2006 - ACM Senior Member
His scientific interests lie mostly in Immunology, Inflammation, Macrophage, Granulocyte macrophage colony-stimulating factor and Internal medicine. Arthritis, Tumor necrosis factor alpha, Cytokine, Rheumatoid arthritis and Immune system are the subjects of his Immunology studies. His Inflammation study combines topics from a wide range of disciplines, such as Cancer, Immunotherapy, Cancer research and Encephalomyelitis, Multiple sclerosis.
His Macrophage study combines topics in areas such as Colony-stimulating factor, Granulocyte, Proinflammatory cytokine, Plasminogen activator and In vivo. In his study, which falls under the umbrella issue of Colony-stimulating factor, Cell biology, Cellular differentiation, Computational biology and Macrophage polarization is strongly linked to Macrophage colony-stimulating factor. His studies in Internal medicine integrate themes in fields like Endocrinology and Cycloheximide.
John A. Hamilton mostly deals with Immunology, Internal medicine, Molecular biology, Cell biology and Endocrinology. His Immunology research is multidisciplinary, relying on both Colony-stimulating factor and Macrophage. His Molecular biology research includes themes of Phosphorylation and DNA synthesis.
His work carried out in the field of Cell biology brings together such families of science as Macrophage colony-stimulating factor, Biochemistry and Growth factor. The Endocrinology study combines topics in areas such as Synovial Cell, Cytokine and Synovial membrane. His Cytokine research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha and Monocyte.
John A. Hamilton mainly focuses on Immunology, Inflammation, Arthritis, Cytokine and Tumor necrosis factor alpha. John A. Hamilton has included themes like Colony-stimulating factor, Macrophage, Granulocyte macrophage colony-stimulating factor and Monocyte in his Inflammation study. His Colony-stimulating factor research is multidisciplinary, incorporating elements of Progenitor cell and Clinical trial.
His Macrophage study integrates concerns from other disciplines, such as IRF4 and Cancer research. The various areas that John A. Hamilton examines in his Cancer research study include Macrophage colony-stimulating factor, Signal transduction and Immune system. His Tumor necrosis factor alpha research incorporates elements of Receptor and Nociceptor.
Inflammation, Immunology, Macrophage, Arthritis and Cytokine are his primary areas of study. His Inflammation study incorporates themes from Colony-stimulating factor, Porphyromonas gingivalis, Granulocyte macrophage colony-stimulating factor and Bioinformatics. His Immunology research incorporates themes from In vitro, Disease and Granulocyte colony-stimulating factor.
The Macrophage study combines topics in areas such as IRF4 and Cancer research. The various areas that John A. Hamilton examines in his Arthritis study include Tumor necrosis factor alpha, Cellular differentiation and Immune system. His Cytokine research includes elements of Chemokine, CXCL1, Monocyte and Cell biology.
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Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines
Peter J Murray;Judith E Allen;Subhra K Biswas;Edward A Fisher.
Colony-stimulating factors in inflammation and autoimmunity.
John A. Hamilton.
Nature Reviews Immunology (2008)
The TIGR Rice Genome Annotation Resource: Improvements and New Features
Shu Ouyang;Wei Zhu;John A. Hamilton;Haining Lin.
Nucleic Acids Research (2007)
Potential antiinflammatory effects of interleukin 4: suppression of human monocyte tumor necrosis factor alpha, interleukin 1, and prostaglandin E2
Prue H. Hart;Gerard F. Vitti;Diana R. Burgess;Genevieve A. Whitty.
Proceedings of the National Academy of Sciences of the United States of America (1989)
Granulocyte-Macrophage Colony-Stimulating Factor (CSF) and Macrophage CSF-Dependent Macrophage Phenotypes Display Differences in Cytokine Profiles and Transcription Factor Activities: Implications for CSF Blockade in Inflammation
Andrew J. Fleetwood;Toby Lawrence;John A. Hamilton;Andrew D. Cook.
Journal of Immunology (2007)
Macrophage plasminogen activator: Modulation of enzyme production by anti-inflammatory steroids, mitotic inhibitors, and cyclic nucleotides
Jean-Dominique Vassalli;John Hamilton;E. Reich.
GM-CSF in inflammation and autoimmunity.
John A Hamilton.
Trends in Immunology (2002)
A null mutation in the gene encoding a type I interferon receptor component eliminates antiproliferative and antiviral responses to interferons alpha and beta and alters macrophage responses
Seung Y. Hwang;Paul J. Hertzog;Kerry A. Holland;Sony H. Sumarsono.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Defining GM-CSF– and Macrophage-CSF–Dependent Macrophage Responses by In Vitro Models
Derek C Lacey;Adrian Achuthan;Andrew J Fleetwood;Hang Dinh.
Journal of Immunology (2012)
Granulocyte Macrophage Colony-Stimulating Factor A New Putative Therapeutic Target in Multiple Sclerosis
Jonathan Luke McQualter;Rima Darwiche;Christine Ewing;Manabu Onuki.
Journal of Experimental Medicine (2001)
Profile was last updated on December 6th, 2021.
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