World's Best Scientists 2026 revealed!
Ken Ichi Yamamura

Ken Ichi Yamamura

D-Index & Metrics

Genetics

D-Index
78
Citations
30444
World Ranking
1677
National Ranking
62

Molecular Biology

D-Index
78
Citations
30444
World Ranking
1062
National Ranking
85

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • DNA

Ken Ichi Yamamura mainly focuses on Molecular biology, Internal medicine, Endocrinology, Gene and Immunology. His Molecular biology research includes elements of Gene expression, Genetically modified mouse, Transgene, Cell growth and Antibody. Ken Ichi Yamamura combines subjects such as Epididymis, CREB-binding protein, CREB and Antigen with his study of Internal medicine.

His study in Endocrinology is interdisciplinary in nature, drawing from both Rete testis, Efferent ducts, Male genital duct and Sperm motility. His studies in Gene integrate themes in fields like Virus and Hepatitis B virus. Ken Ichi Yamamura has researched Immunology in several fields, including Stem cell and Cell biology.

His most cited work include:

  • Establishment of a pancreatic beta cell line that retains glucose-inducible insulin secretion: special reference to expression of glucose transporter isoforms (1029 citations)
  • Globular Adiponectin Protected ob/ob Mice from Diabetes and ApoE-deficient Mice from Atherosclerosis (827 citations)
  • IgG1 plasmacytosis in interleukin 6 transgenic mice (517 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Molecular biology, Genetically modified mouse, Gene, Internal medicine and Endocrinology. Ken Ichi Yamamura works mostly in the field of Molecular biology, limiting it down to topics relating to Mutant and, in certain cases, Mutation. The study incorporates disciplines such as Polyneuropathy, Transthyretin, Messenger RNA and Immunology in addition to Genetically modified mouse.

His work deals with themes such as Amyloid and Amyloidosis, which intersect with Transthyretin. His study on Gene is covered under Genetics. In his work, Embryonic stem cell is strongly intertwined with Cell biology, which is a subfield of Endocrinology.

He most often published in these fields:

  • Molecular biology (36.48%)
  • Genetically modified mouse (26.38%)
  • Gene (22.80%)

What were the highlights of his more recent work (between 2012-2021)?

  • Internal medicine (19.54%)
  • Endocrinology (18.89%)
  • Cell biology (17.26%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Internal medicine, Endocrinology, Cell biology, Gene and Molecular biology. In the field of Internal medicine, his study on Fatty liver, Pulmonary artery, Aorta and Ventricle overlaps with subjects such as Scanning electron microscope. His Endocrinology research includes themes of Citrin, Citrulline, Biochemistry, Immunoglobulin M and Inner plexiform layer.

His work carried out in the field of Cell biology brings together such families of science as BMPR2, Bone morphogenetic protein, Neurodegeneration, Sensory neuron and Genetically modified mouse. His Mutagenesis, Gene trapping and Exon study in the realm of Gene interacts with subjects such as Trap. His research integrates issues of Histone H3, Haematopoiesis, Phenotype, Citrullination and Mutant in his study of Molecular biology.

Between 2012 and 2021, his most popular works were:

  • Obesity-Associated Autoantibody Production Requires AIM to Retain the Immunoglobulin M Immune Complex on Follicular Dendritic Cells (73 citations)
  • Augmentation of smad‐dependent BMP signaling in neural crest cells causes craniosynostosis in mice (66 citations)
  • Conformational Change in Transfer RNA Is an Early Indicator of Acute Cellular Damage (65 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

Cell biology, Internal medicine, Molecular biology, Mutation and Genetics are his primary areas of study. His work deals with themes such as Peripheral nervous system, Neurodegeneration and Sensory neuron, which intersect with Cell biology. His study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology, CREB and DNA damage.

His Endocrinology research is multidisciplinary, incorporating perspectives in RNA, Oxidative phosphorylation and Fragmentation. His Molecular biology research is multidisciplinary, relying on both Histone H3, Haematopoiesis, Multipotent Stem Cell, Progenitor cell and Citrullination. He combines subjects such as Phenotype, PDE4B, Mutant and Protein kinase A with his study of Mutation.

Best Publications

  • Efficient selection for high-expression transfectants with a novel eukaryotic vector

    Hitoshi Niwa;Ken-ichi Yamamura;Jun-ichi Miyazaki

  • Establishment of a pancreatic beta cell line that retains glucose-inducible insulin secretion: special reference to expression of glucose transporter isoforms

    Jun Ichi Miyazaki;Kimi Araki;Eiji Yamato;Hiroshi Ikegami

  • Globular Adiponectin Protected ob/ob Mice from Diabetes and ApoE-deficient Mice from Atherosclerosis

    Toshimasa Yamauchi;Junji Kamon;Hironori Waki;Yasushi Imai

  • IgG1 plasmacytosis in interleukin 6 transgenic mice

    Sachiko Suematsu;Tadashi Matsuda;Katsuyuki Aozasa;Shizuo Akira

  • Targeted pharmacological depletion of serum amyloid P component for treatment of human amyloidosis

    M. B. Pepys;J. Herbert;W. L. Hutchinson;G. A. Tennent

  • Fgf8 is required for pharyngeal arch and cardiovascular development in the mouse.

    Radwan Abu-Issa;Graham Smyth;Ida Smoak;Ken Ichi Yamamura

  • Increased insulin sensitivity and hypoglycaemia in mice lacking the p85α subunit of phosphoinositide 3–kinase

    Y. Terauchi;Y. Tsuji;S. Satoh;H. Minoura

  • Exhaustion of nucleus pulposus progenitor cells with ageing and degeneration of the intervertebral disc

    Daisuke Sakai;Yoshihiko Nakamura;Tomoko Nakai;Taishi Mishima

  • Truncated CBP Protein Leads to Classical Rubinstein—Taybi Syndrome Phenotypes in Mice: Implications for a Dominant-Negative Mechanism

    Yuichi Oike;Akira Hata;Takayoshi Mamiya;Tadashi Kaname

  • Prevention of autoimmune insulitis by expression of I-E molecules in NOD mice

    Hirofumi Nishimoto;Hitoshi Kikutani;Ken-ichi Yamamura;Tadamitsu Kishimoto

  • A Null Mutation in Basigin, an Immunoglobulin Superfamily Member, Indicates Its Important Roles in Peri-implantation Development and Spermatogenesis

    Tadahiko Igakura;Kenji Kadomatsu;Tadashi Kaname;Tadashi Kaname;Hisako Muramatsu

  • Dysfunction of fibroblasts of extrarenal origin underlies renal fibrosis and renal anemia in mice

    Nariaki Asada;Masayuki Takase;Jin Nakamura;Akiko Oguchi

  • The mammalian gene function resource: the international knockout mouse consortium

    Allan Bradley;Konstantinos Anastassiadis;Abdelkader Ayadi;James F. Battey

  • Generation of plasmacytomas with the chromosomal translocation t(12;15) in interleukin 6 transgenic mice.

    Sachiko Suematsu;Taiji Matsusaka;Tadashi Matsuda;Shinsuke Ohno

  • Direct evidence for the contribution of the unique I-ANOD to the development of insulitis in non-obese diabetic mice

    Toru Miyazaki;Masashi Uno;Masahiro Uehira;Hitoshi Kikutani

  • Involvement of autophagy in trypsinogen activation within the pancreatic acinar cells.

    Daisuke Hashimoto;Masaki Ohmuraya;Masahiko Hirota;Akitsugu Yamamoto

  • Expression and replication of hepatitis B virus genome in transgenic mice

    Kimi Araki;Jun-Ichi Miyazaki;Okio Hino;Naohiro Tomita

  • Osteoarthritis associated with mild chondrodysplasia in transgenic mice expressing alpha 1(IX) collagen chains with a central deletion

    Ken Nakata;Keiro Ono;Jun Ichi Miyazaki;Bjorn Reino Olsen

  • Mice Homozygous for a Truncated Form of CREB-Binding Protein Exhibit Defects in Hematopoiesis and Vasculo-angiogenesis

    Yuichi Oike;Nobuyuki Takakura;Nobuyuki Takakura;Akira Hata;Akira Hata;Tadashi Kaname;Tadashi Kaname

  • The mode of hepatitis B virus DNA integration in chromosomes of human hepatocellular carcinoma.

    T Nagaya;T Nakamura;T Tokino;T Tsurimoto

Frequent Co-Authors

Kimi Araki
Kimi Araki Kumamoto University
Misao Suzuki
Misao Suzuki Kumamoto University
Naomi Nakagata
Naomi Nakagata Kumamoto University
Kiyoshi Takahashi
Kiyoshi Takahashi Kumamoto University
Hideo Baba
Hideo Baba Kumamoto University
Jun-ichi Miyazaki
Jun-ichi Miyazaki Osaka University
Yuichi Oike
Yuichi Oike Kumamoto University
Okio Hino
Okio Hino Juntendo University
Motohiro Takeya
Motohiro Takeya Kumamoto Health Science University

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