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Biology and Biochemistry

D-Index
64
Citations
11664
World Ranking
9882
National Ranking
4329

Overview

Robert S. Munford is affiliated with The University of Texas Southwestern Medical Center in the United States. Their research primarily spans areas within Medicine, Biochemistry, Genetics and Molecular Biology, and Immunology and Microbiology.

The scientist's work focuses on several main topics including:

  • Liver Disease Diagnosis and Treatment
  • Diet, Metabolism, and Disease
  • Inflammasome and immune disorders
  • Immune cells in cancer
  • Immune Response and Inflammation
  • Pediatric health and respiratory diseases
  • Phagocytosis and Immune Regulation

Subfields of study related to their publications cover Immunology, Molecular Biology, Epidemiology, Endocrinology, Diabetes and Metabolism, and Surgery.

Recent scientific papers authored or co-authored by Robert S. Munford include:

  • Biochemical transformation of bacterial lipopolysaccharides by acyloxyacyl hydrolase reduces host injury and promotes recovery, 2020, Journal of Biological Chemistry
  • Extracellular Acidity Reprograms Macrophage Metabolism and Innate Responsiveness, 2021, The Journal of Immunology
  • A host lipase prevents lipopolysaccharide-induced foam cell formation, 2021, iScience
  • A host enzyme reduces metabolic dysfunction-associated steatotic liver disease (MASLD) by inactivating intestinal lipopolysaccharide, 2024, eLife
  • A host enzyme reduces metabolic dysfunction-associated steatotic liver disease (MASLD) by inactivating intestinal lipopolysaccharide, 2025, eLife

Frequent co-authors collaborating with Munford include Mingfang Lu, Yiwei Chu, Benkun Zou, Wei Jiang, and Jintao Feng.

Publications often appear in venues such as The Journal of Immunology, eLife, bioRxiv (Cold Spring Harbor Laboratory), Journal of Biological Chemistry, and iScience.

Best Publications

  • Future research directions in acute lung injury: Summary of a National Heart, Lung, and Blood Institute Working Group

    Michael A. Matthay;Guy A. Zimmerman;Charles Esmon;Jahar Bhattacharya

  • Normal responses to injury prevent systemic inflammation and can be immunosuppressive.

    Robert S. Munford;Jérôme Pugin

  • Lipopolysaccharide (LPS) partial structures inhibit responses to LPS in a human macrophage cell line without inhibiting LPS uptake by a CD14-mediated pathway.

    Richard L. Kitchens;Richard J. Ulevitch;Robert S. Munford

  • Risks and benefits of activated protein C treatment for severe sepsis.

    H. Shaw Warren;Anthony F. Suffredini;Peter Q. Eichacker;Robert S. Munford

  • Anti-endotoxin monoclonal antibodies.

    H. Shaw Warren;Robert L. Danner;Robert S. Munford

  • Saposin-like proteins (SAPLIP) carry out diverse functions on a common backbone structure.

    R S Munford;P O Sheppard;P J O'Hara

  • Sensing gram-negative bacterial lipopolysaccharides: a human disease determinant?

    Robert S. Munford

  • Plasma CD14 decreases monocyte responses to LPS by transferring cell-bound LPS to plasma lipoproteins

    Richard L. Kitchens;Patricia A. Thompson;Suganya Viriyakosol;Grant E. O’Keefe

  • Lipopolysaccharide-binding protein and phospholipid transfer protein release lipopolysaccharides from gram-negative bacterial membranes.

    C. J. Vesy;R. L. Kitchens;G. Wolfbauer;J. J. Albers

  • Enzymatically deacylated lipopolysaccharide (LPS) can antagonize LPS at multiple sites in the LPS recognition pathway.

    Richard L. Kitchens;Robert S. Munford

  • Statins and the Acute-Phase Response

    Robert S. Munford

  • Detoxifying endotoxin: time, place and person.

    Robert S Munford

  • Shield as Signal: Lipopolysaccharides and the Evolution of Immunity to Gram-Negative Bacteria

    Robert S. Munford;Alan W. Varley

  • Severe sepsis and septic shock: the role of gram-negative bacteremia.

    Robert S. Munford

  • Biological activity, lipoprotein-binding behavior, and in vivo disposition of extracted and native forms of Salmonella typhimurium lipopolysaccharides.

    Robert S. Munford;Catherine L. Hall;J. M. Lipton;John M. Dietschy

  • CD14-Dependent Internalization of Bacterial Lipopolysaccharide (LPS) Is Strongly Influenced by LPS Aggregation But Not by Cellular Responses to LPS

    Richard L. Kitchens;Robert S. Munford

  • Single-Dose Amoxicillin Therapy for Urinary Tract Infection: Multicenter Trial Using Antibody-Coated Bacteria Localization Technique

    Robert H. Rubin;Leslie S. T. Fang;Stephen R. Jones;Robert S. Munford

  • The G-->A single nucleotide polymorphism at the -308 position in the tumor necrosis factor-alpha promoter increases the risk for severe sepsis after trauma.

    Grant E. O’Keefe;Dixie L. Hybki;Robert S. Munford

  • Plasma lipoproteins promote the release of bacterial lipopolysaccharide from the monocyte cell surface.

    Richard L. Kitchens;Gertrud Wolfbauer;John J. Albers;Robert S. Munford

  • Invited review: Detoxifying endotoxin: time, place and person:

    Robert S. Munford

Frequent Co-Authors

Jerrold Weiss
Jerrold Weiss University of Iowa
John M. Dietschy
John M. Dietschy The University of Texas Southwestern Medical Center
George H. McCracken
George H. McCracken The University of Texas Southwestern Medical Center
Eric J. Hansen
Eric J. Hansen The University of Texas System
Kenneth R. Chien
Kenneth R. Chien Karolinska Institute
Richard J. Ulevitch
Richard J. Ulevitch Scripps Research Institute
Ellen S. Vitetta
Ellen S. Vitetta The University of Texas Southwestern Medical Center
Octavio Ramilo
Octavio Ramilo St. Jude Children's Research Hospital
Kevin J. Tracey
Kevin J. Tracey Feinstein Institute for Medical Research
Robert D. Gerard
Robert D. Gerard The University of Texas Southwestern Medical Center

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