His primary areas of study are Biochemistry, Phospholipase D, Cell biology, PLD2 and Phosphatidylinositol. His study in Signal transduction, Lysophosphatidic acid, Protein kinase C, Binding site and Phospholipase D activity falls within the category of Biochemistry. Andrew J. Morris combines subjects such as Phosphatidic acid, Golgi apparatus, ADP ribosylation factor and Second messenger system with his study of Phospholipase D.
His studies deal with areas such as Secretion, Exocytosis and Bone marrow as well as Cell biology. The PLD2 study combines topics in areas such as Membrane biogenesis, Phospholipase, Complementary DNA, Kinase and RHOA. As part of one scientific family, Andrew J. Morris deals mainly with the area of Phosphatidylinositol, narrowing it down to issues related to the Phospholipase C, and often Myristoylation, MARCKS, Biophysics, Phosphatidylserine and Phospholipid.
His scientific interests lie mostly in Biochemistry, Cell biology, Internal medicine, Endocrinology and Lysophosphatidic acid. Many of his studies on Biochemistry involve topics that are commonly interrelated, such as Cell culture. His research brings together the fields of Cell migration and Cell biology.
His studies link Cardiology with Internal medicine. His Lysophosphatidic acid research is multidisciplinary, relying on both Sphingosine and Lipid signaling. His Phospholipase D research integrates issues from PLD2, Phosphatidic acid, Protein kinase C and ADP ribosylation factor.
Andrew J. Morris mainly focuses on Internal medicine, Endocrinology, Lysophosphatidic acid, Autotaxin and Cancer research. His Lysophosphatidic acid research is multidisciplinary, incorporating perspectives in Cytotoxic T cell and Pharmacology. His study on Autotaxin also encompasses disciplines like
His Choline study improves the overall literature in Biochemistry. Many of his studies on Biochemistry apply to Streptococcus pyogenes as well. His Gene expression study combines topics in areas such as Transcription factor and Cell biology.
The scientist’s investigation covers issues in Endocrinology, Internal medicine, Lysophosphatidic acid, Adipose tissue and Cancer research. He has researched Endocrinology in several fields, including Inflammation, Autotaxin and Fatty liver. His Internal medicine study combines topics from a wide range of disciplines, such as Downregulation and upregulation and Gene expression.
His study on LPAR4 is often connected to CD68 as part of broader study in Lysophosphatidic acid. His work on White adipose tissue as part of general Adipose tissue study is frequently linked to Mirabegron, therefore connecting diverse disciplines of science. His Cancer research research also works with subjects such as
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Phosphatidylinositol 4-Phosphate 5-Kinase α Is a Downstream Effector of the Small G Protein ARF6 in Membrane Ruffle Formation
Akira Honda;Masahiro Nogami;Takeaki Yokozeki;Masakazu Yamazaki.
Phospholipase D2, a distinct phospholipase D isoform with novel regulatory properties that provokes cytoskeletal reorganization
William C. Colley;Tsung-Chang Sung;Richard Roll;John Jenco.
Current Biology (1997)
Characterization of Two Alternately Spliced Forms of Phospholipase D1 ACTIVATION OF THE PURIFIED ENZYMES BY PHOSPHATIDYLINOSITOL 4,5-BISPHOSPHATE, ADP-RIBOSYLATION FACTOR, AND RHO FAMILY MONOMERIC GTP-BINDING PROTEINS AND PROTEIN KINASE C-α
Scott M. Hammond;John M. Jenco;Shigeru Nakashima;Karen Cadwallader.
Journal of Biological Chemistry (1997)
Human ADP-ribosylation Factor-activated Phosphatidylcholine-specific Phospholipase D Defines a New and Highly Conserved Gene Family
Scott M. Hammond;Yelena M. Altshuller;Tsung Chang Sung;Simon A. Rudge.
Journal of Biological Chemistry (1995)
Physiological regulation of G protein-linked signaling.
Andrew J. Morris;Craig C. Malbon.
Physiological Reviews (1999)
Regulation of phospholipase D2: selective inhibition of mammalian phospholipase D isoenzymes by alpha- and beta-synucleins.
John M. Jenco;Andrew Rawlingson;Brenda Daniels;Andrew J. Morris.
Synucleins are a novel class of substrates for G protein-coupled receptor kinases.
Alexey N. Pronin;Andrew J. Morris;Andrei Surguchov;Jeffrey L. Benovic.
Journal of Biological Chemistry (2000)
Bayesian refinement of association signals for 14 loci in 3 common diseases.
Julian B. Maller;Gilean McVean;Gilean McVean;Jake Byrnes;Damjan Vukcevic.
Nature Genetics (2012)
ATR-X Syndrome Protein Targets Tandem Repeats and Influences Allele-Specific Expression in a Size-Dependent Manner
Martin J. Law;Karen M. Lower;Hsiao P.J. Voon;Jim R. Hughes.
Mutagenesis of phospholipase D defines a superfamily including a trans-Golgi viral protein required for poxvirus pathogenicity.
Tsung Chang Sung;Rachel L. Roper;Yue Zhang;Simon A. Rudge.
The EMBO Journal (1997)
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