What is he best known for?
The fields of study he is best known for:
Molecular biology, Carcinogenesis, TSC2, Cancer research and Tumor suppressor gene are his primary areas of study.
Okio Hino combines subjects such as Virology, Complementary DNA, Gene, Polymerase chain reaction and Hepatitis B virus with his study of Molecular biology.
His Carcinogenesis research is multidisciplinary, incorporating elements of Oxidative stress, Transgene, Adenoma and Wild type, Mutant.
Okio Hino has included themes like TSC1, Germline mutation and Tuberous sclerosis in his TSC2 study.
His Cancer research study integrates concerns from other disciplines, such as Mutation, Mitogen-activated protein kinase, Transcription factor and Loss of heterozygosity.
His work carried out in the field of Tumor suppressor gene brings together such families of science as Tumor progression and Locus.
His most cited work include:
- Autophagy-deficient mice develop multiple liver tumors (817 citations)
- Tsc tumour suppressor proteins antagonize amino-acid-TOR signalling. (571 citations)
- Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells (412 citations)
What are the main themes of his work throughout his whole career to date?
Okio Hino mostly deals with Cancer research, Molecular biology, Pathology, Carcinogenesis and TSC2.
The study incorporates disciplines such as Oncogene and Loss of heterozygosity in addition to Cancer research.
His Molecular biology research includes elements of Mutation, Gene expression, Gene, Virology and Genetically modified mouse.
The Carcinogenesis study combines topics in areas such as Endocrinology, Kidney and Transgene.
The concepts of his TSC2 study are interwoven with issues in TSC1, Tuberous sclerosis, Mutant and Germline mutation.
His Mesothelioma research incorporates themes from Internal medicine and Oncology.
He most often published in these fields:
- Cancer research (25.75%)
- Molecular biology (25.75%)
- Pathology (23.65%)
What were the highlights of his more recent work (between 2011-2021)?
- Cancer research (25.75%)
- Pathology (23.65%)
- Mesothelioma (12.28%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Cancer research, Pathology, Mesothelioma, Internal medicine and Cancer.
His research in Cancer research intersects with topics in Cell, Germline mutation, Gene knockdown, Cancer cell and TSC2.
His TSC2 research entails a greater understanding of Cell biology.
His study in the fields of Immunohistochemistry and Carcinoma under the domain of Pathology overlaps with other disciplines such as Restitution.
His study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology and Oncology.
His research investigates the connection between Flow cytometry and topics such as Monoclonal antibody that intersect with problems in Molecular biology.
Between 2011 and 2021, his most popular works were:
- Rapamycin reverses impaired social interaction in mouse models of tuberous sclerosis complex. (129 citations)
- Fenton Reaction Induced Cancer in Wild Type Rats Recapitulates Genomic Alterations Observed in Human Cancer (60 citations)
- Prevention of alveolar destruction and airspace enlargement in a mouse model of pulmonary lymphangioleiomyomatosis (LAM) (45 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Pathology, Cancer research, Mesothelin, Internal medicine and Cancer.
In general Pathology, his work in Mesothelioma, Immunohistochemistry and Lymphangioleiomyomatosis is often linked to Vascular endothelial growth factor linking many areas of study.
His studies deal with areas such as Cell, Tuberous sclerosis, Mesenchymal stem cell and Small GTPase as well as Cancer research.
His Mesothelin research includes themes of Endothelium and Prognostic factor.
His study looks at the relationship between Internal medicine and fields such as Oncology, as well as how they intersect with chemical problems.
His studies examine the connections between Haploinsufficiency and genetics, as well as such issues in TSC2, with regards to Carcinogenesis.
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