David J. Elliott spends much of his time researching RNA splicing, Cell biology, Genetics, Molecular biology and RNA-binding protein. His RNA splicing research is multidisciplinary, relying on both In situ hybridization, RNA polymerase II, Alternative splicing and RNA-Seq. David J. Elliott works mostly in the field of Cell biology, limiting it down to concerns involving RNA and, occasionally, Nucleolus, Green fluorescent protein, Complementary DNA and Transcription.
When carried out as part of a general Genetics research project, his work on Y chromosome, Skewed X-inactivation, X-inactivation and XIST is frequently linked to work in Azoospermia, therefore connecting diverse disciplines of study. His Molecular biology study combines topics from a wide range of disciplines, such as Terminator, Cytoplasm, Saccharomyces cerevisiae, Polyadenylation and Consensus sequence. His RNA-binding protein study is related to the wider topic of Gene.
David J. Elliott mostly deals with RNA splicing, Cell biology, Genetics, Alternative splicing and Gene. His research in RNA splicing intersects with topics in Molecular biology, RNA-binding protein, Intron and Exon. His work investigates the relationship between RNA-binding protein and topics such as Gene expression that intersect with problems in Germ cell.
His research investigates the connection between Cell biology and topics such as RNA that intersect with issues in Messenger RNA. His work on Gene family, Regulation of gene expression, Sperm and Locus as part of general Genetics research is frequently linked to Male infertility, bridging the gap between disciplines. The Alternative splicing study combines topics in areas such as PRPF31, Computational biology and Prostate cancer.
David J. Elliott mainly focuses on RNA splicing, Alternative splicing, Cell biology, Cancer research and Prostate cancer. His RNA splicing research integrates issues from Computational biology, RNA-binding protein and Exon. His work deals with themes such as Exonic splicing enhancer, Gene expression, Intron and Germ cell, which intersect with RNA-binding protein.
His work focuses on many connections between Alternative splicing and other disciplines, such as Regulation of gene expression, that overlap with his field of interest in Gene expression profiling. The various areas that David J. Elliott examines in his Cell biology study include PRPF31, Germline, Splicing factor, Induced pluripotent stem cell and Fungal protein. His research integrates issues of Molecular biology and LNCaP in his study of Androgen receptor.
His main research concerns Alternative splicing, RNA splicing, Cell biology, Exon and Prostate cancer. His study in Alternative splicing is interdisciplinary in nature, drawing from both Retinal pigment epithelium, PRPF31, Regulation of gene expression, RNA-binding protein and Intron. David J. Elliott has included themes like Exonic splicing enhancer, Molecular biology and Minigene in his RNA-binding protein study.
His RNA splicing research is within the category of Genetics. His Cell biology research incorporates themes from Transcriptome and Retinal, Retinitis pigmentosa. His Prostate cancer study incorporates themes from Androgen and Glycosylation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
In Vitro-Differentiated Embryonic Stem Cells Give Rise to Male Gametes that Can Generate Offspring Mice
Karim Nayernia;Jessica Nolte;Hans W. Michelmann;Jae Ho Lee.
Developmental Cell (2006)
Nucleotide sequence and gene organization of sea urchin mitochondrial DNA.
Howard T. Jacobs;David J. Elliott;Veerabhadracharya B. Math;Andrew Farquharson.
Journal of Molecular Biology (1990)
Sam68 sequestration and partial loss of function are associated with splicing alterations in FXTAS patients
Chantal Sellier;Frédérique Rau;Yilei Liu;Flora Tassone.
The EMBO Journal (2010)
Expression of RBM in the nuclei of human germ cells is dependent on a critical region of the Y chromosome long arm
D. J. Elliott;M. R. Millar;K. Oghene;A. Ross.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Hallmarks of glycosylation in cancer
Jennifer Munkley;David J. Elliott.
Oncotarget (2016)
Mouse Homologues of the Human AZF Candidate Gene RBM Are Expressed in Spermatogonia and Spermatids, and Map to a Y Chromosome Deletion Interval Associated with a High Incidence of Sperm Abnormalities
Shantha K. Mahadevaiah;Teresa Odorisio;David J. Elliott;Áine Rattigan.
Human Molecular Genetics (1998)
SUPPA2: fast, accurate, and uncertainty-aware differential splicing analysis across multiple conditions
Juan L. Trincado;Juan C. Entizne;Gerald Hysenaj;Babita Singh.
Genome Biology (2018)
Mutations at the mitochondrial DNA polymerase (POLG) locus associated with male infertility
Anja T. Rovio;David R. Marchington;Susanne Donat;Hans Christian Schuppe.
Nature Genetics (2001)
The RNA helicase p68 is a novel androgen receptor coactivator involved in splicing and is overexpressed in prostate cancer.
Emma L. Clark;Anne Coulson;Caroline Dalgliesh;Prabhakar Rajan.
Cancer Research (2008)
Absence of mDazl produces a final block on germ cell development at meiosis
P T K Saunders;J M A Turner;M Ruggiu;M Taggart.
Reproduction (2003)
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