His primary scientific interests are in Internal medicine, Endocrinology, Beta oxidation, AMPK and Protein kinase A. His Internal medicine research integrates issues from Diabetes mellitus and Mitochondrion. His Endocrinology study typically links adjacent topics like Pulmonary hypertension.
The concepts of his Beta oxidation study are interwoven with issues in Cardiac function curve and Ischemia. Jason R.B. Dyck works in the field of AMPK, namely AMP-activated protein kinase. His biological study spans a wide range of topics, including Lipogenesis and Metabolic syndrome.
Jason R.B. Dyck mostly deals with Internal medicine, Endocrinology, Heart failure, Cardiology and Biochemistry. His Internal medicine study combines topics in areas such as AMPK and Resveratrol. His research integrates issues of AMP-activated protein kinase and Protein kinase A in his study of Endocrinology.
His studies deal with areas such as Diastole and Cohort as well as Heart failure. All of his Biochemistry and Malonyl-CoA decarboxylase and Enzyme investigations are sub-components of the entire Biochemistry study. His work carried out in the field of Beta oxidation brings together such families of science as Carbohydrate metabolism, Mitochondrion and Ischemia.
Internal medicine, Heart failure, Endocrinology, Cardiology and Ejection fraction are his primary areas of study. His Beta oxidation, Cohort, Glycolysis, Left ventricular hypertrophy and Inflammation study are his primary interests in Internal medicine. As part of one scientific family, he deals mainly with the area of Heart failure, narrowing it down to issues related to the Muscle hypertrophy, and often Fatty acid metabolism.
His work on Endocrinology is being expanded to include thematically relevant topics such as Resveratrol. His Cardiology study combines topics from a wide range of disciplines, such as Prospective cohort study and Pulmonary edema. His work deals with themes such as Natriuretic peptide, Heart disease, Diastole and Ventricular remodeling, which intersect with Ejection fraction.
Jason R.B. Dyck mainly investigates Heart failure, Internal medicine, Endocrinology, Cardiology and Resveratrol. His Heart failure research incorporates elements of Muscle hypertrophy and Empagliflozin. His Internal medicine research is multidisciplinary, relying on both Physical therapy and Oncology.
Borrowing concepts from Offspring, Jason R.B. Dyck weaves in ideas under Endocrinology. His work on Ischemia as part of general Cardiology research is frequently linked to Preeclampsia, Endoglin and Gestational hypertension, bridging the gap between disciplines. His Resveratrol research is multidisciplinary, incorporating elements of Oral administration, Intensive care medicine, Cause of death and Glucose homeostasis.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mitochondrial Overload and Incomplete Fatty Acid Oxidation Contribute to Skeletal Muscle Insulin Resistance
Timothy R. Koves;John R. Ussher;Robert C. Noland;Dorothy Slentz.
Cell Metabolism (2008)
Single phosphorylation sites in Acc1 and Acc2 regulate lipid homeostasis and the insulin-sensitizing effects of metformin
Morgan D. Fullerton;Sandra Galic;Katarina Marcinko;Sarah Sikkema.
Nature Medicine (2013)
Phosphodiesterase Type 5 Is Highly Expressed in the Hypertrophied Human Right Ventricle, and Acute Inhibition of Phosphodiesterase Type 5 Improves Contractility
Jayan Nagendran;Stephen L. Archer;Daniel Soliman;Vikram Gurtu.
Circulation (2007)
Intracellular Action of Matrix Metalloproteinase-2 Accounts for Acute Myocardial Ischemia and Reperfusion Injury
Wenjie Wang;Costas J. Schulze;Wilma L. Suarez-Pinzon;Jason R.B. Dyck.
Circulation (2002)
Dichloroacetate prevents and reverses pulmonary hypertension by inducing pulmonary artery smooth muscle cell apoptosis
M. Sean McMurtry;Sebastien Bonnet;Xichen Wu;Jason R.B. Dyck.
Circulation Research (2004)
Increased Hepatic CD36 Expression Contributes to Dyslipidemia Associated With Diet-Induced Obesity
Debby P Y Koonen;René L. Jacobs;Maria Febbraio;Martin E. Young.
Diabetes (2007)
Liver-Specific Inhibition of ChREBP Improves Hepatic Steatosis and Insulin Resistance in ob/ob Mice
Renaud Dentin;Fadila Benhamed;Isabelle Hainault;Véronique Fauveau.
Diabetes (2006)
Dichloroacetate, a Metabolic Modulator, Prevents and Reverses Chronic Hypoxic Pulmonary Hypertension in Rats Role of Increased Expression and Activity of Voltage-Gated Potassium Channels
Evangelos D. Michelakis;M. Sean McMurtry;Xi Chen Wu;Jason R.B. Dyck.
Circulation (2002)
Akt activity negatively regulates phosphorylation of AMP-activated protein kinase in the heart.
Suzanne Kovacic;Carrie-Lynn M. Soltys;Amy J. Barr;Ichiro Shiojima.
Journal of Biological Chemistry (2003)
AMPK alterations in cardiac physiology and pathology: enemy or ally?
Jason R. B. Dyck;Gary D. Lopaschuk.
The Journal of Physiology (2006)
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