Cell biology, Antigen, Cytotoxic T cell, Immunology and Immune system are his primary areas of study. His studies in Cell biology integrate themes in fields like Natural killer T cell, T cell, Interleukin 21, Antigen-presenting cell and Apoptosis. His work in Antigen covers topics such as T-cell receptor which are related to areas like Adoptive cell transfer.
His Cytotoxic T cell research integrates issues from Progenitor cell, Spleen, CD8 and Lymphocyte. Richard G. Miller interconnects Molecular biology, Endocrinology, In vitro and Transplantation in the investigation of issues within Immune system. His Molecular biology research is multidisciplinary, relying on both Precursor cell and Pathology.
Richard G. Miller spends much of his time researching Molecular biology, Immunology, Cytotoxic T cell, T cell and Antigen. His Molecular biology research includes themes of Cell, In vitro, Bone marrow and In vivo. His study in Cytotoxic T cell is interdisciplinary in nature, drawing from both Spleen and Mixed lymphocyte reaction.
His T cell study combines topics in areas such as Cell culture, B cell and Cell biology. The concepts of his Cell biology study are interwoven with issues in Apoptosis, Receptor and Fluorescence. His work carried out in the field of Interleukin 21 brings together such families of science as Natural killer T cell, Perforin and Interleukin 12.
The scientist’s investigation covers issues in Cell biology, Immunology, Lymphokine-activated killer cell, Interleukin 21 and T cell. His Cell biology research is multidisciplinary, incorporating perspectives in Perforin, Caspase, Programmed cell death and Endogeny. He has included themes like Receptor and Disease in his Immunology study.
As a member of one scientific family, Richard G. Miller mostly works in the field of Lymphokine-activated killer cell, focusing on Natural killer cell and, on occasion, Molecular biology, Granzyme and Granzyme M. Richard G. Miller specializes in T cell, namely Antigen-presenting cell. His Antigen-presenting cell research is within the category of Cytotoxic T cell.
Richard G. Miller mainly focuses on Interleukin 21, Lymphokine-activated killer cell, Cell biology, Interleukin 12 and T cell. His Cell biology research includes elements of Virology, Viability assay, NKG2D and Perforin, Granzyme. His studies deal with areas such as Natural killer T cell and Antigen-presenting cell as well as Virology.
Richard G. Miller has researched Viability assay in several fields, including MHC class I, Immunology and Immunosurveillance. His NKG2D study frequently draws connections to adjacent fields such as Natural killer cell. By researching both Granzyme and Cancer immunotherapy, Richard G. Miller produces research that crosses academic boundaries.
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Separation of cells by velocity sedimentation.
Richard G. Miller;R. A. Phillips.
Journal of Cellular Physiology (1969)
Normal development and function of CD8+ cells but markedly decreased helper cell activity in mice lacking CD4.
A. Rahemtulla;W. P. Fung-Leung;M. W. Schilham;T. M. Kündig.
Nature (1991)
Rapamycin Enhances Apoptosis and Increases Sensitivity to Cisplatin in Vitro
Yufang Shi;Andrea Frankel;Laszlo G. Radvanyi;Linda Z. Penn.
Cancer Research (1995)
Programmed cell death of T cells signaled by the T cell receptor and the alpha 3 domain of class I MHC.
Suryaprakash R. Sambhara;Richard G. Miller.
Science (1991)
Activated, But Not Resting, T Cells Can Be Recognized and Killed by Syngeneic NK Cells
Brian A. Rabinovich;Jennifer Li;John Shannon;Rose Hurren.
Journal of Immunology (2003)
An immunological suppressor cell inactivating cytotoxic T-lymphocyte precursor cells recognizing it.
Richard G. Miller.
Nature (1980)
CD28 costimulation inhibits TCR-induced apoptosis during a primary T cell response
Laszlo G. Radvanyi;Yufang Shi;Homayoun Vaziri;Ajay Sharma.
Journal of Immunology (1996)
Oral Beta-Carotene Can Increase the Number of OKT4+ Cells in Human Blood
Michael Alexander;Harold Newmark;Richard G. Miller.
Immunology Letters (1985)
Hoechst 33342 dye uptake as a probe of membrane permeability changes in mammalian cells
Marc E. Lalande;Victor Ling;Richard G. Miller.
Proceedings of the National Academy of Sciences of the United States of America (1981)
Fas (CD95) participates in peripheral T cell deletion and associated apoptosis in vivo
Rona J. Mogil;Laszlo Radvanyi;Rosana Gonzalez-quintial;Rosana Gonzalez-quintial;Richard Miller.
International Immunology (1995)
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