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Hanspeter Pircher

Hanspeter Pircher

D-Index & Metrics

Immunology

D-Index
83
Citations
24354
World Ranking
1450
National Ranking
98

Overview

Hanspeter Pircher is affiliated with the University of Freiburg in Germany and works primarily in the fields of Immunology and Microbiology, Medicine, and Biochemistry, Genetics and Molecular Biology. Their research spans several subfields including Immunology, Epidemiology, and Genetics.

The scientist's work addresses key topics such as Immunodeficiency and Autoimmune Disorders, Virology and Viral Diseases, Cytomegalovirus and herpesvirus research, Immune Cell Function and Interaction, IL-33, ST2, and ILC Pathways, Virus-based gene therapy research, and Diabetes and associated disorders.

Hanspeter Pircher has published research in several notable venues, with frequent contributions to:

  • The Journal of Experimental Medicine
  • European Journal of Immunology

Among their recent papers are:

  • NK1.1 + innate lymphoid cells in salivary glands inhibit establishment of tissue-resident memory CD8 + T cells in mice (2020) published in European Journal of Immunology
  • Patients and mice with deficiency in the SNARE protein SYNTAXIN-11 have a secondary B cell defect (2024) published in The Journal of Experimental Medicine
  • Correction: Patients and mice with deficiency in the SNARE protein SYNTAXIN-11 have a secondary B cell defect (2024) published in The Journal of Experimental Medicine

The scientist frequently collaborates with others, including Kristoffer Weißert, Peter Aichele, Sandra Ammann, Tamara Kögl, and Hsin-Fang Chang, with multiple publications co-authored by each.

Best Publications

  • Virus persistence in acutely infected immunocompetent mice by exhaustion of antiviral cytotoxic effector T cells

    Demetrius Moskophidis;Franziska Lechner;Hanspeter Pircher;Rolf M. Zinkernagel

  • Ablation of "tolerance" and induction of diabetes by virus infection in viral antigen transgenic mice.

    Pamela S. Ohashi;Stephan Oehen;Kurt Buerki;Hanspeter Pircher

  • Tolerance induction in double specific T-cell receptor transgenic mice varies with antigen

    Hanspeter Pircher;Kurt Bürki;Rosemarie Lang;Hans Hengartner

  • Evidence for a differential avidity model of T cell selection in the thymus

    Philip G. Ashton-Rickardt;Antonio Bandeira;Joseph R. Delaney;Luc Van Kaer

  • CD57 defines a functionally distinct population of mature NK cells in the human CD56dimCD16+ NK-cell subset

    Sandra Lopez-Vergès;Jeffrey M. Milush;Suchitra Pandey;Vanessa A. York

  • Viral escape by selection of cytotoxic T cell-resistant virus variants in vivo

    Hanspeter Pircher;Demetrius Moskophidis;Urs Rohrer;Kurt Bürki

  • Partial Impairment of Cytokine Responses in Tyk2-Deficient Mice

    Marina Karaghiosoff;Hans Neubauer;Caroline Lassnig;Pavel Kovarik

  • Aerobic fitness is associated with lower proportions of senescent blood T-cells in man

    Guillaume Spielmann;Brian K. McFarlin;Daniel P. O’Connor;Paula J.W. Smith

  • Coexpression of PD-1, 2B4, CD160 and KLRG1 on Exhausted HCV-Specific CD8+ T Cells Is Linked to Antigen Recognition and T Cell Differentiation

    Bertram Bengsch;Bianca Seigel;Marianne Ruhl;Jörg Timm

  • Lack of proliferative capacity of human effector and memory T cells expressing killer cell lectinlike receptor G1 (KLRG1).

    David Voehringer;Marie Koschella;Hanspeter Pircher

  • A Critical Requirement of Interferon γ-mediated Angiostasis for Tumor Rejection by CD8+ T Cells

    Zhihai Qin;Johannes Schwartzkopff;Felicia Pradera;Thomas Kammertoens

  • Lower receptor avidity required for thymic clonal deletion than for effector T-cell function.

    Hanspeter Pircher;Urs Hoffmann Rohrer;Demetrius Moskophidis;Rolf M. Zinkernagel

  • β‐Galactoside‐binding protein secreted by activated T cells inhibits antigen‐induced proliferation of T cells

    Claudine Blaser;Martina Kaufmann;Christoph Müller;Christine Zimmermann

  • Age-associated accumulation of CMV-specific CD8+ T cells expressing the inhibitory killer cell lectin-like receptor G1 (KLRG1)

    Qin Ouyang;Wolfgang M Wagner;David Voehringer;Anders Wikby

  • Virus-specific CD8+ cells can switch to interleukin 5 production and induce airway eosinophilia.

    A J Coyle;F Erard;C Bertrand;S Walti

  • Visualization, characterization, and turnover of CD8+ memory T cells in virus-infected hosts.

    C Zimmerman;K Brduscha-Riem;C Blaser;R M Zinkernagel

  • Inhibitory Receptor Expression Depends More Dominantly on Differentiation and Activation than "Exhaustion" of Human CD8 T Cells.

    Amandine Legat;Daniel E. Speiser;Hanspeter Pircher;Dietmar Zehn

  • Viral infections induce abundant numbers of senescent CD8 T cells.

    David Voehringer;Claudine Blaser;Pierre Brawand;David H. Raulet

  • T cell immunity after a viral infection versus T cell tolerance induced by soluble viral peptides.

    Kyburz D;Aichele P;Speiser De;Hengartner H

  • KLRG1 signaling induces defective Akt (ser473) phosphorylation and proliferative dysfunction of highly differentiated CD8+ T cells

    Sian M. Henson;Ornella Franzese;Ornella Franzese;Richard Macaulay;Valentina Libri

  • T cell priming versus T cell tolerance induced by synthetic peptides

    P Aichele;K Brduscha-Riem;R M Zinkernagel;H Hengartner

Frequent Co-Authors

Hans Hengartner
Hans Hengartner University of Zurich
Rolf M. Zinkernagel
Rolf M. Zinkernagel University of Zurich
Pamela S. Ohashi
Pamela S. Ohashi Princess Margaret Cancer Centre
Kurt Bürki
Kurt Bürki University of Zurich
Richard J. Simpson
Richard J. Simpson La Trobe University
Bernhard Odermatt
Bernhard Odermatt University of Zurich
Stephan Ehl
Stephan Ehl University of Freiburg
Tak W. Mak
Tak W. Mak Princess Margaret Cancer Centre
Daniel E. Speiser
Daniel E. Speiser University of Lausanne
Thomas M. Kündig
Thomas M. Kündig University of Zurich

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Related Online Degrees & Career Pathways

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Additionally, many aspiring nurses find success enrolling in the easiest ABSN to get into. These accelerated BSN pathways provide a streamlined route to nursing licensure, enabling quicker transitions into immunology-related healthcare roles.

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