2016 - Distinguished Scientist Award, American Heart Association
2011 - Distinguished Fellowship Award, American College of Cardiology (ACC)
His primary areas of study are Pulmonary hypertension, Internal medicine, Endocrinology, Hypoxia and Vasoconstriction. Stephen L. Archer interconnects Respiratory disease, Sildenafil, Hemodynamics, Vascular resistance and Pulmonary artery in the investigation of issues within Pulmonary hypertension. His research on Internal medicine often connects related topics like Cardiology.
His Cardiology research includes themes of Expert consensus and Surgery. His Hypoxia research includes elements of Circulatory system and Antimycin A, Kinase, Mitochondrion, Cell biology. His Vasoconstriction study combines topics from a wide range of disciplines, such as Pharmacology, Potassium channel and Vasodilation.
His primary scientific interests are in Internal medicine, Cardiology, Pulmonary hypertension, Endocrinology and Pulmonary artery. His Vasoconstriction, Hypoxia, Right ventricular hypertrophy, Nitric oxide and Hypoxic pulmonary vasoconstriction investigations are all subjects of Internal medicine research. His work deals with themes such as Anesthesia and Mitochondrion, which intersect with Hypoxia.
The study incorporates disciplines such as Potassium channel and Cell biology in addition to Hypoxic pulmonary vasoconstriction. The Ventricle, Heart failure and Right ventricular failure research Stephen L. Archer does as part of his general Cardiology study is frequently linked to other disciplines of science, such as In patient, therefore creating a link between diverse domains of science. His Pulmonary hypertension research is multidisciplinary, relying on both Respiratory disease, Lung, Pathology and Hemodynamics, Vascular resistance.
His scientific interests lie mostly in Internal medicine, Cardiology, Pulmonary hypertension, Mitochondrion and Mitochondrial fission. His Internal medicine research integrates issues from Endocrinology and Metabolic syndrome. Within one scientific family, Stephen L. Archer focuses on topics pertaining to Diastole under Cardiology, and may sometimes address concerns connected to Cardiac catheterization.
His Pulmonary hypertension study integrates concerns from other disciplines, such as Cancer research, Inflammation, Ventricular function, Pulmonary artery and Fibrosis. The Mitochondrion study combines topics in areas such as Cell, Reactive oxygen species, Hypoxic pulmonary vasoconstriction and Pyruvate dehydrogenase complex. His Mitochondrial fission research is multidisciplinary, incorporating perspectives in mitochondrial fusion, FIS1, Cell cycle and Cell growth.
Stephen L. Archer mainly focuses on Pulmonary hypertension, Internal medicine, Cardiology, Mitochondrion and Cell biology. The various areas that Stephen L. Archer examines in his Pulmonary hypertension study include Cancer research, Inflammation, Pulmonary artery, Fibrosis and Vasodilation. The concepts of his Cancer research study are interwoven with issues in Hypoxia, Lung and Pathogenesis, Pathology.
Stephen L. Archer has researched Cardiology in several fields, including Microtubule and Colchicine. Stephen L. Archer combines subjects such as Vasoconstriction, Hypoxic pulmonary vasoconstriction and Pyruvate dehydrogenase complex with his study of Mitochondrion. His work in the fields of Cell biology, such as Mitochondrial fission, GTPase and Reactive oxygen species generation, intersects with other areas such as Mitophagy and Dynamin.
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ACCF/AHA 2009 Expert Consensus Document on Pulmonary Hypertension: A Report of the American College of Cardiology Foundation Task Force on Expert Consensus Documents and the American Heart Association: Developed in Collaboration With the American College of Chest Physicians, American Thoracic Society, Inc., and the Pulmonary Hypertension Association
Vallerie V. McLaughlin;Stephen L. Archer;David B. Badesch;Robyn J. Barst.
Journal of the American College of Cardiology (2009)
Management of Massive and Submassive Pulmonary Embolism, Iliofemoral Deep Vein Thrombosis, and Chronic Thromboembolic Pulmonary Hypertension A Scientific Statement From the American Heart Association
Michael R. Jaff;M. Sean McMurtry;Stephen L. Archer;Mary Cushman.
Circulation (2011)
Cellular and molecular pathobiology of pulmonary arterial hypertension.
Marc Humbert;Nicholas W Morrell;Stephen L Archer;Kurt R Stenmark.
Journal of the American College of Cardiology (2004)
A Mitochondria-K+ Channel Axis Is Suppressed in Cancer and Its Normalization Promotes Apoptosis and Inhibits Cancer Growth
Sébastien Bonnet;Stephen L. Archer;Joan Allalunis-Turner;Alois Haromy.
Cancer Cell (2007)
ACCF/AHA 2009 Expert Consensus Document on Pulmonary Hypertension
Vallerie V. McLaughlin;Stephen L. Archer;David B. Badesch;Robyn J. Barst.
Circulation (2009)
Measurement of nitric oxide in biological models.
Stephen Archer.
The FASEB Journal (1993)
Nitric oxide and cGMP cause vasorelaxation by activation of a charybdotoxin-sensitive K channel by cGMP-dependent protein kinase
Stephen L. Archer;James M.C. Huang;Václav Hampl;Daniel P. Nelson.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Cellular and molecular basis of pulmonary arterial hypertension.
Nicholas W. Morrell;Serge Adnot;Stephen L. Archer;Jocelyn Dupuis.
Journal of the American College of Cardiology (2009)
Oral Sildenafil Is an Effective and Specific Pulmonary Vasodilator in Patients With Pulmonary Arterial Hypertension Comparison With Inhaled Nitric Oxide
Evangelos Michelakis;Wayne Tymchak;Dale Lien;Linda Webster.
Circulation (2002)
An Abnormal Mitochondrial–Hypoxia Inducible Factor-1α–Kv Channel Pathway Disrupts Oxygen Sensing and Triggers Pulmonary Arterial Hypertension in Fawn Hooded Rats Similarities to Human Pulmonary Arterial Hypertension
Sébastien Bonnet;Evangelos D. Michelakis;Christopher J. Porter;Miguel A. Andrade-Navarro.
Circulation (2006)
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