D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 89 Citations 29,079 325 World Ranking 685 National Ranking 23
Medicine D-index 95 Citations 33,919 412 World Ranking 6128 National Ranking 177

Research.com Recognitions

Awards & Achievements

2006 - ACM Senior Member

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Cytokine

His scientific interests lie mostly in Immunology, Inflammation, Macrophage, Granulocyte macrophage colony-stimulating factor and Internal medicine. Arthritis, Tumor necrosis factor alpha, Cytokine, Rheumatoid arthritis and Immune system are the subjects of his Immunology studies. His Inflammation study combines topics from a wide range of disciplines, such as Cancer, Immunotherapy, Cancer research and Encephalomyelitis, Multiple sclerosis.

His Macrophage study combines topics in areas such as Colony-stimulating factor, Granulocyte, Proinflammatory cytokine, Plasminogen activator and In vivo. In his study, which falls under the umbrella issue of Colony-stimulating factor, Cell biology, Cellular differentiation, Computational biology and Macrophage polarization is strongly linked to Macrophage colony-stimulating factor. His studies in Internal medicine integrate themes in fields like Endocrinology and Cycloheximide.

His most cited work include:

  • Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines (2761 citations)
  • Colony-stimulating factors in inflammation and autoimmunity. (952 citations)
  • The TIGR Rice Genome Annotation Resource: Improvements and New Features (866 citations)

What are the main themes of his work throughout his whole career to date?

John A. Hamilton mostly deals with Immunology, Internal medicine, Molecular biology, Cell biology and Endocrinology. His Immunology research is multidisciplinary, relying on both Colony-stimulating factor and Macrophage. His Molecular biology research includes themes of Phosphorylation and DNA synthesis.

His work carried out in the field of Cell biology brings together such families of science as Macrophage colony-stimulating factor, Biochemistry and Growth factor. The Endocrinology study combines topics in areas such as Synovial Cell, Cytokine and Synovial membrane. His Cytokine research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha and Monocyte.

He most often published in these fields:

  • Immunology (27.21%)
  • Internal medicine (20.97%)
  • Molecular biology (18.72%)

What were the highlights of his more recent work (between 2015-2020)?

  • Immunology (27.21%)
  • Inflammation (14.73%)
  • Arthritis (7.45%)

In recent papers he was focusing on the following fields of study:

John A. Hamilton mainly focuses on Immunology, Inflammation, Arthritis, Cytokine and Tumor necrosis factor alpha. John A. Hamilton has included themes like Colony-stimulating factor, Macrophage, Granulocyte macrophage colony-stimulating factor and Monocyte in his Inflammation study. His Colony-stimulating factor research is multidisciplinary, incorporating elements of Progenitor cell and Clinical trial.

His Macrophage study integrates concerns from other disciplines, such as IRF4 and Cancer research. The various areas that John A. Hamilton examines in his Cancer research study include Macrophage colony-stimulating factor, Signal transduction and Immune system. His Tumor necrosis factor alpha research incorporates elements of Receptor and Nociceptor.

Between 2015 and 2020, his most popular works were:

  • Anti-colony-stimulating factor therapies for inflammatory and autoimmune diseases (93 citations)
  • Immune Cytokines and Their Receptors in Inflammatory Pain (79 citations)
  • Granulocyte macrophage colony-stimulating factor induces CCL17 production via IRF4 to mediate inflammation (72 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Cytokine

Inflammation, Immunology, Macrophage, Arthritis and Cytokine are his primary areas of study. His Inflammation study incorporates themes from Colony-stimulating factor, Porphyromonas gingivalis, Granulocyte macrophage colony-stimulating factor and Bioinformatics. His Immunology research incorporates themes from In vitro, Disease and Granulocyte colony-stimulating factor.

The Macrophage study combines topics in areas such as IRF4 and Cancer research. The various areas that John A. Hamilton examines in his Arthritis study include Tumor necrosis factor alpha, Cellular differentiation and Immune system. His Cytokine research includes elements of Chemokine, CXCL1, Monocyte and Cell biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines

Peter J Murray;Judith E Allen;Subhra K Biswas;Edward A Fisher.
Immunity (2014)

4506 Citations

Colony-stimulating factors in inflammation and autoimmunity.

John A. Hamilton.
Nature Reviews Immunology (2008)

1666 Citations

The TIGR Rice Genome Annotation Resource: Improvements and New Features

Shu Ouyang;Wei Zhu;John A. Hamilton;Haining Lin.
Nucleic Acids Research (2007)

1170 Citations

Potential antiinflammatory effects of interleukin 4: suppression of human monocyte tumor necrosis factor alpha, interleukin 1, and prostaglandin E2

Prue H. Hart;Gerard F. Vitti;Diana R. Burgess;Genevieve A. Whitty.
Proceedings of the National Academy of Sciences of the United States of America (1989)

1045 Citations

Granulocyte-Macrophage Colony-Stimulating Factor (CSF) and Macrophage CSF-Dependent Macrophage Phenotypes Display Differences in Cytokine Profiles and Transcription Factor Activities: Implications for CSF Blockade in Inflammation

Andrew J. Fleetwood;Toby Lawrence;John A. Hamilton;Andrew D. Cook.
Journal of Immunology (2007)

705 Citations

Macrophage plasminogen activator: Modulation of enzyme production by anti-inflammatory steroids, mitotic inhibitors, and cyclic nucleotides

Jean-Dominique Vassalli;John Hamilton;E. Reich.
Cell (1976)

557 Citations

Defining GM-CSF– and Macrophage-CSF–Dependent Macrophage Responses by In Vitro Models

Derek C Lacey;Adrian Achuthan;Andrew J Fleetwood;Hang Dinh.
Journal of Immunology (2012)

541 Citations

GM-CSF in inflammation and autoimmunity.

John A Hamilton.
Trends in Immunology (2002)

525 Citations

A null mutation in the gene encoding a type I interferon receptor component eliminates antiproliferative and antiviral responses to interferons alpha and beta and alters macrophage responses

Seung Y. Hwang;Paul J. Hertzog;Kerry A. Holland;Sony H. Sumarsono.
Proceedings of the National Academy of Sciences of the United States of America (1995)

515 Citations

Granulocyte Macrophage Colony-Stimulating Factor A New Putative Therapeutic Target in Multiple Sclerosis

Jonathan Luke McQualter;Rima Darwiche;Christine Ewing;Manabu Onuki.
Journal of Experimental Medicine (2001)

481 Citations

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