2020 - Member of the National Academy of Medicine (NAM)
Member of the Association of American Physicians
His primary areas of study are Cancer research, Internal medicine, PI3K/AKT/mTOR pathway, Endocrinology and Protein kinase B. B. Mark Evers has researched Cancer research in several fields, including Carcinogenesis, Cancer stem cell, Downregulation and upregulation, Signal transduction and Metastasis. In his research, AKT2, P110α and Targeted Molecular Therapy is intimately related to Colorectal cancer, which falls under the overarching field of PI3K/AKT/mTOR pathway.
His work deals with themes such as Cholecystokinin, Receptor, Neurotensin and Curcumin, which intersect with Endocrinology. His work in Protein kinase B addresses subjects such as Intestinal mucosa, which are connected to disciplines such as Catenin, Wnt signaling pathway and Cell biology. His Cancer study combines topics from a wide range of disciplines, such as Oncology and Pathology.
The scientist’s investigation covers issues in Cancer research, Internal medicine, Endocrinology, Cell biology and PI3K/AKT/mTOR pathway. The Cancer research study combines topics in areas such as Cancer cell, Cancer, Colorectal cancer, Protein kinase B and Cell growth. His work in Cell growth addresses issues such as Molecular biology, which are connected to fields such as Cellular differentiation, Transcription factor and Cell culture.
His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology, Gene expression and Oncology. His Receptor research extends to the thematically linked field of Endocrinology. His Cell biology study integrates concerns from other disciplines, such as Apoptosis and Intestinal mucosa.
B. Mark Evers mainly investigates Cancer research, Internal medicine, Colorectal cancer, Neurotensin and Cancer. He combines subjects such as Wnt signaling pathway, Apoptosis, PI3K/AKT/mTOR pathway, Gene knockdown and AMPK with his study of Cancer research. He interconnects Cancer cell, Protein kinase B and Cell growth in the investigation of issues within PI3K/AKT/mTOR pathway.
His Internal medicine study combines topics in areas such as Endocrinology and Oncology. His work carried out in the field of Endocrinology brings together such families of science as Inflammation and Gut flora. His study explores the link between Colorectal cancer and topics such as Fatty acid synthase that cross with problems in Survivin.
B. Mark Evers spends much of his time researching Cancer research, Cancer, Cell biology, Gene knockdown and Wnt signaling pathway. B. Mark Evers has included themes like PI3K/AKT/mTOR pathway, Apoptosis, Protein kinase B, Signal transduction and Chemotherapy in his Cancer research study. Cancer is a primary field of his research addressed under Internal medicine.
His Internal medicine study often links to related topics such as Gut flora. His Cell biology study combines topics in areas such as Glycolysis, Cell culture and Ligand. The Catenin research B. Mark Evers does as part of his general Wnt signaling pathway study is frequently linked to other disciplines of science, such as Mechanism, therefore creating a link between diverse domains of science.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Stabilization of Snail by NF-κB Is Required for Inflammation-Induced Cell Migration and Invasion
Yadi Wu;Jiong Deng;Piotr G. Rychahou;Suimin Qiu.
Cancer Cell (2009)
Loss of FBP1 by Snail-mediated repression provides metabolic advantages in basal-like breast cancer.
Chenfang Dong;Tingting Yuan;Yadi Wu;Yifan Wang.
Cancer Cell (2013)
mTORC1 and mTORC2 regulate EMT, motility and metastasis of colorectal cancer via RhoA and Rac1 signaling pathways
Pat Gulhati;Kanika A. Bowen;Jianyu Liu;Payton D. Stevens.
Cancer Research (2011)
FOXO proteins regulate tumor necrosis factor-related apoptosis inducing ligand expression. Implications for PTEN mutation in prostate cancer.
Vijayanand Modur;Rakesh Nagarajan;B. Mark Evers;Jeffrey Milbrandt.
Journal of Biological Chemistry (2002)
G9a interacts with Snail and is critical for Snail-mediated E-cadherin repression in human breast cancer
Chenfang Dong;Yadi Wu;Jun Yao;Yifan Wang.
Journal of Clinical Investigation (2012)
Inflammation and the development of pancreatic cancer.
Buckminster Farrow;B.Mark Evers.
Surgical Oncology-oxford (2002)
Disrupting the Interaction of BRD4 With Diacetylated Twist Suppresses Tumorigenesis in Basal-Like Breast Cancer
Jian Shi;Yifan Wang;Yifan Wang;Lei Zeng;Yadi Wu.
Cancer Cell (2014)
The SNAG domain of Snail1 functions as a molecular hook for recruiting lysine‐specific demethylase 1
Yiwei Lin;Yadi Wu;Junlin Li;Chenfang Dong.
The EMBO Journal (2010)
mTOR inhibitors in cancer therapy
Yekaterina Y. Zaytseva;Joseph D. Valentino;Prateek gulhati;B. Mark Evers.
Cancer Letters (2012)
Increased Incidence of Well-Differentiated Thyroid Cancer Associated with Hashimoto Thyroiditis and the Role of the PI3k/Akt Pathway
Shawn D. Larson;Lindsey N. Jackson;Taylor S. Riall;Tatsuo Uchida.
Journal of The American College of Surgeons (2007)
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