What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Biochemistry, Bactericidal/permeability-increasing protein, Escherichia coli, Microbiology and Bacteria.
His study brings together the fields of Molecular biology and Biochemistry.
His Bactericidal/permeability-increasing protein research is multidisciplinary, incorporating perspectives in Lipopolysaccharide, Cytotoxic T cell, Membrane and Active protein.
The study incorporates disciplines such as Neutrophil elastase and Gram-negative bacteria in addition to Microbiology.
His Bacteria study integrates concerns from other disciplines, such as Extracellular, Amino acid sequence analysis, Human neutrophil and Host defence.
His work deals with themes such as Proinflammatory cytokine, Cell culture and TLR4, which intersect with Cell activation.
His most cited work include:
- Purification and characterization of a potent bactericidal and membrane active protein from the granules of human polymorphonuclear leukocytes. (402 citations)
- Isolation of an endotoxin–MD-2 complex that produces Toll-like receptor 4-dependent cell activation at picomolar concentrations (316 citations)
- The TLR4 antagonist Eritoran protects mice from lethal influenza infection (289 citations)
What are the main themes of his work throughout his whole career to date?
Jerrold Weiss mainly investigates Biochemistry, Microbiology, Escherichia coli, Bacteria and Bactericidal/permeability-increasing protein.
His research in Phospholipase A2, Cell activation, Phospholipase, Enzyme and Receptor are components of Biochemistry.
His Microbiology study combines topics from a wide range of disciplines, such as Extracellular, Lipopolysaccharide, Gram-negative bacteria and Staphylococcus aureus.
Jerrold Weiss usually deals with Escherichia coli and limits it to topics linked to Membrane protein and Membrane glycoproteins.
His Bacteria research incorporates elements of Respiratory burst, Cell envelope and Virulence.
His study explores the link between Bactericidal/permeability-increasing protein and topics such as Cytotoxic T cell that cross with problems in Degranulation.
He most often published in these fields:
- Biochemistry (57.78%)
- Microbiology (35.56%)
- Escherichia coli (25.00%)
What were the highlights of his more recent work (between 2010-2020)?
- TLR4 (24.44%)
- Biochemistry (57.78%)
- Cell biology (13.89%)
In recent papers he was focusing on the following fields of study:
Jerrold Weiss spends much of his time researching TLR4, Biochemistry, Cell biology, Lipid A and Lipopolysaccharide.
His biological study spans a wide range of topics, including Extracellular, Molecular biology and Tumor necrosis factor alpha.
His Biochemistry research includes themes of Biophysics and Bacteria.
The Bacteria study combines topics in areas such as Phospholipase A2 and Potency.
Within one scientific family, Jerrold Weiss focuses on topics pertaining to Microbiology under Potency, and may sometimes address concerns connected to Escherichia coli.
His studies in Lipopolysaccharide integrate themes in fields like Bacterial outer membrane and Vesicle.
Between 2010 and 2020, his most popular works were:
- The TLR4 antagonist Eritoran protects mice from lethal influenza infection (289 citations)
- High-mobility group nucleosome-binding protein 1 acts as an alarmin and is critical for lipopolysaccharide-induced immune responses (90 citations)
- Respiratory Syncytial Virus Fusion Protein-Induced Toll-Like Receptor 4 (TLR4) Signaling Is Inhibited by the TLR4 Antagonists Rhodobacter sphaeroides Lipopolysaccharide and Eritoran (E5564) and Requires Direct Interaction with MD-2 (77 citations)
In his most recent research, the most cited papers focused on:
His main research concerns TLR4, Lymphocyte antigen 96, Lipid A, Immunology and Cell biology.
He has included themes like Molecular biology and Eritoran in his TLR4 study.
His work in Molecular biology addresses subjects such as Tumor necrosis factor alpha, which are connected to disciplines such as Virology, CD14, Fusion protein, Toll-like receptor and Proinflammatory cytokine.
His Lymphocyte antigen 96 research is multidisciplinary, incorporating elements of Wild type, Stereochemistry and Ectodomain.
His Lipid A study combines topics in areas such as Acetylation and Mutant.
His work on Bone marrow, Antigen, T cell and Lipopolysaccharide as part of his general Immunology study is frequently connected to Nucleosome binding, thereby bridging the divide between different branches of science.
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