Poul H. Sorensen mostly deals with Molecular biology, Cancer research, Sarcoma, Genetics and Rhabdomyosarcoma. His biological study spans a wide range of topics, including Plasma protein binding, Gene expression, Ribonucleoprotein, Transcription factor and Amplicon. His Cancer research research incorporates elements of Carcinogenesis, ETV6, Gene, Chromosomal translocation and Immunology.
His research in Sarcoma intersects with topics in Osteosarcoma, Fusion gene, Metastasis and Medulloblastoma. Cancer is closely connected to Human genetics in his research, which is encompassed under the umbrella topic of Medulloblastoma. His study in Rhabdomyosarcoma is interdisciplinary in nature, drawing from both Fluorescence in situ hybridization, Internal medicine, Oncology and PAX3.
His primary areas of study are Cancer research, Cell biology, Sarcoma, Molecular biology and Cancer. His Cancer research research includes themes of Tyrosine kinase, Pediatric cancer and Metastasis. His research integrates issues of Biochemistry, Ubiquitin ligase and Transcription factor in his study of Cell biology.
The various areas that Poul H. Sorensen examines in his Sarcoma study include Oxidative stress and Genetics, Gene. His Molecular biology study incorporates themes from Fluorescence in situ hybridization, Protein kinase B and ETV6. His Pathology research is multidisciplinary, relying on both Fusion gene, Internal medicine, Oncology and Cytogenetics.
Poul H. Sorensen mainly focuses on Cancer research, Cell biology, Cancer, Metastasis and Sarcoma. Poul H. Sorensen interconnects Pediatric cancer, Downregulation and upregulation and Immunotherapy in the investigation of issues within Cancer research. His Cell biology study combines topics in areas such as Carcinogenesis, RNA, Transcription factor, Alternative splicing and Stress granule.
His work deals with themes such as Reverse transcription polymerase chain reaction, Gene, Protein family and Tropomyosin, which intersect with Cancer. He has researched Metastasis in several fields, including Cancer cell, Germline, DNA sequencing and Protein biosynthesis. His work carried out in the field of Sarcoma brings together such families of science as Oxidative stress, Immune system, HIF1A and Puma.
His primary scientific interests are in Cancer research, Cell biology, Cancer, Immunotherapy and Medulloblastoma. His biological study spans a wide range of topics, including Immunohistochemistry, Epithelial–mesenchymal transition, Ependymoma, Epigenetics and Epigenome. His Cell biology research is multidisciplinary, incorporating elements of Oxidative stress, Transcription factor and Y box binding protein 1.
His Cancer research integrates issues from mTORC1, Protein biosynthesis, Untranslated region, Stress granule and EIF4E. The various areas that Poul H. Sorensen examines in his Immunotherapy study include Chemotherapy, Carcinogenesis, Antigen, Proinflammatory cytokine and Radiation therapy. His studies in Medulloblastoma integrate themes in fields like Cell, Chimeric antigen receptor, Cerebrospinal fluid and Pediatric cancer.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A second Ewing's sarcoma translocation, t(21;22), fuses the EWS gene to another ETS–family transcription factor, ERG
Poul H.B. Sorensen;Stephen L. Lessnick;Dolores Lopez-Terrada;Xian F. Liu.
Nature Genetics (1994)
Expression of the ETV6-NTRK3 gene fusion as a primary event in human secretory breast carcinoma.
Cristina Tognon;Stevan R Knezevich;David Huntsman;Calvin D Roskelley.
Cancer Cell (2002)
Lin28 promotes transformation and is associated with advanced human malignancies
Srinivas R Viswanathan;John T Powers;William Einhorn;Yujin Hoshida.
Nature Genetics (2009)
PAX3-FKHR and PAX7-FKHR Gene Fusions Are Prognostic Indicators in Alveolar Rhabdomyosarcoma: A Report From the Children’s Oncology Group
Poul H B Sorensen;James C Lynch;Stephen J Qualman;Roberto Tirabosco.
Journal of Clinical Oncology (2002)
A novel ETV6-NTRK3 gene fusion in congenital fibrosarcoma
Stevan R. Knezevich;Deborah E. McFadden;Wen Tao;Jerian F. Lim.
Nature Genetics (1998)
Targeted disruption of SHIP leads to hemopoietic perturbations, lung pathology, and a shortened life span
Helgason Cd;Damen Je;Rosten P;Grewal R.
Genes & Development (1998)
Subgroup-specific structural variation across 1,000 medulloblastoma genomes
Paul A. Northcott;Paul A. Northcott;David J.H. Shih;John Peacock;Livia Garzia.
Nature (2012)
EWS-FLI1 fusion transcript structure is an independent determinant of prognosis in Ewing's sarcoma.
E. De Alava;A. Kawai;J. H. Healey;I. Fligman.
Journal of Clinical Oncology (1998)
The der(17)t(x;17)(p11;q25) of human alveolar soft part sarcoma fuses the TFE3 transcription factor gene to ASPL, a novel gene at 17q25
Marc Ladanyi;Man Yee Lui;Cristina R Antonescu;Amber Krause-Boehm.
Oncogene (2001)
Translational Activation of Snail1 and Other Developmentally Regulated Transcription Factors by YB-1 Promotes an Epithelial-Mesenchymal Transition
Valentina Evdokimova;Cristina Tognon;Tony Ng;Peter Ruzanov.
Cancer Cell (2009)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
Van Andel Institute
University of British Columbia
University of Toronto
German Cancer Research Center
PSL University
Children's Hospital of Philadelphia
University of British Columbia
University of British Columbia
University of South Australia
McGill University
University of Bern
Chinese University of Hong Kong
National Institute of Standards and Technology
Curtin University
University of Illinois at Urbana-Champaign
University of Queensland
The Ohio State University
University of Vienna
Virginia Tech
Flinders University
University of Birmingham
Ottawa Hospital
Washington University in St. Louis
Tufts University
University College London
University of Birmingham