown;Joanne Green;Ricardo Pires das Neves;Helen A.C. Wallace

  • Long-range chromosomal interactions regulate the timing of the transition between poised and active gene expression.

    Douglas Vernimmen;Marco De Gobbi;Jacqueline A Sloane‐Stanley;William G Wood

  • Globin gene activation during haemopoiesis is driven by protein complexes nucleated by GATA-1 and GATA-2

    Eduardo Anguita;Jim Hughes;Clare M Heyworth;Gerd A Blobel

  • Manipulating the mouse genome to engineer precise functional syntenic replacements with human sequence.

    Helen A.C. Wallace;Fatima Marques-Kranc;Melville Richardson;Francisco Luna-Crespo

  • Healing of broken human chromosomes by the addition of telomeric repeats.

    J Flint;C F Craddock;A Villegas;D P Bentley

  • Molecular basis for dominantly inherited inclusion body beta-thalassemia.

    S L Thein;C Hesketh;P Taylor;I J Temperley

  • Coregulated human globin genes are frequently in spatial proximity when active

    Jill M. Brown;Joanne Leach;Joyce E. Reittie;Ann Atzberger

  • Characterization of the major regulatory element upstream of the human alpha-globin gene cluster.

    A P Jarman;W G Wood;J A Sharpe;G Gourdon

  • Loss of Atrx Affects Trophoblast Development and the Pattern of X-Inactivation in Extraembryonic Tissues

    David Garrick;Jackie A Sharpe;Ruth M. Arkell;Lorraine Dobbie

  • A human PKD1 transgene generates functional polycystin-1 in mice and is associated with a cystic phenotype

    Lynn Pritchard;Jackie A. Sloane-Stanley;Jackie A. Sharpe;Richard Aspinwall

  • Identification of acquired somatic mutations in the gene encoding chromatin-remodeling factor ATRX in the alpha-thalassemia myelodysplasia syndrome (ATMDS).

    Richard J Gibbons;Andrea Pellagatti;David Garrick;William G Wood

  • Fetal haemoglobin production and the sickle gene in the oases of eastern Saudi Arabia.

    M. E. Pembrey;W. G. Wood;D. J. Weatherall;R. P. Perrine

  • Advances in the understanding of the congenital dyserythropoietic anaemias.

    Sunitha N. Wickramasinghe;William G. Wood

  • Alpha-thalassemia caused by a large (62 kb) deletion upstream of the human alpha globin gene cluster.

    C S Hatton;A O Wilkie;H C Drysdale;W G Wood

  • Long-range chromosomal interactions regulate the timing of the transition between poised and active gene expression

    D Vernimmen;M De Gobbi;J A Sloane-Stanley;W G Wood

Frequent Co-Authors

David J. Weatherall
David J. Weatherall University of Oxford
Douglas R. Higgs
Douglas R. Higgs University of Oxford
John B. Clegg
John B. Clegg John Radcliffe Hospital
James Hughes
James Hughes Emory University
Veronica J. Buckle
Veronica J. Buckle University of Oxford
Richard J. Gibbons
Richard J. Gibbons University of Oxford
Paresh Vyas
Paresh Vyas University of Oxford
Andrew J.H. Smith
Andrew J.H. Smith University of Edinburgh
Swee Lay Thein
Swee Lay Thein King's College London
Emmanuel Kanavakis
Emmanuel Kanavakis National and Kapodistrian University of Athens

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