Biochemistry, Molecular biology, ADP ribosylation factor, Lymphangioleiomyomatosis and Pathology are his primary areas of study. His research in Biochemistry tackles topics such as Cholera toxin which are related to areas like NAD+ kinase. His Molecular biology research is multidisciplinary, incorporating elements of Nitric oxide synthase, Transcriptional regulation and Complementary DNA, Gene, Transfection.
His ADP ribosylation factor study also includes
Joel Moss focuses on Biochemistry, Molecular biology, Lymphangioleiomyomatosis, ADP ribosylation factor and Pathology. His Biochemistry study frequently intersects with other fields, such as Cholera toxin. Joel Moss has researched Molecular biology in several fields, including Amino acid and Complementary DNA, Protein subunit, Gene, Messenger RNA.
The study incorporates disciplines such as Pulmonary function testing and Sirolimus in addition to Lymphangioleiomyomatosis. Joel Moss usually deals with ADP ribosylation factor and limits it to topics linked to Brefeldin A and Guanine nucleotide exchange factor. In his work, Pertussis toxin is strongly intertwined with Cyclase, which is a subfield of NAD+ kinase.
Joel Moss mainly focuses on Lymphangioleiomyomatosis, Tuberous sclerosis, Pathology, Sirolimus and TSC2. His Lymphangioleiomyomatosis study results in a more complete grasp of Internal medicine. His work carried out in the field of Internal medicine brings together such families of science as Gastroenterology and Oncology.
His Tuberous sclerosis research incorporates themes from Genetics, Lesion, Dermatology, Angiofibroma and Fibrofolliculoma. His Pathology research includes elements of Buttocks and Cell. The TSC2 study combines topics in areas such as Cancer research, TSC1 and Mesenchymal stem cell.
His primary scientific interests are in Lymphangioleiomyomatosis, Tuberous sclerosis, Pathology, Sirolimus and TSC2. His research on Internal medicine and Lung is centered around Lymphangioleiomyomatosis. His Tuberous sclerosis research incorporates elements of Cohort study, Linkage disequilibrium, Chromosome, Gene and Cohort.
His Pathology study integrates concerns from other disciplines, such as Dermatology and Pulmonary carcinoid tumour. His TSC2 study incorporates themes from Penetrance, Cancer research, TSC1 and Family history. His biological study spans a wide range of topics, including Arginine and ADP-ribosylation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Intracellular processing of endothelial nitric oxide synthase isoforms associated with differences in severity of cardiopulmonary diseases: Cleavage of proteins with aspartate vs. glutamate at position 298
M. Tesauro;W. C. Thompson;P. Rogliani;L. Qi.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Idiopathic nonspecific interstitial pneumonia: prognostic significance of cellular and fibrosing patterns: survival comparison with usual interstitial pneumonia and desquamative interstitial pneumonia.
William D. Travis;Kazuhiro Matsui;Joel Moss;Victor J. Ferrans.
The American Journal of Surgical Pathology (2000)
Pertussis toxin-catalyzed ADP-ribosylation of transducin. Cysteine 347 is the ADP-ribose acceptor site.
R. E. West;J. Moss;M. Vaughan;T. Liu.
Journal of Biological Chemistry (1985)
Evidence for a Common Mechanism of SIRT1 Regulation by Allosteric Activators
Basil P. Hubbard;Ana P. Gomes;Ana P. Gomes;Han Dai;Jun Li.
Science (2013)
Molecules in the ARF orbit.
Joel Moss;Martha Vaughan.
Journal of Biological Chemistry (1998)
Cytokine-mediated Transcriptional Induction of the Human Inducible Nitric Oxide Synthase Gene Requires Both Activator Protein 1 and Nuclear Factor κB-binding Sites
Joanna Marks-Konczalik;Shan C. Chu;Joel Moss.
Journal of Biological Chemistry (1998)
STRUCTURE AND FUNCTION OF ARF PROTEINS : ACTIVATORS OF CHOLERA TOXIN AND CRITICAL COMPONENTS OF INTRACELLULAR VESICULAR TRANSPORT PROCESSES
Joel Moss;Martha Vaughan.
Journal of Biological Chemistry (1995)
β2-Adrenergic receptor regulation by GIT1, a G protein-coupled receptor kinase-associated ADP ribosylation factor GTPase-activating protein
Richard T. Premont;Audrey Claing;Nicolas Vitale;Jennifer L. R. Freeman.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Pulmonary nontuberculous mycobacterial disease: prospective study of a distinct preexisting syndrome.
Richard D. Kim;David E. Greenberg;Mary E. Ehrmantraut;Shireen V. Guide.
American Journal of Respiratory and Critical Care Medicine (2008)
Oxidation of either Methionine 351 or Methionine 358 in α1-Antitrypsin Causes Loss of Anti-neutrophil Elastase Activity
Clifford Taggart;Daniel Cervantes-Laurean;Geumsoo Kim;Noel G. McElvaney.
Journal of Biological Chemistry (2000)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
National Institutes of Health
Nagasaki University
National Institutes of Health
National Institutes of Health
University of Cincinnati
University of Virginia
Kennedy Krieger Institute
Centre national de la recherche scientifique, CNRS
Brigham and Women's Hospital
Memorial Sloan Kettering Cancer Center
University of Brasília
University of Connecticut
University of Toronto
Okayama University
Xi'an Jiaotong University
Jilin University
King Abdullah University of Science and Technology
Ludwig-Maximilians-Universität München
Oregon State University
Los Alamos National Laboratory
Los Alamos National Laboratory
National Center for Atmospheric Research
Goddard Space Flight Center
University of Pittsburgh
University of Regensburg
Universidade de São Paulo