D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 84 Citations 24,245 329 World Ranking 10075 National Ranking 394

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Gene
  • Enzyme

His primary scientific interests are in Internal medicine, Endocrinology, Beta oxidation, AMPK and Protein kinase A. His Internal medicine research integrates issues from Diabetes mellitus and Mitochondrion. His Endocrinology study typically links adjacent topics like Pulmonary hypertension.

The concepts of his Beta oxidation study are interwoven with issues in Cardiac function curve and Ischemia. Jason R.B. Dyck works in the field of AMPK, namely AMP-activated protein kinase. His biological study spans a wide range of topics, including Lipogenesis and Metabolic syndrome.

His most cited work include:

  • Mitochondrial Overload and Incomplete Fatty Acid Oxidation Contribute to Skeletal Muscle Insulin Resistance (1369 citations)
  • Phosphodiesterase Type 5 Is Highly Expressed in the Hypertrophied Human Right Ventricle, and Acute Inhibition of Phosphodiesterase Type 5 Improves Contractility (443 citations)
  • Single phosphorylation sites in Acc1 and Acc2 regulate lipid homeostasis and the insulin-sensitizing effects of metformin (439 citations)

What are the main themes of his work throughout his whole career to date?

Jason R.B. Dyck mostly deals with Internal medicine, Endocrinology, Heart failure, Cardiology and Biochemistry. His Internal medicine study combines topics in areas such as AMPK and Resveratrol. His research integrates issues of AMP-activated protein kinase and Protein kinase A in his study of Endocrinology.

His studies deal with areas such as Diastole and Cohort as well as Heart failure. All of his Biochemistry and Malonyl-CoA decarboxylase and Enzyme investigations are sub-components of the entire Biochemistry study. His work carried out in the field of Beta oxidation brings together such families of science as Carbohydrate metabolism, Mitochondrion and Ischemia.

He most often published in these fields:

  • Internal medicine (66.26%)
  • Endocrinology (48.94%)
  • Heart failure (21.28%)

What were the highlights of his more recent work (between 2015-2021)?

  • Internal medicine (66.26%)
  • Heart failure (21.28%)
  • Endocrinology (48.94%)

In recent papers he was focusing on the following fields of study:

Internal medicine, Heart failure, Endocrinology, Cardiology and Ejection fraction are his primary areas of study. His Beta oxidation, Cohort, Glycolysis, Left ventricular hypertrophy and Inflammation study are his primary interests in Internal medicine. As part of one scientific family, he deals mainly with the area of Heart failure, narrowing it down to issues related to the Muscle hypertrophy, and often Fatty acid metabolism.

His work on Endocrinology is being expanded to include thematically relevant topics such as Resveratrol. His Cardiology study combines topics from a wide range of disciplines, such as Prospective cohort study and Pulmonary edema. His work deals with themes such as Natriuretic peptide, Heart disease, Diastole and Ventricular remodeling, which intersect with Ejection fraction.

Between 2015 and 2021, his most popular works were:

  • The Emerging Role of Metabolomics in the Diagnosis and Prognosis of Cardiovascular Disease (115 citations)
  • Improved Glucose Homeostasis in Obese Mice Treated With Resveratrol Is Associated With Alterations in the Gut Microbiome. (90 citations)
  • Empagliflozin Prevents Worsening of Cardiac Function in an Experimental Model of Pressure Overload-Induced Heart Failure (72 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Gene
  • Enzyme

Jason R.B. Dyck mainly investigates Heart failure, Internal medicine, Endocrinology, Cardiology and Resveratrol. His Heart failure research incorporates elements of Muscle hypertrophy and Empagliflozin. His Internal medicine research is multidisciplinary, relying on both Physical therapy and Oncology.

Borrowing concepts from Offspring, Jason R.B. Dyck weaves in ideas under Endocrinology. His work on Ischemia as part of general Cardiology research is frequently linked to Preeclampsia, Endoglin and Gestational hypertension, bridging the gap between disciplines. His Resveratrol research is multidisciplinary, incorporating elements of Oral administration, Intensive care medicine, Cause of death and Glucose homeostasis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mitochondrial Overload and Incomplete Fatty Acid Oxidation Contribute to Skeletal Muscle Insulin Resistance

Timothy R. Koves;John R. Ussher;Robert C. Noland;Dorothy Slentz.
Cell Metabolism (2008)

1886 Citations

Single phosphorylation sites in Acc1 and Acc2 regulate lipid homeostasis and the insulin-sensitizing effects of metformin

Morgan D. Fullerton;Sandra Galic;Katarina Marcinko;Sarah Sikkema.
Nature Medicine (2013)

687 Citations

Phosphodiesterase Type 5 Is Highly Expressed in the Hypertrophied Human Right Ventricle, and Acute Inhibition of Phosphodiesterase Type 5 Improves Contractility

Jayan Nagendran;Stephen L. Archer;Daniel Soliman;Vikram Gurtu.
Circulation (2007)

584 Citations

Intracellular Action of Matrix Metalloproteinase-2 Accounts for Acute Myocardial Ischemia and Reperfusion Injury

Wenjie Wang;Costas J. Schulze;Wilma L. Suarez-Pinzon;Jason R.B. Dyck.
Circulation (2002)

516 Citations

Dichloroacetate prevents and reverses pulmonary hypertension by inducing pulmonary artery smooth muscle cell apoptosis

M. Sean McMurtry;Sebastien Bonnet;Xichen Wu;Jason R.B. Dyck.
Circulation Research (2004)

510 Citations

Increased Hepatic CD36 Expression Contributes to Dyslipidemia Associated With Diet-Induced Obesity

Debby P Y Koonen;René L. Jacobs;Maria Febbraio;Martin E. Young.
Diabetes (2007)

481 Citations

Liver-Specific Inhibition of ChREBP Improves Hepatic Steatosis and Insulin Resistance in ob/ob Mice

Renaud Dentin;Fadila Benhamed;Isabelle Hainault;Véronique Fauveau.
Diabetes (2006)

464 Citations

Dichloroacetate, a Metabolic Modulator, Prevents and Reverses Chronic Hypoxic Pulmonary Hypertension in Rats Role of Increased Expression and Activity of Voltage-Gated Potassium Channels

Evangelos D. Michelakis;M. Sean McMurtry;Xi Chen Wu;Jason R.B. Dyck.
Circulation (2002)

463 Citations

Akt activity negatively regulates phosphorylation of AMP-activated protein kinase in the heart.

Suzanne Kovacic;Carrie-Lynn M. Soltys;Amy J. Barr;Ichiro Shiojima.
Journal of Biological Chemistry (2003)

456 Citations

AMPK alterations in cardiac physiology and pathology: enemy or ally?

Jason R. B. Dyck;Gary D. Lopaschuk.
The Journal of Physiology (2006)

448 Citations

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