What is he best known for?
The fields of study he is best known for:
Gregory M. Acland mostly deals with Genetics, Retinal, Retina, Progressive retinal atrophy and Retinal degeneration.
His study in Genome, Candidate gene, Locus, Allele and Linkage is carried out as part of his Genetics studies.
As a part of the same scientific family, Gregory M. Acland mostly works in the field of Retinal, focusing on Cell biology and, on occasion, Point mutation, Central nervous system, Retinal ganglion and Green fluorescent protein.
The Retina study combines topics in areas such as Ophthalmology and Optic nerve.
His Progressive retinal atrophy study incorporates themes from Frameshift mutation and Exon.
His biological study spans a wide range of topics, including Electroretinography, Anatomy, Tissue Degeneration, Retinitis pigmentosa and Rhodopsin.
His most cited work include:
- Gene therapy restores vision in a canine model of childhood blindness. (1025 citations)
- Long-Term Restoration of Rod and Cone Vision by Single Dose rAAV-Mediated Gene Transfer to the Retina in a Canine Model of Childhood Blindness (392 citations)
- Encapsulated cell-based delivery of CNTF reduces photoreceptor degeneration in animal models of retinitis pigmentosa. (287 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Genetics, Retinal, Molecular biology, Retinal degeneration and Gene.
His study in Genetic linkage, Progressive retinal atrophy, Locus, Candidate gene and Allele is carried out as part of his studies in Genetics.
Progressive retinal atrophy is the subject of his research, which falls under Retinitis pigmentosa.
The various areas that he examines in his Retinal study include Retina and Pathology.
His Retina research focuses on Cell biology and how it connects with Mutant.
His Molecular biology study combines topics from a wide range of disciplines, such as Complementary DNA, Gene expression and Transducin.
He most often published in these fields:
- Genetics (47.15%)
- Retinal (21.54%)
- Molecular biology (19.92%)
What were the highlights of his more recent work (between 2008-2020)?
- Genetics (47.15%)
- Gene (16.67%)
- Retina (15.85%)
In recent papers he was focusing on the following fields of study:
His main research concerns Genetics, Gene, Retina, Cell biology and Retinal degeneration.
His Genetics and Genome-wide association study, Phenotype, Single-nucleotide polymorphism, Genome and Haplotype investigations all form part of his Genetics research activities.
His research links Ophthalmology with Gene.
His research in Retina intersects with topics in Gene expression, Mutant and Frameshift mutation.
Retinal covers Gregory M. Acland research in Retinal degeneration.
His research in the fields of Retinitis pigmentosa and Retinal Disorder overlaps with other disciplines such as Retinopathy.
Between 2008 and 2020, his most popular works were:
- An Expressed Fgf4 Retrogene Is Associated with Breed-Defining Chondrodysplasia in Domestic Dogs (248 citations)
- An Expressed Fgf4 Retrogene Is Associated with Breed-Defining Chondrodysplasia in Domestic Dogs (248 citations)
- Gene therapy rescues cone function in congenital achromatopsia (212 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Genetics, Gene, Genome, Domestication and Phenotype.
His study in Single-nucleotide polymorphism, Candidate gene, Haplotype, Genetic linkage and Genome-wide association study is done as part of Genetics.
Gregory M. Acland has researched Candidate gene in several fields, including Retinal degeneration, Penetrance and Chromosome 17.
As a member of one scientific family, Gregory M. Acland mostly works in the field of Retinal degeneration, focusing on Retinal Rod Photoreceptor Cells and, on occasion, Photoreceptor cell and Retina.
In his research, Retinal is intimately related to Frameshift mutation, which falls under the overarching field of Photoreceptor cell.
His studies deal with areas such as Channelopathy and Genetic enhancement as well as Retinal.
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