D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Genetics and Molecular Biology D-index 67 Citations 16,353 164 World Ranking 1700 National Ranking 866

Research.com Recognitions

Awards & Achievements

2010 - Robert J. and Claire Pasarow Foundation Medical Research Award

2008 - Distinguished Scientist Award, American Heart Association

2007 - Member of the National Academy of Sciences

2005 - Fellow of the American Academy of Arts and Sciences

2001 - Fellow of the American Association for the Advancement of Science (AAAS)

1999 - Member of the National Academy of Medicine (NAM)

Member of the Association of American Physicians

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • DNA
  • Enzyme

David Ginsburg mostly deals with Molecular biology, Von Willebrand factor, Immunology, Plasminogen activator inhibitor-1 and Plasminogen activator. His Molecular biology study combines topics in areas such as Complementary DNA, Biochemistry, Recombinant DNA and Golgi apparatus. His Von Willebrand factor research integrates issues from Genetics, Gene and Protein primary structure.

His work on Thrombotic thrombocytopenic purpura, ADAMTS13, Bone marrow and Chronic myelogenous leukemia as part of general Immunology research is frequently linked to ABL, bridging the gap between disciplines. His Plasminogen activator inhibitor-1 research is multidisciplinary, incorporating perspectives in Serpin, Mutant and Pathology. His Von Willebrand disease research focuses on subjects like Point mutation, which are linked to Locus.

His most cited work include:

  • Mutations in a member of the ADAMTS gene family cause thrombotic thrombocytopenic purpura (1428 citations)
  • Bleomycin-induced pulmonary fibrosis in transgenic mice that either lack or overexpress the murine plasminogen activator inhibitor-1 gene. (505 citations)
  • A computer-derived protocol to aid in the diagnosis of emergency room patients with acute chest pain. (459 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Molecular biology, Genetics, Von Willebrand factor, Immunology and Gene. His work deals with themes such as Gene expression, Recombinant DNA, Mutation, Factor V and Exon, which intersect with Molecular biology. In his research on the topic of Von Willebrand factor, Pathogenesis is strongly related with Thrombotic thrombocytopenic purpura.

His studies deal with areas such as Thrombosis, Coagulopathy and Gene mutation as well as Immunology. His Gene study deals with Cell biology intersecting with Secretion. David Ginsburg has included themes like Hemostasis and Endocrinology in his Platelet study.

He most often published in these fields:

  • Molecular biology (38.82%)
  • Genetics (28.82%)
  • Von Willebrand factor (27.94%)

What were the highlights of his more recent work (between 2016-2021)?

  • Cell biology (14.12%)
  • Gene (20.88%)
  • COPII (5.88%)

In recent papers he was focusing on the following fields of study:

David Ginsburg focuses on Cell biology, Gene, COPII, Endoplasmic reticulum and Secretion. The study incorporates disciplines such as Endothelial stem cell, Embryonic stem cell, Gene expression, Germline and In vivo in addition to Cell biology. His research in Gene intersects with topics in Molecular biology, Endocytosis and Factor V Leiden.

His work is dedicated to discovering how Secretion, Receptor are connected with Cellular secretion, Biotinylation, Cell culture and Extracellular and other disciplines. The Genetic association study which covers SNP that intersects with Von Willebrand factor. David Ginsburg is involved in the study of Von Willebrand factor that focuses on Von Willebrand disease in particular.

Between 2016 and 2021, his most popular works were:

  • Functions of the COPII gene paralogs SEC23A and SEC23B are interchangeable in vivo. (39 citations)
  • Functions of the COPII gene paralogs SEC23A and SEC23B are interchangeable in vivo. (39 citations)
  • The cargo receptor SURF4 promotes the efficient cellular secretion of PCSK9. (34 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Enzyme

His primary scientific interests are in Cell biology, COPII, Secretory pathway, Locus and Gene. The various areas that David Ginsburg examines in his Secretory pathway study include Secretion and Receptor. His study focuses on the intersection of Secretion and fields such as Gene targeting with connections in the field of Molecular biology.

His Molecular biology study combines topics in areas such as Point mutation, Tissue factor pathway inhibitor, Haploinsufficiency, Factor V Leiden and Genetic screen. His Locus study combines topics from a wide range of disciplines, such as Phenotype, Complementation, Zebrafish and Transgene. David Ginsburg has researched Gene in several fields, including Cell sorting and Tissue factor.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mutations in a member of the ADAMTS gene family cause thrombotic thrombocytopenic purpura

Gallia G. Levy;William C. Nichols;Eric C. Lian;Tatiana Foroud.
Nature (2001)

1873 Citations

Bleomycin-induced pulmonary fibrosis in transgenic mice that either lack or overexpress the murine plasminogen activator inhibitor-1 gene.

Daniel T. Eitzman;Ronald D. McCoy;Xianxian Zheng;William P. Fay.
Journal of Clinical Investigation (1996)

667 Citations

A computer-derived protocol to aid in the diagnosis of emergency room patients with acute chest pain.

Lee Goldman;Robert Jarret;Geoffrey Priest;John D'Avella.
The New England Journal of Medicine (1982)

608 Citations

Impact, Diagnosis and Treatment of von Willebrand Disease*

J. E. Sadler;P. M. Mannucci;E. Berntorp;N. Bochkov.
Thrombosis and Haemostasis (2000)

589 Citations

Genome-wide mapping of DNase hypersensitive sites using massively parallel signature sequencing (MPSS)

Gregory E. Crawford;Ingeborg E. Holt;James Whittle;Bryn D. Webb.
Genome Research (2005)

534 Citations

Human von Willebrand factor (vWF): isolation of complementary DNA (cDNA) clones and chromosomal localization

David Ginsburg;Robert I. Handin;David T. Bonthron;Timothy A. Donlon.
Science (1985)

532 Citations

Mutations in the ER–Golgi Intermediate Compartment Protein ERGIC-53 Cause Combined Deficiency of Coagulation Factors V and VIII

William C. Nichols;Uri Seligsohn;Ariella Zivelin;Valeri H. Terry.
Cell (1998)

476 Citations

Detectable clonal mosaicism from birth to old age and its relationship to cancer

Cathy C. Laurie;Cecelia A Laurie;Kenneth Rice;Kimberly F. Doheny.
Nature Genetics (2012)

445 Citations

cDNA cloning of human plasminogen activator-inhibitor from endothelial cells

D Ginsburg;R Zeheb;A Y Yang;U M Rafferty.
Journal of Clinical Investigation (1986)

426 Citations

von Willebrand disease.

William C. Nichols;David Ginsburg.
Medicine (1997)

423 Citations

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